IP Library Granted Patent US 10,376,484
Granted Patent B2
US 10,376,484 · App. 15/105,116 · Granted Aug 13, 2019

Combination of bezafibrate and of resveratrol or resveratrol derivatives for the treatment and prevention of diseases involving a mitochondrial energy dysfunction

Inventors: Jean Bastin (Vitry sur Seine, FR); Fatima Djouadi (Vitry sur Seine, FR)
Assignees: Institut National de la Santé et de la Recherche Médicale (INSERM); Universite Paris Descartes
A61K31/195A61K31/05A61K31/7034
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Quick Facts
Patent No.
US 10,376,484
App. No.
15/105,116
Granted
Aug 13, 2019
Kind
B2
Abstract

The present invention relates to the combined use of bezafibrate and of resveratrol or resveratrol derivatives for the treatment of diseases involving a mitochondrial energy dysfunction, and also to a pharmaceutical kit comprising both bezafibrate and resveratrol or resveratrol derivatives. The combination is more particularly used in the treatment of moderate defects of β-oxidation of long-chain fatty acids or of the respiratory chain of mitochondria.

Claims (6)

1. A method of therapeutic treatment of a disease involving mitochondrial energy dysfunction in a patient in need thereof, comprising administering to said patient effective amounts in combination 1) of bezafibrate and 2) of resveratrol, cis-resveratrol, dihydro-resveratrol, piceid or mixtures thereof, wherein said disease involving mitochondrial energy dysfunction is characterized by moderate deficiency in mitochondrial long-chain fatty acid β-oxidation due to the presence of at least one mutation on each allele of one of the genes encoding one of the following enzymes: CPT2, VLCAD, and LCHAD.

2. The method according to claim 1 , wherein the combination 1) of bezafibrate and 2) of resveratrol, cis-resveratrol, dihydro-resveratrol, piceid or mixtures thereof is administered to a subject whose cells have palmitate β-oxidation activity of at least 30% of the activity of this same process in healthy control cells.

3. The method according to claim 1 , wherein the moderate deficiency in mitochondrial long-chain fatty acid β-oxidation is due to the presence of at least one mutation on each allele of the gene encoding CPT2.

4. The method according to claim 1 , wherein the treated subject suffers from moderate deficiency in mitochondrial long-chain fatty acid β-oxidation due to the presence of at least one mutation on each allele of the gene encoding VLCAD.

5. The method according to claim 1 , wherein the patient is selected from those for whom the combined presence 1) of bezafibrate and 2) of resveratrol, cis-resveratrol, dihydro-resveratrol, piceid or mixtures thereof improves β-oxidation in the patient's cells in vitro.

6. The method according to claim 1 , wherein a combination 1) of bezafibrate and 2) of resveratrol is used.

Assignments (2)
MERGER AND CHANGE OF NAME Recorded Dec 30, 2020
From: UNIVERSITE PARIS DESCARTES; UNIVERSITE PARIS DIDEROT PARIS 7; UNIVERSITÉ DE PARIS
To: UNIVERSITÉ DE PARIS
Reel/Frame 054871/0669 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Feb 3, 2017
From: BASTIN, JEAN; DJOUADI, FATIMA
To: INSTITUT NATIONAL DE LA SANTÉ ET DE LA RECHERCHE MÉDICALE (INSERM); UNIVERSITE PARIS DESCARTES
Reel/Frame 041168/0432 →
Priority Claims (1)
FR 13 63027 · Dec 19, 2013 · national
Continuity (1)
Related Publication 20160317483A1 · Nov 3, 2016