IP Library Granted Patent US 10,774,327
Granted Patent B2
US 10,774,327 · App. 16/263,200 · Granted Sep 15, 2020

Oligonucleotide compounds for targeting huntingtin mRNA

Inventors: Anastasia Khvorova (Westborough, MA); Neil Aronin (Newtonville, MA); Julia Alterman (Worcester, MA)
Assignee: UNIVERSITY OF MASSACHUSETTS
C12N15/113A61K9/0085A61K31/713C12N2310/14C12N2310/315C12N2310/343C12N2310/344C12N2310/346C12N2310/3515C12N2310/3517C12N2310/3519C12N2310/52C12N2320/11C12N2320/30C12N2320/32C12N2320/51
View Patent ↗
Loading inventors, assignments & file history…
Monitor This Case
Get email alerts when status or documents change.
Order Certified Copies
Most orders are placed with the USPTO same day — all within 24 business hours.
Order via The Patent Place →
Pre-filled with this patent's details
Quick Facts
Patent No.
US 10,774,327
App. No.
16/263,200
Granted
Sep 15, 2020
Kind
B2
Abstract

This disclosure relates to novel huntingtin targets. Novel oligonucleotides for the treatment of Huntington's disease are also provided.

Claims (20)

1. A vector comprising a regulatory sequence operably linked to a nucleotide sequence that encodes an RNA duplex comprising a sense strand and an antisense strand wherein the sense strand is between 15 and 30 nucleotides in length and comprises a nucleotide sequence which is at least 80% identical to SEQ ID NO: 1056 and wherein antisense strand comprises a nucleotide sequence which is at least 85% identical to 5′UAAGCAUGGAGCUAGCAGGC3′ (SEQ ID NO: 328) and is complementary to at least 7 contiguous nucleotides of SEQ ID NO: 1056.

2. The vector of claim 1 wherein the antisense strand comprises no more than 3 mismatches with SEQ ID NO: 1056.

3. The vector of claim 1 wherein the antisense strand is complementary to at least 10 contiguous nucleotides of SEQ ID NO: 1056.

4. The vector of claim 1 wherein the antisense strand is complementary to at least 13 contiguous nucleotides of SEQ ID NO: 1056.

5. The vector of claim 4 , wherein the antisense strand comprises a nucleotide sequence which is at least 90% identical to 5′UAAGCAUGGAGCUAGCAGGC3′ (SEQ ID NO: 328); and wherein the sense strand is between 15 and 30 nucleotides in length and comprises a nucleotide sequence which is at least 85% identical to SEQ ID NO: 1056.

6. A recombinant adeno-associated viruses (rAAV) comprising the vector of claim 1 and an AAV capsid.

7. A pharmaceutical composition comprising the rAAV of claim 6 and a pharmaceutically acceptable carrier.

8. A vector comprising a regulatory sequence operably linked to a nucleotide sequence that encodes the RNA duplex of claim 1 .

9. A recombinant adeno-associated viruses (rAAV) comprising the vector of claim 8 and an AAV capsid.

10. A pharmaceutical composition comprising the rAAV of claim 9 and a pharmaceutically acceptable carrier.

11. A method for inhibiting expression of HTT gene in a cell, the method comprising introducing into the cell the RNA duplex of claim 1 or a vector comprising a nucleotide sequence encoding the RNA duplex of claim 1 .

12. A method for inhibiting expression of HTT gene in a cell, the method comprising introducing into the cell the RNA duplex of claim 1 or a vector comprising a nucleotide sequence encoding the RNA duplex of claim 1 .

13. A method of treating Huntington's Disease in a patient, the method comprising administering to the patient a therapeutically effective amount of the rAAV of claim 6 .

14. The method of claim 13 , wherein the rAAV is administered to a putamen of the patient.

15. The method of claim 13 , wherein the rAAV is administered to one or more regions of the parenchyma of the brain, with at least one region being a putamen.

16. The method of claim 13 , wherein administering the rAAV to the patient causes a decrease in HTT gene mRNA in the striatum, the cortex, or both the striatum and the cortex of the patient.

17. A method of treating Huntington's Disease in a patient, the method comprising administering to the patient a therapeutically effective amount of the rAAV of claim 9 .

18. The method of claim 17 , wherein the rAAV is administered to a putamen of the patient.

19. The method of claim 17 , wherein the rAAV is administered to one or more regions of the parenchyma of the brain, with at least one region being a putamen.

20. The method of claim 17 , wherein administering the rAAV to the patient causes a decrease in HTT gene mRNA in the striatum, the cortex, or both the striatum and the cortex of the patient.

Assignments (2)
INVENTION OWNERSHIP AGREEMENT AND CONSENT JUDGEMENT Recorded Jun 2, 2021
From: PHIO PHARMACEUTICALS CORP.
To: UNIVERSITY OF MASSACHUSETTS
Reel/Frame 056450/0417 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Mar 6, 2020
From: KHVOROVA, ANASTASIA; ARONIN, NEIL; ALTERMAN, JULIA
To: UNIVERSITY OF MASSACHUSETTS
Reel/Frame 052040/0587 →
Continuity (5)
Continuation 15697120 · Sep 6, 2017
Continuation 15089319 · Apr 1, 2016
Provisional Application 62289274 · Jan 31, 2016
Provisional Application 62142731 · Apr 3, 2015
Related Publication 20190225965A1 · Jul 25, 2019
Cited By (5)
US 12,297,430 US 12,365,894 US 12,534,724 US 12,692,498 US 12,709,748