IP Library Granted Patent US 9,598,488
Granted Patent B2
US 9,598,488 · App. 12/865,673 · Granted Mar 21, 2017

Blockage of interferon-gamma for prevention of polymicrobial synergy

Inventors: Dennis W. Metzger (Niskayuna, NY); Keer Sun (Latham, NY)
Assignee: Albany Medical College
C07K16/249A61K2039/505C07K2317/73C07K2317/77
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Quick Facts
Patent No.
US 9,598,488
App. No.
12/865,673
Granted
Mar 21, 2017
Kind
B2
Abstract

The present invention is directed to methods for treating a viral respiratory infection in a subject, preventing polymicrobial synergy in a subject, or preventing a bacterial infection in a subject. These methods include selecting a subject with a viral respiratory infection, a subject susceptible to polymicrobial synergy, or a subject susceptible to bacterial infection, respectively. In each case a therapeutic agent that inhibits interferon-gamma (IFNγ) is provided and administered to the selected subject under conditions effective to treat the viral respiratory infection, to prevent polymicrobial synergy, or to prevent a bacterial infection, respectively.

Claims (9)

1. A method of protecting a subject with a respiratory tract viral infection against developing a subsequent bacterial infection in the subject's lungs, said method comprising:

selecting a subject with a respiratory tract viral infection who is susceptible to bacterial infection in the lungs as a result of production of interferon-gamma (IFNγ) in the subject's lungs, said subject being in contact with said bacteria, wherein said bacteria is selected from the group consisting of S. pneumoniae, H. influenzae, S. aureus, M. catarrhalis, P. carinii, S. pyogenes , and P. aeruginosa ;

providing a therapeutic agent that inhibits IFNγ; and

administering the therapeutic agent to the selected subject under conditions effective to protect against a bacterial infection in the lungs as a result of production of interferon-gamma (IFNγ) in the subject's lungs, wherein said bacterial infection is caused by a bacteria selected from the group consisting of S. pneumoniae, H. influenzae, S. aureus, M. catarrhalis, P. carinii, S. pyogenes , and P. aeruginosa.

2. The method of claim 1 , wherein the selected subject is human.

3. The method of claim 1 , wherein said administering is carried out orally, intranasally, subcutaneously, intravenously, intramuscularly, parenterally, intraperitoneally, by application to the mucous membranes, or by inhalation.

4. The method according to claim 1 , wherein the therapeutic agent that inhibits IFNγ binds to IFNγ.

5. The method according to claim 1 , wherein the therapeutic agent that inhibits IFNγ binds to the IFNγ-receptor.

6. The method according to claim 1 , wherein the therapeutic agent is selected from the group consisting of an anti-IFNγ antibody or fragment thereof which binds to the IFNγ receptor, a soluble IFNγ receptor, a hybrid IFNγ receptor molecule, and an anti-IFNγ receptor antibody.

Assignments (2)
CONFIRMATORY LICENSE Recorded Apr 23, 2015
From: ALBANY MEDICAL COLLEGE
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 035496/0374 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Dec 28, 2010
From: METZGER, DENNIS W.; SUN, KEER
To: ALBANY MEDICAL COLLEGE
Reel/Frame 025543/0334 →
Continuity (2)
Provisional Application 61025623 · Feb 1, 2008
Related Publication 20110104104A1 · May 5, 2011