IP Library Granted Patent US 10,287,586
Granted Patent B2
US 10,287,586 · App. 15/661,750 · Granted May 14, 2019

Antisense molecules and methods for treating pathologies

Inventors: Stephen Donald Wilson (Applecross, AU); Sue Fletcher (Bayswater, AU); Abbie Adams (Kalamunda, AU); Penny Meloni (Mount Hawthorn, AU)
Assignee: The University of Western Australia
C12N15/113C12N15/111C12N2310/11C12N2310/315C12N2310/3181C12N2310/321C12N2310/3233C12N2310/351C12N2320/33
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Quick Facts
Patent No.
US 10,287,586
App. No.
15/661,750
Granted
May 14, 2019
Kind
B2
Abstract

An antisense molecule capable of binding to a selected target site to induce exon skipping in the dystrophin gene, as set forth in SEQ ID NO: 1 to 59.

Claims (12)

1. An antisense oligonucleotide of 22 bases in length, wherein the antisense oligonucleotide is 100% complementary to a target region of exon 45 of the human dystrophin pre-mRNA, wherein the target region is annealing site H45A(−03+19), wherein the antisense oligonucleotide is a morpholino antisense oligonucleotide, and wherein the antisense oligonucleotide specifically hybridizes to the annealing site inducing exon 45 skipping, or a pharmaceutically acceptable salt thereof.

2. The antisense oligonucleotide of claim 1 , wherein the antisense oligonucleotide or pharmaceutically acceptable salt thereof is chemically linked to one or more moieties or conjugates that enhance the activity, cellular distribution or cellular uptake of the oligonucleotide.

3. The antisense oligonucleotide of claim 1 , wherein the antisense oligonucleotide or pharmaceutically acceptable salt thereof is chemically linked to a polyethylene glycol chain.

4. An antisense oligonucleotide of 22 bases in length, wherein the antisense oligonueotide is 100% complementary to a target region of exon 45 of the human dystrophin pre-mRNA, wherein the target region is annealing site H45A(−03+19), wherein the antisense oligonucleotide is a morpholino antisense oligonucleotide, and wherein the antisense oligonucleotide specifically hybridizes to the annealing site inducing exon 45 skipping.

5. The antisense oligonucleotide of claim 4 , wherein the antisense oligonucleotide is chemically linked to one or more moieties or conjugates that enhance the activity, cellular distribution or cellular uptake of the oligonucleotide.

6. The antisense oligonucleotide of claim 4 , wherein the antisense oligonucleotide is chemically linked to a polyethylene glycol chain.

7. A pharmaceutical composition comprising (i) an antisense oligonucleotide of 22 bases in length, wherein the antisense oligonucleotide is 100% complementary to a target region of exon 45 of the human dystrophin pre-mRNA, wherein the target region is annealing site H45A(−03+19), wherein the antisense oligonucleotide is a morpholino antisense oligonucleotide, and wherein the antisense oligonucleotide specifically hybridizes to the annealing site inducing exon 45 skipping, or a pharmaceutically acceptable salt thereof, and (ii) a pharmaceutically acceptable carrier.

8. The pharmaceutical composition of claim 7 , wherein the antisense oligonucleotide or pharmaceutically acceptable, salt thereof is chemically linked to one or more moieties or conjugates that enhance the activity, cellular distribution or cellular uptake of the oligonucleotide.

9. The pharmaceutical composition of claim 7 , wherein the antisense oligonucleotide or pharmaceutically acceptable salt thereof is chemically linked to a polyethylene glycol chain.

10. A pharmaceutical composition comprising (i) an antisense oligonucleotide of 22bases in length, wherein the antisense oligonucleotide is 100% complementary to a target region of exon 45 of the human dystrophin pre-mRNA, wherein the target region is annealing site H45A(−03+19), wherein the antisense oligonucleotide is a morpholino antisense oligonucleotide, and wherein the antisense oligonucleotide specifically hybridizes to the annealing site inducing exon 45 skipping, and (ii) a pharmaceutically acceptable carrier.

11. The pharmaceutical composition of claim 10 , wherein the antisense oligonucleotide is chemically linked to one or more moieties or conjugates that enhance the activity, cellular distribution or cellular uptake of the oligonucleotide.

12. The pharmaceutical composition of claim 10 , wherein the antisense oligonucleotide is chemically linked to a polyethylene glycol chain.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Aug 7, 2017
From: WILTON, STEPHEN DONALD; FLETCHER, SUE; ADAMS, ABBIE; MELONI, PENNY
To: THE UNIVERSITY OF WESTERN AUSTRALIA
Reel/Frame 043221/0143 →
Priority Claims (1)
AU 2009905549 · Nov 12, 2009 · national
Continuity (4)
Continuation 14944886 · Nov 18, 2015
Continuation 14108137 · Dec 16, 2013
Continuation 13509331
Related Publication 20180171333A1 · Jun 21, 2018
Cited By (17)
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