IP Library Granted Patent US 7,166,763
Granted Patent B2
US 7,166,763 · App. 10/365,140 · Granted Jan 23, 2007

Mouse model of myxomatous valvular disease

Assignees: Trustees of Dartmouth College; Massachusetts Institute of Technology (MIT)
View Patent ↗
Loading inventors, assignments & file history…
Monitor This Case
Get email alerts when status or documents change.
Order Certified Copies
Most orders are placed with the USPTO same day — all within 24 business hours.
Order via The Patent Place →
Pre-filled with this patent's details
Quick Facts
Patent No.
US 7,166,763
App. No.
10/365,140
Granted
Jan 23, 2007
Kind
B2
Abstract

An animal selected for lacking heparan sulfate 3-O-sulfotransferase-1 activity is provided. This animal exhibits characteristics associated with myxomatous valvular disease and is useful for identifying agents which prevent, delay or treat myxomatous valvular disease. Methods of diagnosing myxomatous valvular disease are also provided.

Claims (10)

1. A method of producing a mouse model of myxomatous valvular disease comprising the steps of:

a) introducing a transgene containing 3-O-sulfotransferase-1nucleic acid sequences that flank a selectable marker gene into a mouse embryonic stem cell, wherein the transgene integrates into the genome of the mouse embryonic stem cell and the selectable marker gene disrupts the endogenous 3-O-sulfotransferase-1 gene, thereby producing a mouse embryonic stem cell wherein the endogenous 3-O-sulfotransferase-1 gene has been disrupted;

b) introducing the mouse embryonic stem cell wherein the endogenous 3-O-sulfotransferase-1 gene has been disrupted, into a mouse embryo;

c) implanting the resulting mouse embryo comprising the mouse ES cell of (b), into the uterus of a pseudopregnant mouse, wherein the pseudopregnant mouse gives birth to a chimeric mouse;

d) breeding the chimeric mouse to produce a mouse heterozygous for a disruption in the 3-O-sulfotransferase-1 gene;

e) backcrossing the heterozygous mouse for at least 10 backcrosses to produce a mouse that is heterozygous for the disrupted 3-O-sulfotransferase1 gene; and

f) crossing a first mouse obtained in step (e) with a second mouse obtained in step (e) to produce a homozygous knock-out mouse lacking heparan sulfate 3-O-sulfotransferase-1 activity and exhibiting mitral valve degeneration, indicative of myxomatous valvular disease.

2. A method of screening an agent for the treatment of myxomatous valvular disease comprising administering an agent to a homozygous knock-out mouse produced by step (f) of the method of claim 1 , and determining whether the agent at least partially abates at least one of the characteristics of myxomatous valvular disease in said mouse.

3. A method of screening for an agent that prevents or delays the development of myxomatous valvular disease comprising administering an agent to a homozygous knock-out mouse produced by step (f) of the method of claim 1 and determining whether the agent at least partially prevents or delays the age of development of at least one of the characteristics of myxomatous valvular disease in said mouse compared to the age development of said characteristic in an untreated mouse.

4. A mouse model of myxomatous valvular disease produced by step (f) of the method of claim 1 , wherein the mouse lacks heparan sulfate 3-O-sulfotransferase-1 activity and exhibits mitral valve degeneration, indicative of myxomatous valvular disease.

Assignments (3)
CONFIRMATORY LICENSE Recorded Apr 14, 2011
From: MASSACHUSETTS INSTITUTE OF TECHNOLOGY
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 026123/0992 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Oct 20, 2003
From: ROSENBERG, ROBERT D.
To: MASSACHUSETTS INSTITUTE OF TECHNOLOGY (MIT)
Reel/Frame 014600/0780 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Oct 20, 2003
From: SHWORAK, NICHOLAS W; PALAC, ROBERT T
To: TRUSTEES OF DARTMOUTH COLLEGE
Reel/Frame 014602/0689 →
Continuity (1)
Related Publication 20040158881A1 · Aug 12, 2004