IP Library Granted Patent US 9,161,971
Granted Patent B2
US 9,161,971 · App. 13/537,330 · Granted Oct 20, 2015

Method for treating chronic lymphocytic leukemia (CC)

Inventors: Carl H. June (Merion Station, PA); Bruce Levine (Cherry Hill, NJ); Anne Chew (Cherry Hill, NJ); Stephen J. Schuster (Springfield, PA)
Assignee: The Trustees of the University of Pennsylvania
A61K39/0008C12N5/0636A61K2035/122A61K2039/5158C12N2501/51C12N2501/515
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Quick Facts
Patent No.
US 9,161,971
App. No.
13/537,330
Granted
Oct 20, 2015
Kind
B2
Abstract

The present invention relates generally to the treatment of PML by infusion of activated and expanded autologous lymphocytes.

Claims (15)

1. A method for treating Chronic Lymphocytic Leukemia (CLL) comprising administering to a patient with CLL a single dose comprising a population of about 5×10 9 to about 2×10 10 autologous T cells activated and expanded according to a method comprising,

(a) activating a population of T cells in vitro with an agent which stimulates a TCR/CD3 complex-associated signal in the T cells, wherein the agent is attached on a surface; and

(b) stimulating a CD28 accessory molecule on the surface of the T cells in vitro with a ligand that binds the CD28 accessory molecule on the surface of the T cells, wherein the ligand is attached on the same surface as the agent; and

wherein the activating and stimulating steps thereby induce proliferation of the T cells such that T cell counts are increased in the CLL patient at least one month after administration.

2. The method of claim 1 wherein the agent is selected from the group consisting of an anti-CD3 antibody or antigen-binding fragment thereof, an anti-TCR antibody or antigen-binding fragment thereof, a superantigen, an MHC-peptide tetramer, and an antigen in a form suitable to trigger a primary activation signal in the T cell when complexed with the TCR/CD3 complex.

3. The method of claim 1 wherein the ligand is selected from the group consisting of an anti-CD28 antibody or antigen-binding fragment thereof, CD80 or a CD28-binding fragment thereof, and CD86 or a CD28-binding fragment thereof.

4. The method of claim 1 wherein the surface is selected from the group consisting of a bead, a lipid bilayer, a cell surface, and a tissue-culture dish.

5. The method of claim 1 wherein the activated and expanded T cells are administered intravenously.

6. The method of claim 4 wherein said cell surface is of a human cell line.

7. The method of claim 6 wherein said human cell line is K562.

8. The method of claim 6 wherein said cell is genetically modified to express a human Fcγ receptor.

9. The method of claim 8 wherein said human Fcγ receptor comprises CD32 or CD64.

10. The method of claim 6 wherein said cell is further genetically modified to express a co-stimulatory molecule selected from the group consisting of CD80, CD86, 4-1BBL, OX40L, ICOS-L, ICAM, PD-L1 and PD-L2.

11. The method of claim 6 wherein said cell has been genetically modified to express a cytokine.

12. The method of claim 11 wherein said cytokine is selected from the group consisting of IL-2, GM-CSF, IL-4, TNF-α, and IFN-γ.

Continuity (3)
Division 13201829
Provisional Application 61155026 · Feb 24, 2009
Related Publication 20120263693A1 · Oct 18, 2012