IP Library Granted Patent US 9,568,483
Granted Patent B2
US 9,568,483 · App. 14/112,358 · Granted Feb 14, 2017

Molecular subtyping, prognosis and treatment of prostate cancer

Inventors: Mark A. Rubin (New York, NY); Himisha Beltran (New York, NY)
Assignee: CORNELL UNIVERSITY
G01N33/6893A61K31/00A61K31/437A61K31/444A61K31/496A61K31/506A61K31/519A61K31/5377A61K31/55A61K31/675A61K31/713C12Q1/6886G01N33/57434C12Q2600/112C12Q2600/156C12Q2600/158G01N2333/82G01N2333/91215
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Quick Facts
Patent No.
US 9,568,483
App. No.
14/112,358
Granted
Feb 14, 2017
Kind
B2
Abstract

Disclosed herein are new prognostic molecular markers for prostate cancer. More specifically, the invention has identified that overexpression or amplification of at least one of AURKA or MYCN define a distinct subgroup of prostate cancer that is predisposed to the development of lethal NEPC, who will benefit from early intervention.

Claims (13)

1. A method of subtyping prostate cancer, comprising:

obtaining a biological sample containing prostate cancer cells from a human subject having prostate cancer,

detecting amplification of AURKA in the biological sample by a fluorescent in situ hybridization (FISH) assay,

subtyping the prostate cancer as neuroendocrine prostate cancer (NEPC) or likely to develop into NEPC based on presence of the amplification of AURKA in the biological sample; and

administering a small molecule Aurora A kinase inhibitor to the human subject for treating prostate cancer subtyped as having NEPC or likely to develop NEPC.

2. The method of claim 1 , wherein said biological sample is a prostate tissue biopsy, urine, blood, semen, or prostatic secretions.

3. The method of claim 1 , further comprising detecting ERG rearrangement.

4. The method of claim 3 , wherein said ERG rearrangement is detected in a break-apart FISH assay.

5. The method of claim 1 , wherein said human subject has not been subjected to hormone therapy.

6. The method of claim 1 , wherein said human subject is undergoing hormone therapy.

7. The method of claim 1 , wherein said small molecule Aurora A kinase inhibitor is selected from the group consisting of VX-680/MK-0457, PHA-739358, MLN8054, MLN8237, SNS-314CYC116, PF-3814735, ENMD2076, AT-9283, R-763/AS-703569, and AMG900.

8. The method of claim 1 , wherein said amplification is determined based on a gain of at least one in the copy number of the AURKA gene.

9. The method of claim 8 , wherein said gain of at least one in the copy number of the AURKA gene is determined based on detecting at least three fluorescent signals in an interphase nucleus using a fluorescently labeled nucleic acid probe specific for the AURKA gene.

Assignments (2)
CONFIRMATORY LICENSE Recorded Apr 20, 2017
From: CORNELL UNIVERSITY
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 042286/0783 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Oct 17, 2013
From: RUBIN, MARK A.; BELTRAN, HIMISHA
To: CORNELL UNIVERSITY
Reel/Frame 031425/0604 →
Continuity (4)
Provisional Application 61476628 · Apr 18, 2011
Provisional Application 61490441 · May 26, 2011
Provisional Application 61544905 · Oct 7, 2011
Related Publication 20140037647A1 · Feb 6, 2014