IP Library Patent Application 17715218
Patent Application
App. No. 17/715,218

METHODS FOR ENGINEERING ALLOGENEIC AND HIGHLY ACTIVE T CELL FOR IMMUNOTHERAPHY

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Patent No.
US None
App. No.
17/715,218
Abstract

The present invention relates to methods for developing engineered T-cells for immunotherapy that are non-alloreactive. The present invention relates to methods for modifying T-cells by inactivating both genes encoding T-cell receptor and an immune checkpoint gene to unleash the potential of the immune response. This method involves the use of specific rare cutting endonucleases, in particular TALE-nucleases (TAL effector endonuclease) and polynucleotides encoding such polypeptides, to precisely target a selection of key genes in T-cells, which are available from donors or from culture of primary cells. The invention opens the way to standard and affordable adoptive immunotherapy strategies for treating cancer and viral infections.

Claims (20)

1 - 28 . (canceled)

29 . A method of treatment comprising administering a population of isolated primary human T cells comprising at least 10 5 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

30 . The method of claim 29 , wherein the population of isolated primary human T cells has the CTLA4 alleles in the cells inactivated.

31 . The method of claim 29 , wherein the population of isolated primary human T cells has the TCRα alleles and the CD52 alleles in the cells inactivated.

32 . The method of claim 29 , wherein the population of isolated primary human T cells express a fragment of preTalpha sufficient to support CD3 surface expression.

33 . The method of claim 29 , wherein the population of isolated primary human T cells has the CD52 alleles in the cells inactivated.

34 . The method of claim 29 , wherein the population of isolated primary human T cells has the TCRα alleles in the cells inactivated.

35 . The method of claim 29 , wherein the population of isolated primary human T cells has both PD1 alleles in the cells inactivated.

36 . The method of claim 29 , wherein the population of isolated primary human T cells comprises at least 10 6 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

37 . The method of claim 30 , wherein the population of isolated primary human T cells comprises at least 10 6 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

38 . The method of claim 31 , wherein the population of isolated primary human T cells comprises at least 10 6 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

39 . The method of claim 33 , wherein the population of isolated primary human T cells comprises at least 10 6 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

40 . The method of claim 34 , wherein the population of isolated primary human T cells comprises at least 10 6 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

41 . The method of claim 35 , wherein the population of isolated primary human T cells comprises at least 10 6 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

42 . The method of claim 29 , wherein the population of isolated primary human T cells comprises at least 10 7 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

43 . The method of claim 30 , wherein the population of isolated primary human T cells comprises at least 10 7 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

44 . The method of claim 31 , wherein the population of isolated primary human T cells comprises at least 10 7 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

45 . The method of claim 33 , wherein the population of isolated primary human T cells comprises at least 10 7 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

46 . The method of claim 34 , wherein the population of isolated primary human T cells comprises at least 10 7 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.

47 . The method of claim 35 , wherein the population of isolated primary human T cells comprises at least 10 7 isolated primary human T cells that express a chimeric antigen receptor (CAR) comprising the amino acid sequence of SEQ ID NO:73 and have at least one PD1 allele inactivated.