IP Library Granted Patent US 10,456,449
Granted Patent B2
US 10,456,449 · App. 15/316,108 · Granted Oct 29, 2019

Methods and uses for modulating bile acid homeostasis and treatment of bile acid disorders and diseases

Inventors: Lei Ling (Foster City, CA); Hui Tian (Foster City, CA)
Assignee: NGM Biopharmaceuticals, Inc.
A61K38/1825A61K31/155A61K31/165A61K31/519A61K31/52A61K31/573A61K31/575A61K31/64A61K38/13A61K38/208A61K38/26A61K38/28A61K45/06C07K14/50C07K2319/00
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Quick Facts
Patent No.
US 10,456,449
App. No.
15/316,108
Granted
Oct 29, 2019
Kind
B2
Abstract

Provided herein are methods of modulating bile acid homeostasis or treating a bile-acid related or associated disorder, comprising using variants and fusions of fibroblast growth factor 19 (FGF19), variants and fusions of fibroblast growth factor 21 (FGF21), fusions of FGF19 and/or FGF21, and variants or fusions of FGF19 and/or FGF21 proteins and peptide sequences (and peptidomimetics), in combination with agents effective in modulating bile acid homeostasis or treating a bile-acid related or associated disorder.

Claims (72)

1. A method of reducing bile acid synthesis in a subject in need thereof, comprising administering to the subject a therapeutically effective amount of

(i) a polypeptide comprising or consisting of the amino acid sequence set forth in SEQ ID NO:70, and

(ii) at least one additional agent, wherein the additional agent is an anti-CD20 agent, an anti-CD80 agent, an anti-cytokine antibody, an anti-retroviral therapy, an apical sodium bile acid transporter (ASBT) inhibitor, an autoimmune agent, azathioprine, cochicine, a CSCL10 neutralizing antibody, a CXCR3 ligand, cyclosporine, a cytokine anti-inflammatory drug (CSAID), a cytokine, a growth factor, a fibrate, a fish oil, an immune checkpoint inhibitor, or a non-steroidal anti-inflammatory drug (NSAID);

thereby reducing bile acid synthesis in the subject without inducing HGG hepatocellular carcinoma (HCC) formation.

