IP Library Granted Patent US 10,821,112
Granted Patent B2
US 10,821,112 · App. 16/084,852 · Granted Nov 3, 2020

Compositions and methods for the treatment of a beta-catenin-associated disease or disorder

Inventors: Marc Abrams (Natick, MA); Shanthi Ganesh (Shrewsbury, MA)
Assignee: Dicerna Pharmaceuticals, Inc.
A61K31/519A61K9/0019A61K9/5123A61K31/7088A61P35/00C12N15/113C12N2310/14
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Quick Facts
Patent No.
US 10,821,112
App. No.
16/084,852
Granted
Nov 3, 2020
Kind
B2
Abstract

Disclosed herein are methods for the treatment of cancer, comprising administering to a subject a β-catenin nucleic acid inhibitor molecule and a therapeutically effective amount of an MEK inhibitor or a c-Myc nucleic acid inhibitor molecule. Also disclosed herein is a pharmaceutical composition comprising a therapeutically effective amount of a β-catenin nucleic acid inhibitor molecule; a therapeutically effective amount of an MEK inhibitor or a c-Myc nucleic acid inhibitor molecule and at least one pharmaceutical carrier.

Claims (32)

1. A method of treating a β-catenin-associated cancer in a subject, comprising administering to the subject:

a therapeutically effective amount of a β-catenin nucleic acid inhibitor molecule; and

a therapeutically effective amount of a MEK inhibitor,

wherein the β-catenin nucleic acid inhibitor molecule is a dsRNAi inhibitor molecule,

wherein prior to administering the β-catenin nucleic acid inhibitor molecule, the subject has undergone at least two administrations of a prior treatment for the β-catenin-associated cancer, and

wherein the prior treatment is administration of a MEK inhibitor.

2. The method of claim 1 , wherein the MEK inhibitor is trametinib.

3. The method of claim 1 , wherein the β-catenin-associated cancer is colorectal cancer, hepatocellular carcinoma, or melanoma.

4. The method of claim 1 , wherein the subject is a human.

5. The method of claim 1 , wherein the double-stranded region of the dsRNAi inhibitor molecule is between 15 and 40 nucleotides in length.

6. The method of claim 1 , wherein the dsRNAi inhibitor molecule comprises a sense and an antisense strand and a duplex region of between 18 and 40 nucleotides, wherein the sense strand is 25-34 nucleotides in length and the antisense strand is 26-38 nucleotides in length and comprises 1-5 single-stranded nucleotides at its 3′ terminus.

7. The method of claim 6 , wherein the sense strand comprises the sequence of SEQ ID NO: 1 and the antisense strand comprises the sequence of SEQ ID NO: 2.

8. The method of claim 1 , wherein the β-catenin nucleic acid inhibitor molecule is formulated with a lipid nanoparticle.

9. The method of claim 8 , wherein the lipid nanoparticle comprises a cationic lipid and a pegylated lipid.

10. The method of claim 1 ,

wherein prior to administering the β-catenin nucleic acid inhibitor molecule, the subject has undergone prior treatment for the β-catenin-associated cancer and developed resistance to that treatment.

11. The method of claim 10 , wherein the MEK inhibitor of the prior treatment is trametinib.

12. The method of claim 10 , wherein the MEK inhibitor administered to the subject is trametinib.

13. The method of claim 11 , wherein the MEK inhibitor administered to the subject is trametinib.

14. The method of claim 1 , wherein the prior treatment is administration of a MEK inhibitor.

15. The method of claim 14 , wherein the MEK inhibitor of the prior treatment is trametinib.

16. The method of claim 14 , wherein the MEK inhibitor administered to the subject is trametinib.

17. The method of claim 15 , wherein the MEK inhibitor administered to the subject is trametinib.

18. The method of claim 1 , wherein prior to administering the β-catenin nucleic acid inhibitor molecule, the subject has undergone at least three, four, five, or six administrations of a prior treatment for the β-catenin-associated cancer.

19. The method of claim 1 , wherein the β-catenin-associated cancer has metastasized.

20. The method of claim 19 , wherein the β-catenin-associated cancer is colorectal cancer.

21. The method of claim 20 , wherein the colorectal cancer has metastasized to the liver.

22. The method of claim 19 , wherein the treatment reduces metastases in the subject.

23. A method of treating a β-catenin-associated cancer in a subject, comprising administering to the subject:

a therapeutically effective amount of a β-catenin nucleic acid inhibitor molecule; and

a therapeutically effective amount of a MEK inhibitor,

wherein the β-catenin nucleic acid inhibitor molecule is a dsRNAi inhibitor molecule comprising a sense and an antisense strand and a duplex region of between 18 and 40 nucleotides, wherein the sense strand is 25-34 nucleotides in length and the antisense strand is 26-38 nucleotides in length and comprises 1-5 single-stranded nucleotides at its 3′ terminus, and wherein the sense strand comprises the sequence of SEQ ID NO: 1 and the antisense strand comprises the sequence of SEQ ID NO: 2.

Assignments (2)
CHANGE OF ADDRESS Recorded Aug 13, 2020
From: DICERNA PHARMACEUTICALS, INC.
To: DICERNA PHARMACEUTICALS, INC.
Reel/Frame 053493/0724 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Sep 13, 2018
From: ABRAMS, MARC; GANESH, SHANTHI
To: DICERNA PHARMACEUTICALS, INC.
Reel/Frame 046870/0352 →
Continuity (4)
Provisional Application 62309449 · Mar 16, 2016
Provisional Application 62318529 · Apr 5, 2016
Provisional Application 62365164 · Jul 21, 2016
Related Publication 20190070180A1 · Mar 7, 2019