IP Library Granted Patent US 7,563,772
Granted Patent B2
US 7,563,772 · App. 11/317,806 · Granted Jul 21, 2009

Methods of increasing cerebral blood flow

View Patent ↗
Loading inventors, assignments & file history…
Monitor This Case
Get email alerts when status or documents change.
Order Certified Copies
Most orders are placed with the USPTO same day — all within 24 business hours.
Order via The Patent Place →
Pre-filled with this patent's details
Quick Facts
Patent No.
US 7,563,772
App. No.
11/317,806
Granted
Jul 21, 2009
Kind
B2
Abstract

Methods of increasing blood flow in a mammalian brain blood vessel characterized by, or otherwise experiencing, decreased blood flow due to an ischemic or other hypoxic event, vasoconstriction or vasospasm following hemorrhagic stroke; due to chronic high blood pressure; and/or due to idiopathic causes are provided. The method for increasing blood flow in such a mammalian brain blood vessel includes administering to a patient in need thereof a therapeutically effective amount of an inhibitor of δ protein kinase C. In certain embodiments, the inhibitor can be chronically administered without causing desensitization of the patient to the inhibitor. Kits for increasing blood flow in a mammalian brain blood vessel characterized by, or otherwise experiencing, decreased blood flow due to an ischemic or other hypoxic event, vasoconstriction or vasospasm following hemorrhagic stroke; due to chronic high blood pressure; and/or due to idiopathic causes are provided.

Claims (21)

1. A method of increasing blood flow in a mammalian brain blood vessel experiencing decreased blood flow, comprising administering to a patient a therapeutically effective amount of a peptide inhibitor of δ protein kinase C (δPKC), wherein said peptide is selected from the group consisting of SEQ ID NO: 67, SEQ NO: 68, SEQ NO:69, SEQ NO: 70, SEQ NO: 71, SEQ NO: 72, SEQ NO: 73, SEQ NO: 74, SEQ NO: 75, SEQ NO: 76, SEQ NO: 77, SEQ NO: 78, SEQ NO: 79, SEQ NO: 80, SEQ NO: 81, SEQ NO: 82, and SEQ NO: 83, wherein said patient is suffering from an ischemic event, reperfusion injury, vasoconstriction, vasospasm, stroke, or chronic high blood pressure.

2. The method of claim 1 , wherein said blood vessel has been subjected to an ischemic or reperfusion event.

3. The method to claim 1 , wherein said blood vessel has been subjected to vasoconstriction or vasospasm.

4. The method of claim 2 , wherein the blood flow is increased in an ischemic penumbra arising from said ischemic event compared to the blood flow during said ischemic event.

5. The method of claim 3 , wherein the blood flow is increased in a hypoperfused area due to vasoconstriction or vasospasm compared to the blood flow during said vasoconstriction or vasospasm.

6. The method of claim 1 , wherein said blood vessel is a cerebral artery, capillary, or arteriole.

7. The method of claim 6 , wherein said cerebral artery is an anterior cerebral artery, a middle cerebral artery or a posterior cerebral artery.

8. The method of claim 1 , wherein said blood vessel is a carotid artery.

9. The method of claim 1 , wherein said therapeutically effective amount of an inhibitor of δ protein kinase C is an amount effective in increasing blood flow at least about 10% relative to blood flow prior to administering said inhibitor.

10. The method of claim 1 , wherein said therapeutically effective amount of an inhibitor of δPKC is an amount effective in increasing blood flow at least 50% relative to blood flow prior to administering said inhibitor.

11. The method of claim 1 , wherein said inhibitor is administered by a parenteral route.

12. The method of claim 1 , wherein said inhibitor is capable of chronic administration without causing desensitization to said inhibitor.

13. The method of claim 12 , wherein said inhibitor is administered multiple time to a patient.

14. A method of increasing blood flow in a mammalian brain blood vessel experiencing decreased blood flow, comprising administering to a patient a therapeutically effective amount of a peptide inhibitor of δ protein kinase C (δPKC), wherein said peptide inhibitor of δPKC is attached to a cell permeable carrier peptide, and wherein said peptide inhibitor of δPKC is selected from the group consisting of SEQ ID NO: 67, SEQ NO: 68, SEQ NO:69, SEQ NO: 70, SEQ NO: 71, SEQ NO: 72, SEQ NO: 73, SEQ NO: 74, SEQ NO: 75, SEQ NO: 76, SEQ NO: 77, SEQ NO: 78, SEQ NO: 79, SEQ NO: 80, SEQ NO: 81, SEQ NO: 82, and SEQ NO: 83, wherein said patient is suffering from an ischemic event, reperfusion injury, vasoconstriction, vasospasm, stroke, or chronic high blood pressure.

15. The method of claim 14 , wherein said cell permeable carrier peptide is selected from the group consisting of a Drosophilia Antennapedia homeodomain peptide, a Transactivating Regulatory Protein (Tat) peptide, and a polyarginine peptide.

16. The method of claim 14 , wherein said blood vessel is a cerebral artery, capillary, or arteriole.

17. The method of claim 14 , wherein said cerebral artery is an anterior cerebral artery, a middle cerebral artery or a posterior cerebral artery.

18. The method of claim 14 , wherein said blood vessel is a carotid artery.

19. The method of claim 14 , wherein said peptide inhibitor is administered by a parenteral route.

20. The method of claim 14 , wherein said peptide inhibitor is capable of chronic administration without causing desensitization to said inhibitor.

21. The method of claim 14 , wherein said peptide inhibitor is administered multiple times to a patient.

Assignments (2)
CONFIRMATORY LICENSE Recorded Nov 4, 2008
From: STANFORD UNIVERSITY
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 021782/0635 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Mar 13, 2006
From: MOCHLY-ROSEN, DARIA; BRIGHT, RACHEL
To: BOARD OF TRUSTEES OF THE LELAND STANFORD JUNIOR UNIVERSITY, THE
Reel/Frame 017332/0703 →