IP Library Granted Patent US 7,829,283
Granted Patent B2
US 7,829,283 · App. 11/693,673 · Granted Nov 9, 2010

Methylation of estrogen receptor alpha and uses thereof

Assignee: John Wayne Cancer Institute
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Quick Facts
Patent No.
US 7,829,283
App. No.
11/693,673
Granted
Nov 9, 2010
Kind
B2
Abstract

Methods for diagnosis, prognosis, and treatment of cancer based on the methylation status of the ER-α gene promoter are disclosed. Methylation of the ER-α gene promoter is indicative of cancer and unfavorable prognosis. The cancer can be treated with a demethylation agent.

Claims (25)

1. A method of determining whether a human subject is suffering from melanoma cancer, comprising:

providing a serum-sample from a human subject suspected of having melanoma cancer;

isolating an acellular DNA sample from the serum sample;

determining the methylation level of the estrogen receptor-alpha (ER-α) gene promoter in the acellular DNA sample; and

determining that the human subject is suffering from melanoma cancer when the methylation level of the ER-α gene promoter in the acellular DNA sample is higher than a control methylation level.

2. The method of claim 1 , wherein the melanoma cancer is a primary or metastatic melanoma cancer.

3. A method of predicting survival in a human subject suffering from melanoma cancer, comprising:

providing a serum sample from a human subject having melanoma cancer;

isolating an acellular DNA sample from the serum sample;

determining the methylation level of the ER-α gene promoter in the acellular DNA sample; and

predicting a shorter survival when the methylation level of the ER-α gene promoter in the acellular DNA sample is higher than a control methylation level.

4. The method of claim 3 , wherein the shorter survival is a shorter progression-free survival or overall survival.

5. The method of claim 3 , wherein the melanoma cancer is a primary or metastatic melanoma cancer.

6. A method of predicting tumor progression in a human subject having melanoma cancer, comprising:

providing a melanoma cancer tumor tissue sample of the human subject having melanoma cancer;

isolating a cellular DNA sample from the melanoma cancer tumor tissue sample;

determining the methylation level of the ER-α gene promoter in the cellular DNA sample;

predicting a more advanced tumor progression when the methylation level of the ER-α gene promoter in the cellular DNA sample is higher than a control methylation level.

7. The method of claim 6 , wherein the melanoma cancer is a primary or metastatic melanoma cancer.

8. A method of predicting tumor progression in a human subject having melanoma cancer, comprising:

providing a serum sample from the human subject having melanoma cancer;

isolating an acellular DNA sample from the serum sample;

determining the methylation level of the ER-α gene promoter in the acellular DNA sample;

predicting a more advanced tumor progression when the methylation level of the ER-α gene promoter in the acellular DNA sample is higher than a control methylation level.

9. The method of claim 3 , further comprising predicting a poor response to biochemotherapy (BC) when the subject having melanoma cancer has a methylation level of the ER-α gene promoter in the sample DNA that is higher than a control methylation level.

Assignments (2)
CONFIRMATORY LICENSE Recorded Apr 19, 2017
From: JOHN WAYNE CANCER INSTITUTE
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 042094/0424 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Aug 21, 2007
From: HOON, DAVE S.B.; JOHN WAYNE CANCER INSTITUTE
To: JOHN WAYNE CANCER INSTITUTE
Reel/Frame 019723/0743 →
Continuity (2)
Provisional Application 6078771900 · Mar 29, 2006
Related Publication 20080299551A1 · Dec 4, 2008