2. The method of claim 1 , wherein the additional agent is an anti-CD20 agent.

3. The method of claim 1 , wherein the additional agent is an anti-CD80 agent.

4. The method of claim 1 , wherein the additional agent is an anti-cytokine antibody.

5. The method of claim 1 , wherein the additional agent is an anti-retroviral therapy.

6. The method of claim 1 , wherein the additional agent is an ASBT inhibitor.

7. The method of claim 1 , wherein the additional agent is an autoimmune agent.

8. The method of claim 1 , wherein the additional agent is azathioprine.

9. The method of claim 1 , wherein the additional agent is cochicine.

10. The method of claim 1 , wherein the additional agent is a CSCL10 neutralizing antibody.

11. The method of claim 1 , wherein the additional agent is a CXCR3 ligand.

12. The method of claim 1 , wherein the additional agent is cyclosporine.

13. The method of claim 1 , wherein the additional agent is a CSAID.

14. The method of claim 1 , wherein the additional agent is a cytokine.

15. The method of claim 14 , wherein the cytokine is IL-12.

16. The method of claim 1 , wherein the additional agent is a growth factor.

17. The method of claim 1 , wherein the additional agent is a fibrate.

18. The method of claim 1 , wherein the additional agent is a fish oil.

19. The method of claim 1 , wherein the additional agent is an immune checkpoint inhibitor.

20. The method of claim 1 , wherein the additional agent is a NSAID.

21. The method of claim 1 , wherein the subject has nonalcoholic steatohepatitis (NASH).

22. The method of claim 1 , wherein the subject has primary biliary cirrhosis (PBC).

23. The method of claim 1 , wherein the subject has cholestasis.

24. The method of claim 1 , wherein the subject has primary sclerosing cholangitis (PSC).

25. The method of claim 1 , wherein the subject has bile acid diarrhea (BAD) or bile acid malabsorption.

26. The method of claim 1 , wherein the subject has pregnancy intrahepatic cholestasis (PIC).

27. The method of claim 1 , wherein the subject has liver fibrosis.

28. The method of claim 1 , wherein the subject has nonalcoholic fatty liver disease (NAFLD).

29. The method of claim 1 , wherein the subject has cirrhosis.

30. The method of claim 1 , wherein the polypeptide comprises the amino acid sequence set forth in SEQ ID NO:70.

31. The method of claim 1 , wherein the polypeptide consists of the amino acid sequence set forth in SEQ ID NO:70.

32. A method of reducing bile acid synthesis in a subject in need thereof, comprising administering to the subject a therapeutically effective amount of

(i) a polypeptide comprising or consisting of the amino acid sequence set forth in SEQ ID NO:70, and

(ii) at least one additional agent, wherein the additional agent is a farnesoid X receptor (FXR) agonist or a steroid;

thereby reducing bile acid synthesis in the subject without inducing HCC formation.

33. The method of claim 32 , wherein the additional agent is a FXR agonist.

34. The method of claim 32 , wherein the additional agent is a steroid.

35. The method of claim 32 , wherein the steroid is a glucocorticoid.

36. The method of claim 32 , wherein the subject has NASH.

37. The method of claim 32 , wherein the subject has PBC.

38. The method of claim 32 , wherein the subject has cholestasis.

39. The method of claim 32 , wherein the subject has PSC.

40. The method of claim 32 , wherein the subject has an error of bile acid synthesis.

41. The method of claim 32 , wherein the subject has BAD or bile acid malabsorption.

42. The method of claim 32 , wherein the subject has PIC.

43. The method of claim 32 , wherein the subject has liver fibrosis.

44. The method of claim 32 , wherein the subject has NAFLD.

45. The method of claim 32 , wherein the subject has cirrhosis.

46. The method of claim 32 , wherein the polypeptide comprises the amino acid sequence set forth in SEQ ID NO:70.

47. The method of claim 32 , wherein the polypeptide consists of the amino acid sequence set forth in SEQ ID NO:70.

48. A method of reducing bile acid synthesis in a subject in need thereof, comprising administering to the subject a therapeutically effective amount of

(i) a polypeptide comprising or consisting of the amino acid sequence set forth in SEQ ID NO:70, and

(ii) at least one additional agent, wherein the additional agent is chenodeoxycholic acid (CDCA), obeticholic acid (OCA), or ursodeoxycholic acid (UDCA);

thereby reducing bile acid synthesis in the subject without inducing HCC formation.

49. The method of claim 48 , wherein the additional agent is CDCA.

50. The method of claim 48 , wherein the additional agent is OCA.

51. The method of claim 48 , wherein the additional agent is UDCA.

52. The method of claim 48 , wherein the subject has NASH.

53. The method of claim 48 , wherein the subject has PBC.

54. The method of claim 48 , wherein the subject has cholestasis.

55. The method of claim 48 , wherein the subject has PSC.

56. The method of claim 48 , wherein the subject has an error of bile acid synthesis.

57. The method of claim 48 , wherein the subject has BAD or bile acid malabsorption.

58. The method of claim 48 , wherein the subject has PIC.

59. The method of claim 48 , wherein the subject has liver fibrosis.

60. The method of claim 48 , wherein the subject has NAFLD.

61. The method of claim 48 , wherein the subject has cirrhosis.

62. The method of claim 48 , wherein the polypeptide comprises the amino acid sequence set forth in SEQ ID NO:70.

63. The method of claim 48 , wherein the polypeptide consists of the amino acid sequence set forth in SEQ ID NO:70.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Feb 22, 2017
From: LING, LEI; TIAN, HUI
To: NGM BIOPHARMACEUTICALS, INC.
Reel/Frame 041344/0894 →
Continuity (2)
Provisional Application 62012899 · Jun 16, 2014
Related Publication 20170182123A1 · Jun 29, 2017