IP Library Granted Patent US 9,487,764
Granted Patent B2
US 9,487,764 · App. 14/960,333 · Granted Nov 8, 2016

Bacteria engineered to treat diseases associated with hyperammonemia

Inventors: Dean Falb (Sherborn, MA); Vincent M. Isabella (Cambridge, MA); Jonathan W. Kotula (Somerville, MA); Paul F. Miller (Salem, CT)
Assignee: Synlogic, Inc.
C12N9/1029A61K35/741C12N9/1025C12N15/52C12N15/70C12P13/10C12R1/01C12R1/19C12Y203/01001
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Quick Facts
Patent No.
US 9,487,764
App. No.
14/960,333
Granted
Nov 8, 2016
Kind
B2
Abstract

Genetically engineered bacteria, pharmaceutical compositions thereof, and methods of modulating and treating disorders associated with hyperammonemia are disclosed.

Claims (22)

1. A genetically engineered bacterium comprising an arginine regulon,

wherein the bacterium comprises a gene encoding an arginine feedback resistant N-acetylglutamate synthetase (ArgA fbr ), wherein the ArgA fbr has reduced arginine feedback inhibition as compared to a wild-type N-acetylglutamate synthetase from the same bacterial subtype under the same conditions and wherein expression of the gene encoding ArgA fbr is controlled by a promoter that is induced by low-oxygen or anaerobic conditions; and

wherein the bacterium has been genetically engineered to lack a functional ArgR.

2. The bacterium of claim 1 , wherein each copy of a functional argR gene normally present in a corresponding wild-type bacterium has been deleted.

3. The bacterium of claim 1 , wherein each copy of a functional argG gene normally present in a corresponding wild-type bacterium has been independently deleted or rendered inactive by one or more nucleotide deletions, insertions or substitutions.

4. The bacterium of claim 3 , wherein each copy of the functional argG gene normally present in a corresponding wild-type bacterium has been deleted.

5. The bacterium of claim 1 , wherein under low-oxygen or anaerobic conditions, the transcription of each gene that is present in an operon comprising a functional ARG box and which encodes an arginine biosynthesis enzyme is increased as compared to a corresponding gene in a wild-type bacterium under the same conditions.

6. The bacterium of claim 1 , wherein the promoter that is induced under low-oxygen or anaerobic conditions is an FNR promoter.

7. The bacterium of claim 1 , wherein the arginine feedback resistant N-acetylglutamate synthetase gene has a DNA sequence selected from:

a) SEQ ID NO:28, and

b) a DNA sequence that, but for the redundancy of the genetic code, encodes the same polypeptide as encoded by SEQ ID NO:28.

8. The bacterium of claim 1 , wherein the bacterium is a non-pathogenic bacterium.

9. The bacterium of claim 1 , wherein the bacterium is a probiotic bacterium.

10. The bacterium of claim 1 , wherein the bacterium is selected from the group consisting of Bacteroides, Bifidobacterium, Clostridium, Escherichia, Lactobacillus , and Lactococcus.

11. The bacterium of claim 1 , wherein the bacterium is Escherichia coli strain Nissle.

12. The bacterium of claim 1 , wherein the gene encoding the arginine feedback resistant N-acetylglutamate synthetase (ArgA fbr ) is present on a plasmid in the bacterium and operably linked on the plasmid to the promoter that is induced under low-oxygen or anaerobic conditions.

13. The bacterium of claim 1 , wherein the gene encoding the arginine feedback resistant N-acetylglutamate synthetase (ArgA fbr ) is present in the bacterial chromosome and is operably linked in the chromosome to the promoter that is induced under low-oxygen or anaerobic conditions.

14. The bacterium of claim 1 , wherein the bacterium is an auxotroph in a gene that is complemented when the bacterium is present in a mammalian gut.

15. The bacterium of claim 14 , wherein the mammalian gut is a human gut.

16. A pharmaceutically acceptable composition comprising a genetically engineered bacterium comprising an arginine regulon, wherein the bacterium comprises a gene encoding an arginine feedback resistant N-acetylglutamate synthetase (ArgA fbr ), wherein the ArgA fbr has reduced arginine feedback inhibition as compared to a wild-type N-acetylglutamate synthetase from the same bacterial subtype under the same conditions, wherein expression of the gene encoding ArgA fbr is controlled by a promoter that is induced by low-oxygen or anaerobic conditions; and wherein the bacterium has been genetically engineered to lack a functional ArgR; and a pharmaceutically acceptable carrier.

17. The bacterium of claim 14 , wherein the bacterium is a thyA or dapB auxotroph.

18. The bacterium of claim 1 , wherein the bacterium comprises an antibiotic resistance gene.

Assignments (2)
MERGER Recorded Nov 28, 2018
From: SYNLOGIC, INC.
To: SYNLOGIC OPERATING COMPANY, INC.
Reel/Frame 047604/0094 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Mar 29, 2016
From: FALB, DEAN; ISABELLA, VINCENT M.; KOTULA, JONATHAN W.; MILLER, PAUL F.
To: SYNLOGIC, INC.
Reel/Frame 038124/0056 →
Continuity (11)
Provisional Application 62087854 · Dec 5, 2014
Provisional Application 62173706 · Jun 10, 2015
Provisional Application 62256041 · Nov 16, 2015
Provisional Application 62103513 · Jan 14, 2015
Provisional Application 62150508 · Apr 21, 2015
Provisional Application 62173710 · Jun 10, 2015
Provisional Application 62256039 · Nov 16, 2015
Provisional Application 62184811 · Jun 25, 2015
Provisional Application 62183935 · Jun 24, 2015
Provisional Application 62263329 · Dec 4, 2015
Related Publication 20160177274A1 · Jun 23, 2016