IP Library Granted Patent US 9,970,010
Granted Patent B2
US 9,970,010 · App. 15/466,631 · Granted May 15, 2018

Oligomers

Inventors: Linda Popplewell (Surrey, GB); Ian Graham (Evesham, GB); John George Dickson (Surrey, GB)
Assignee: Royal Holloway, University of London
C12N15/113A61K31/7088C12N2310/11C12N2310/321C12N2310/3233C12N2310/351
View Patent ↗
Loading inventors, assignments & file history…
Monitor This Case
Get email alerts when status or documents change.
Order Certified Copies
Most orders are placed with the USPTO same day — all within 24 business hours.
Order via The Patent Place →
Pre-filled with this patent's details
Quick Facts
Patent No.
US 9,970,010
App. No.
15/466,631
Granted
May 15, 2018
Kind
B2
Abstract

Molecules are provided for inducing or facilitating exon skipping in forming spliced mRNA products from pre-mRNA molecules in cells. The molecules may be provided directly as oligonucleotides or expression products of vectors that are administered to a subject. High rates of skipping can be achieved. High rates of skipping reduce the severity of a disease like Duchene Muscular Dystrophy so that the disease is more like Becker Muscular Dystrophy. This is a severe reduction in symptom severity and mortality.

Claims (22)

1. An oligomer for ameliorating Duchenne muscular dystrophy (DMD), the oligomer comprising at least 25 contiguous bases of:

a) 

(SEQ ID NO: 10)

CXG XXG CCX CCG GXX CXG AAG GXG XXC XXG; 

or

b)

(SEQ ID NO: 12)

XXG CCX CCG GXX CXG AAG GXG XXC XXG XAC; 

wherein X=U or T, wherein the sequence of the at least 25 contiguous bases can vary from SEQ ID NO: 10 or 12 at up to two base positions, and wherein the oligomer comprises one or more synthetically modified nucleotides.

2. The oligomer according to claim 1 , wherein the oligomer can bind to a target site to cause exon skipping of exon 53 of the dystrophin gene.

3. The oligomer according to claim 1 , wherein the oligomer can bind to a target site to cause exon skipping of an exon of the dystrophin gene at a rate of at least 50%.

4. The oligomer according to claim 1 , wherein the oligomer can bind to a target site to cause exon skipping of an exon of the dystrophin gene at a rate of at least 70%.

5. The oligomer according to claim 1 , wherein the oligomer can bind to a target site to cause exon skipping of an exon of the dystrophin gene at a rate of at least 90%.

6. The oligomer according to claim 1 , wherein the one or more synthetically modified nucleotides are modified at the 2′ position of the ribose.

7. The oligomer according to claim 6 , wherein the one or more synthetically modified nucleotides are 2′-O-methyl oligonucleotide.

8. The oligomer according to claim 1 , wherein the one or more synthetically modified nucleotides are a phosphorodiamidate morpholino oligonucleotide.

9. The oligomer according to claim 1 , wherein the oligomer is at least 28 and at most 32 bases in length.

10. The oligomer according to claim 1 , wherein the oligomer is at least 29 and at most 31 bases in length.

11. The oligomer according to claim 1 , wherein the oligomer is conjugated to or complexed with a targeting protein that targets the oligomer to muscle tissue.

12. The oligomer according to claim 1 , wherein the oligomer is conjugated to or complexed with a drug for treating Duchenne muscular dystrophy.

13. The oligomer according to claim 1 , wherein the oligomer is conjugated to or complexed with an arginine-rich cell penetrating peptide.

14. A pharmaceutical composition for ameliorating DMD, the composition comprising an oligomer according to claim 1 , and a pharmaceutically acceptable carrier, adjuvant or vehicle.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Apr 11, 2017
From: POPPLEWELL, LINDA; GRAHAM, IAN; DICKSON, JOHN GEORGE
To: ROYAL HOLLOWAY, UNIVERSITY OF LONDON
Reel/Frame 041960/0070 →
Continuity (7)
Division 14736154 · Aug 24, 2015
Division 14045841 · Oct 4, 2013
Continuation 13307926 · Nov 30, 2011
Division 12556626 · Sep 10, 2009
Provisional Application 61164987 · Mar 31, 2009
Provisional Application 61096073 · Sep 11, 2008
Related Publication 20170204413A1 · Jul 20, 2017