IP Library Granted Patent US 9,206,408
Granted Patent B2
US 9,206,408 · App. 13/620,306 · Granted Dec 8, 2015

Microorganisms genetically engineered to have modified N-glycosylation activity

Inventors: Nico Luc Marc Callewaert (Hansbeke-Nevele, BE); Wouter Vervecken (Landskouter, BE); Karen Jacqueline Marcel De Pourcq (Ghent, BE); Steven Christian Jozef Geysens (Wannegem-Lede, BE); Mouna Guerfal (Veltem-Beisem, BE)
Assignees: Oxyrane UK Limited; VIB vzw; Universiteit Gent
C12N9/2488C07K14/00C12N9/1051C12N9/24C12N9/2402C12N9/2465C12P21/005C12P21/06C12Y204/01232C12Y302/01024C12Y302/01045C12Y302/01046C12Y302/01084C12Y302/01113A61K38/00
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Quick Facts
Patent No.
US 9,206,408
App. No.
13/620,306
Granted
Dec 8, 2015
Kind
B2
Abstract

Described herein are methods and genetically engineered cells useful for producing an altered N-glycosylation form of a target molecule. Also described are methods and molecules with altered N-glycosylation useful for treating a variety of disorders such as metabolic disorders.

Claims (49)

1. A method of producing an altered N-glycosylation form of a target protein, the method comprising:

providing a Yarrowia lipolytica or an Arxula adeninivorans cell genetically engineered to express an α-1,2-mannosidase polypeptide; and

introducing into the cell a nucleic acid encoding a target protein, wherein the cell produces the target protein in an altered N-glycosylation form.

2. The method of claim 1 , further comprising isolating the altered N-glycosylation form of the target protein.

3. The method of claim 1 , wherein the target protein is an exogenous protein.

4. The method of claim 1 , wherein the target protein is an endogenous protein.

5. The method of claim 1 , wherein the target protein is a mammalian protein.

6. The method of claim 1 , wherein the target protein is a pathogen protein, a lysosomal protein, a growth factor, a cytokine, a chemokine, or a fusion protein.

7. The method of claim 1 , wherein the target protein is a protein associated with a lysosomal storage disorder (LSD).

8. The method of claim 7 , wherein the lysosomal storage disorder is Gaucher disease, Tay-Sachs disease, Pompe disease, Niemann-Pick disease, or Fabry disease.

9. The method of claim 7 , wherein the target protein is selected from the group consisting of glucocerebrosidase, alpha-galactosidase, galactocerebrosidase, alpha-L-iduronidase, beta-D-galactosidase, beta-glucosidase, beta-hexosaminidase, beta-Dmannosidase, alpha-L-fucosidase, arylsulfatase B, arylsulfatase A, alpha-Nacteylgalactosaminidase, aspartylglucosaminidase, iduronate-2-sulfatase, alpha-glucosaminide-N-acetyltransferase, beta-D-glucoronidase, hyaluronidase, alpha-L-mannosidase, alphaneuraminidase, phosphotransferase, acid lipase, acid ceramidase, sphinogmyelinase, thioesterase, cathepsin K, and lipoprotein lipase.

10. The method of claim 7 , wherein the target protein is human alpha-galactosidase A.

11. The method of claim 1 , wherein the altered N-glycosylation form comprises one or more N-glycan structures selected from group consisting of Man 5 GlcNAc 2 , Man 8 GlcNAc 2 , and Man 9 GlcNAc 2 .

12. The method of claim 1 , wherein the α-1,2-mannosidase polypeptide is a MNS1 polypeptide.

13. The method of claim 1 , wherein the α-1,2-mannosidase polypeptide is targeted to the endoplasmic reticulum.

14. The method of claim 1 , further comprising additional processing of the target protein in its altered N-glycosylation form.

15. The method of claim 14 , wherein the additional processing comprises enzymatic or chemical treatment of the altered N-glycosylation form of the target protein.

16. The method of claim 1 , wherein the cell is further genetically engineered to comprise at least one additional modification of an N-glycosylation activity.

17. The method of claim 16 , wherein the at least one additional modification of an N-glycosylation activity comprises a deficiency in an N-glycosylation activity.

18. The method of claim 16 , wherein the at least one additional modification of an N-glycosylation activity comprises expression of a protein having N-glycosylation activity.

19. The method of claim 1 , wherein the target protein is a human protein.

20. The method of claim 18 , wherein the expressed protein is a polypeptide capable of effecting mannosyl phosphorylation of N-glycans.

21. The method of claim 1 , wherein the α-1,2-mannosidase is the Saccharomyces cerevisiae MNS1 polypeptide or the S. cerevisiae MNS1 polypeptide comprising one or more amino substitutions selected from the group consisting of R 273 L, R 273 G, R 269 S, and S 272 G.

22. An isolated Yarrowia lipolytica or Arxula adeninivorans cell genetically engineered to express an α-1,2-mannosidase polypeptide, wherein the cell is capable of producing a target protein in an altered glycosylation form if a nucleic acid encoding the target protein is introduced into the cell.

