IP Library Granted Patent US 9,127,268
Granted Patent B2
US 9,127,268 · App. 13/514,952 · Granted Sep 8, 2015

Aminoacyl tRNA synthetases for modulating inflammation

Inventors: Jeffry Dean Watkins (Encinitas, CA); Alain Philippe Vasserot (Carlsbad, CA); Leslie Ann Greene (San Diego, CA); Ryan Andrew Adams (San Diego, CA); Kristi Helen Piehl (San Diego, CA); Fei Hong (San Diego, CA); Kyle P. Chiang (San Diego, CA); Wei Zhang (San Diego, CA); Alina He (San Diego, CA)
Assignee: aTyr Pharma, Inc.
C12N9/93A61K38/53A61K39/39541A61K45/06
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Quick Facts
Patent No.
US 9,127,268
App. No.
13/514,952
Granted
Sep 8, 2015
Kind
B2
Abstract

Inflammatory and other cellular response-modulating compositions are provided comprising aminoacyl-tRNA synthetase polypeptides, including active fragments and/or variants thereof. Also provided are methods of using such compositions in the treatment of conditions that benefit from the modulation of inflammation, such as inflammatory diseases or conditions.

Claims (49)

1. A method for treating an inflammatory or autoimmune condition in a subject in need thereof, comprising; administering to the subject a composition that comprises a pharmaceutically acceptable carrier and a histidyl-tRNA synthetase (HRS) polypeptide, where the HRS polypeptide comprises the amino acid sequence of SEQ ID NO:28 or a variant thereof that differs from SEQ ID NO:28 by 1-5 amino acids, where the HRS polypeptide has an anti-inflammatory activity and is at least about 90% pure, and where the composition is sterile and pyrogen-free.

2. The method of claim 1 , where the HRS polypeptide is about 509 amino acids in length.

3. The method of claim 2 , where the HRS polypeptide consists essentially of the amino acid sequence of SEQ ID NO:28.

4. The method of claim 1 , where the HRS polypeptide is modified by pegylation.

5. The method of claim 1 , where the HRS polypeptide is a fusion protein that further comprises a heterologous fusion partner.

6. The method of claim 5 , where the HRS polypeptide fusion protein comprises a linker.

7. The method of claim 5 , where the HRS polypeptide fusion protein comprises a heterologous signal peptide.

8. The method of claim 5 , where the heterologous fusion partner comprises an Fc fragment.

9. The method of claim 1 , where the HRS polypeptide is a recombinant polypeptide.

10. The method of claim 1 , where the HRS polypeptide is a synthetic polypeptide.

11. The method of claim 1 , where the composition is formulated with histidine.

12. The method of claim 1 , where the composition comprises a surfactant.

13. The method of claim 1 , where the composition is administered by oral, parenteral, intravenous, intranasal, inhalation, aerosol, intracranial, or intramuscular administration.

14. The method of claim 1 , further comprising administering an anti-inflammatory agent, or immunosuppressive agent.

15. The method of claim 14 , where the anti-inflammatory agent is selected from non-steroidal anti-inflammatory drugs (NSAIDs), immune selective anti-inflammatory derivatives (ImSAIDs), steroids, anti-oxidants, and cytokines.

16. The method of claim 1 , where the inflammatory condition is acute.

17. The method of claim 1 , where the inflammatory condition is chronic.

18. The method of claim 1 , where the inflammatory or autoimmune condition is associated with the vascular system.

19. The method of claim 1 , where the inflammatory or autoimmune condition is associated with the musculoskeletal system.

20. The method of claim 1 , where the inflammatory or autoimmune condition is associated with the respiratory system.

21. The method of claim 1 , where the inflammatory condition or autoimmune is associated with the gastrointestinal system.

22. The method of claim 1 , where the inflammatory condition or autoimmune is associated with the skin.

23. The method of claim 19 , where the inflammatory or autoimmune condition of the musculoskeletal system is selected from auto-immune myositis, Sjogren's syndrome, smooth muscle auto-immune disease, myositis, polymyositis, scleroderma, and chronic muscle inflammation.

24. The method of claim 20 , where the inflammatory or autoimmune condition of the respiratory system is selected from inflammatory lung disease and COPD.

