IP Library Granted Patent US 7,776,588
Granted Patent B2
US 7,776,588 · App. 12/110,972 · Granted Aug 17, 2010

MDR1 variants and methods for their use

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Quick Facts
Patent No.
US 7,776,588
App. No.
12/110,972
Granted
Aug 17, 2010
Kind
B2
Abstract

This invention provides the identification of a truncation polymorphism of the mdr1 gene that is linked to ivermectin sensitivity in subjects, such as collies. Also provided are methods for detecting drug transport sensitivity in a subject, and animal models and in vitro cell systems using cells from animals having an mdr1 truncation.

Claims (18)

1. A method of screening for whether a compound has a biological effect in a canine cell having a truncated P-gp protein, comprising:

contacting a first canine cell with the compound, wherein the first canine cell has a truncation mutation in a mdr1 gene at residues 294-297 of SEQ ID NO: 1, which mutation results in the canine cell having a truncated P-gp protein, to produce a contacted canine cell; and

comparing a characteristic of the contacted canine cell with

a) the same characteristic of the first canine cell prior to contact with the compound,

b) the same characteristic of a similar canine cell not contacted with the compound, or

c) the same characteristic of a second canine cell contacted with the compound, wherein the second canine cell does not have a truncation mutation in its mdr1 gene;

wherein a difference in the characteristic indicates the compound has a biological effect in the canine cell having the truncated P-gp protein.

2. The method of claim 1 , wherein contacting the canine cells with the compound occurs in vivo.

3. The method of claim 1 , wherein the canine cell is a gastrointestinal tissue cell, a renal tissue cell, a brain capillary endothelial cell, a liver tissue cell, a placental cell, bronchiolar epithelial cell, or an adrenal cortical cell.

4. The method of claim 1 , wherein the characteristic of the cell being compared is genetic, physiological, chemical, or morphological.

5. The method of claim 1 , wherein the biological effect results from a defect in efflux of the compound from the contacted canine cell.

6. The method of claim 1 , wherein the first canine cell is a neoplastic canine cell.

7. The method of claim 1 , wherein the compound is a neurokinin receptor antagonist, anti-emetic agent, beta-adrenergic receptor antagonist, antiinfective agent, antiepileptic agent, antineoplastic agent, analgesic agent, anti-psychotic agent, anti-parasitic agent or anti-depressive agent.

8. The method of claim 1 , wherein the compound is an antiviral agent.

9. The method of claim 1 , wherein the canine cell is a dog cell.

10. The method of claim 9 , wherein the dog cell is a Collie cell.

11. The method of claim 1 , wherein the first canine cell is contacted in vitro.

12. The method of claim 2 , wherein contacting the first canine cell in vivo comprises administering the compound to a dog.

Assignments (2)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Aug 9, 2013
From: WASHINGTON STATE UNIVERSITY RESEARCH FOUNDATION
To: WASHINGTON STATE UNIVERSITY
Reel/Frame 030979/0119 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jun 27, 2008
From: MEALEY, KATRINA L.
To: WASHINGTON STATE UNIVERSITY RESEARCH FOUNDATION
Reel/Frame 021164/0697 →