23. The cell of claim 22 , said cell further comprising a nucleic acid encoding a target protein, wherein the cell produces the target protein in an altered N-glycosylation form.

24. The cell of claim 22 , wherein the cell is further genetically engineered to comprise at least one additional modification of an N-glycosylation activity.

25. The cell of claim 24 , wherein the at least one additional modification of an N-glycosylation activity comprises a deficiency in an N-glycosylation activity.

26. The cell of claim 24 , wherein the at least one additional modification of an N-glycosylation activity comprises expression of a protein having N-glycosylation activity.

27. The cell of claim 22 , wherein the α-1,2-mannosidase polypeptide is a MNS1 polypeptide.

28. The cell of claim 22 , wherein the α-1,2-mannosidase polypeptide is targeted to the endoplasmic reticulum.

29. The cell of claim 26 , wherein the expressed protein is a polypeptide capable of effecting mannosyl phosphorylation of N-glycans.

30. The cell of claim 22 , wherein the α-1,2-mannosidase is the Saccharomyces cerevisiae MNS1 polypeptide or the S. cerevisiae MNS1 polypeptide comprising one or more amino substitutions selected from the group consisting of R 273 L, R 273 G, R 269 S, and S 272 G.

31. A method of producing an altered N-glycosylation form of a target protein, the method comprising contacting a target protein with a cell lysate prepared from a Yarrowia lipolytica or an Arxula adeninivorans cell genetically engineered to express an α-1,2-mannosidase polypeptide, wherein the contacting of the target protein with the cell lysate results in an altered N-glycosylation form of the target protein.

32. The method of claim 31 , wherein the cell is further genetically engineered to comprise at least one additional modification of an N-glycosylation activity.

33. The method of claim 32 , wherein the at least one additional modification of an N-glycosylation activity comprises a deficiency in an N-glycosylation activity.

34. The method of claim 32 , wherein the at least one additional modification of an N-glycosylation activity comprises expression of a protein having N-glycosylation activity.

35. The method of claim 31 , wherein the alpha α-1,2-mannosidase polypeptide is a MNS1 polypeptide.

36. The method of claim 31 , wherein the α-1,2-mannosidase polypeptide is targeted to the endoplasmic reticulum.

37. The method of claim 34 , wherein the expressed protein is a polypeptide capable of effecting mannosyl phosphorylation of N-glycans.

38. The method of claim 31 , wherein the α-1,2-mannosidase is the Saccharomyces cerevisiae MNS1 polypeptide or the S. cerevisiae MNS1 polypeptide comprising one or more amino substitutions selected from the group consisting of R 273 L, R 273 G, R 269 S, and S 272 G.

39. A substantially pure culture of Yarrowia lipolytica or Arxula adeninivorans cells, wherein the culture comprises a cell genetically engineered to express an α-1,2-mannosidase polypeptide, and wherein the cell is capable of producing a target protein in an altered glycosylation form if a nucleic acid encoding the target protein is introduced into the cell.

40. The substantially pure culture of claim 39 , said cell further comprising a nucleic acid encoding a target protein, wherein the cell produces the target protein in an altered N-glycosylation form.

41. The substantially pure culture of claim 39 , wherein the α-1,2-mannosidase polypeptide is a MNS1 polypeptide.

42. The substantially pure culture of claim 39 , wherein the α-1,2-mannosidase polypeptide is targeted to the endoplasmic reticulum.

43. The substantially pure culture of claim 39 , wherein the cell is further genetically engineered to comprise at least one additional modification of an N-glycosylation activity.

44. The substantially pure culture of claim 43 , wherein the at least one additional modification of an N-glycosylation activity comprises a deficiency in an N-glycosylation activity.

45. The substantially pure culture of claim 43 , wherein the at least one additional modification of an N-glycosylation activity comprises expression of a protein having N-glycosylation activity.

46. The substantially pure culture of claim 45 , wherein the expressed protein is a polypeptide capable of effecting mannosyl phosphorylation of N-glycans.

47. The substantially pure culture of claim 39 , wherein the α-1,2-mannosidase is the Saccharomyces cerevisiae MNS1 polypeptide or the S. cerevisiae MNS1 polypeptide comprising one or more amino substitutions selected from the group consisting of R 273 L, R 273 G, R 269 S, and S 272 G.

Assignments (2)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Apr 30, 2024
From: OXYRANE UK LTD
To: VIB VZW
Reel/Frame 067265/0154 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jan 15, 2014
From: CALLEWAERT, NICO LUC MARC; VERVECKEN, WOUTER; DE POURCQ, KAREN JACQUELINE MARCEL; GEYSENS, STEVEN CHRISTIAN JOZEF; GUERFAL, MOUNA
To: OXYRANE UK LIMITED; VIB VZW; UNIVERSITEIT GENT
Reel/Frame 031978/0951 →
Continuity (5)
Continuation 13095532 · Apr 27, 2011
Continuation 12062469 · Apr 3, 2008
Provisional Application 60940212 · May 25, 2007
Provisional Application 60909904 · Apr 3, 2007
Related Publication 20130190253A1 · Jul 25, 2013