25. The method of claim 21 , where the inflammatory or autoimmune condition of the gastrointestinal system is selected from inflammatory bowel disease (IBD) and colitis.

26. The method of claim 22 , where the inflammatory or autoimmune condition of the skin is selected from irritant dermatitis, allergic contact dermatitis, drug-induced dermatitis, dermatomyositis, pemphigus, and autoimmune dermatitis.

27. A method for treating an inflammatory or autoimmune condition in a subject in need thereof, comprising; a) administering to the subject a composition that comprises a pharmaceutically acceptable carrier and a histidyl-tRNA synthetase (HRS) polypeptide, where the HRS polypeptide comprises the amino acid sequence of SEQ ID NO:28 or a variant thereof that differs from SEQ ID NO:28 by 1-5 amino acids, where the HRS polypeptide has an anti-inflammatory activity and is at least about 90% pure, and where the composition is sterile and pyrogen-free; and b) administering an anti-inflammatory or immunosuppressive therapy.

28. The method of claim 27 , wherein the anti-inflammatory therapy comprises administration of an anti-inflammatory agent selected from a non-steroidal anti-inflammatory drug (NSAIDs), immune selective anti-inflammatory derivatives (ImSAIDs), steroids, anti-oxidants, and cytokines.

29. The method of claim 27 , where the HRS polypeptide is modified by pegylation.

30. The method of claim 27 , where the HRS polypeptide further comprises a heterologous fusion partner.

31. The method of claim 30 , where the HRS polypeptide fusion protein comprises a linker.

32. The method of claim 30 , where the HRS polypeptide fusion protein comprises a heterologous signal peptide.

33. The method of claim 30 , where the heterologous fusion partner comprises an Fc fragment.

34. The method of claim 27 , where the HRS polypeptide is a recombinant polypeptide.

35. The method of claim 27 , where the HRS polypeptide is a synthetic polypeptide.

36. The method of claim 27 , where the composition is formulated with histidine.

37. The method of claim 27 , where the composition comprises a surfactant.

38. The method of claim 27 , where the composition is administered by oral, parenteral, intravenous, intranasal, inhalation, aerosol, intracranial, or intramuscular administration.

39. The method of claim 27 , where the inflammatory condition is acute.

40. The method of claim 27 , where the inflammatory condition is chronic.

41. The method of claim 27 , where the inflammatory or autoimmune condition is associated with the vascular system.

42. The method of claim 27 , where the inflammatory or autoimmune condition is associated with the musculoskeletal system.

43. The method of claim 27 , where the inflammatory or autoimmune condition is associated with the respiratory system.

44. The method of claim 27 , where the inflammatory condition or autoimmune is associated with the gastrointestinal system.

45. The method of claim 27 , where the inflammatory condition or autoimmune is associated with the skin.

46. The method of claim 42 , where the inflammatory or autoimmune condition of the musculoskeletal system is selected from auto-immune myositis, Sjogren's syndrome, smooth muscle auto-immune disease, myositis, polymyositis, scleroderma, and chronic muscle inflammation.

47. The method of claim 43 , where the inflammatory or autoimmune condition of the respiratory system is selected from inflammatory lung disease and COPD.

48. The method of claim 44 , where the inflammatory or autoimmune condition of the gastrointestinal system is selected from inflammatory bowel disease (IBD) and colitis.

49. The method of claim 45 , where the inflammatory or autoimmune condition of the skin is selected from irritant dermatitis, allergic contact dermatitis, drug-induced dermatitis, dermatomyositis, pemphigus, and autoimmune dermatitis.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Sep 17, 2012
From: WATKINS, JEFFRY DEAN; VASSEROT, ALAIN PHILIPPE; GREENE, LESLIE ANN; ADAMS, RYAN ANDREW; CHAING, KYLE B.; ZHANG, WEI; PIEHL, KRISTI HELEN; HONG, FEI; HE, ALINA
To: ATYR PHARMA, INC.
Reel/Frame 028970/0752 →
Continuity (4)
Provisional Application 61285913 · Dec 11, 2009
Provisional Application 61285923 · Dec 11, 2009
Provisional Application 61285919 · Dec 11, 2009
Related Publication 20130108630A1 · May 2, 2013