IP Library Granted Patent US 11,603,539
Granted Patent B2
US 11,603,539 · App. 13/892,805 · Granted Mar 14, 2023

Methods for engineering allogeneic and immunosuppressive resistant T cell for immunotherapy

Inventors: Roman Galetto (Paris, FR); Agnès Gouble (Paris, FR); Stéphanie Grosse (Saint-Cyr sur Morin, FR); Cécile Mannioui (Villiers sur Marne, FR); Laurent Poirot (Paris, FR); Andrew Scharenberg (Seattle, WA); Julianne Smith (Le Plessis robinson, FR)
Assignee: CELLECTIS
C12N15/85A61K35/17C07K14/7051C07K14/70517C07K14/70521C07K14/70578C07K16/28C07K16/2803C12N5/0636A61K38/00A61K39/00C07K2317/14C07K2317/24C07K2317/569C07K2317/622C07K2319/00C07K2319/03C07K2319/74C12N2501/39C12N2501/51C12N2501/515C12N2501/599C12N2502/99C12N2510/00
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Quick Facts
Patent No.
US 11,603,539
App. No.
13/892,805
Granted
Mar 14, 2023
Kind
B2
Abstract

Methods for developing engineered T-cells for immunotherapy that are both non-alloreactive and resistant to immunosuppressive drugs. The present invention relates to methods for modifying T-cells by inactivating both genes encoding target for an immunosuppressive agent and T-cell receptor, in particular genes encoding CD52 and TCR. This method involves the use of specific rare cutting endonucleases, in particular TALE-nucleases (TAL effector endonuclease) and polynucleotides encoding such polypeptides, to precisely target a selection of key genes in T-cells, which are available from donors or from culture of primary cells. The invention opens the way to standard and affordable adoptive immunotherapy strategies for treating cancer and viral infections.

Claims (15)

1. A method for generating a population of doubly-inactivated primary human T-cells for immunotherapy comprising:

providing at least 5×10 6 primary human T cells from a single donor;

co-electroporating into said primary human T-cells:

(a) two different RNAs each encoding one half TALE-nuclease, wherein the two half TALE-nucleases dimerize to form a first TALE-nuclease that binds and cleaves the T-cell receptor alpha (TCRα) sequence consisting of SEQ ID NO:37 in a TCRα gene, and

(b) two different RNAs each encoding one half TALE-nuclease, wherein the two half TALE-nucleases dimerize to form a second TALE-nuclease that binds and cleaves the CD52 sequence consisting of SEQ ID NO:40 in a CD52 gene,

to generate a population of transfected human T cells comprising doubly-inactivated human T-cells having both TCRα and CD52 genes inactivated; and

expanding the population of doubly inactivated human T-cells; whereby a population of doubly-inactivated human T-cells sufficient for infusing into an individual for immunotherapy treatment is generated.

2. The method of claim 1 , wherein the population of doubly inactivated human T-cells is further modified to express a chimeric antigen receptor.

3. The method of claim 1 , wherein the first TALE-nuclease is encoded by the nucleotide sequences of SEQ ID NO:49 and SEQ ID NO:50.

4. The method of claim 3 , wherein the population of doubly inactivated human T-cells is further modified to express a chimeric antigen receptor.

5. The method of claim 1 , wherein the second TALE-nuclease is encoded by the nucleotide sequences of SEQ ID NO:55 and SEQ ID NO:56.

6. The method of claim 5 , wherein the population of doubly inactivated human T-cells is further modified to express a chimeric antigen receptor.

7. The method of claim 2 , further comprising purifying the doubly-inactivated cells.

8. The method of claim 4 , further comprising purifying the doubly-inactivated cells.

9. The method of claim 6 , further comprising purifying the doubly-inactivated cells.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jun 26, 2013
From: GALETTO, ROMAN; GOUBLE, AGNES; GROSSE, STEPHANIE; MANNIOUI, CECILE; POIROT, LAURENT; SCHARENBERG, ANDREW; SMITH, JULIANNE
To: CELLECTIS
Reel/Frame 030690/0162 →
Continuity (3)
Provisional Application 61651933 · May 25, 2012
Provisional Application 61696612 · Sep 4, 2012
Related Publication 20130315884A1 · Nov 28, 2013