IP Library Granted Patent US 9,289,448
Granted Patent B2
US 9,289,448 · App. 14/216,065 · Granted Mar 22, 2016

Compositions and methods for treating alzheimer's disease and other tauopathies

Inventors: Bingwei Lu (Stanford, CA); Seongsoo Lee (Stanford, CA)
Assignee: Board of Trustees of the Leland Stanford Junior University
A61K31/713A61K31/44A61K48/00
View Patent ↗
Loading inventors, assignments & file history…
Monitor This Case
Get email alerts when status or documents change.
Order Certified Copies
Most orders are placed with the USPTO same day — all within 24 business hours.
Order via The Patent Place →
Pre-filled with this patent's details
Quick Facts
Patent No.
US 9,289,448
App. No.
14/216,065
Granted
Mar 22, 2016
Kind
B2
Abstract

Compounds, pharmaceutical compositions, and methods of using such compounds and compositions to treat or prevent Alzheimer's disease and related tauopathies through the inhibition of USP-9X and/or enhancement of SCF(β-TrCP).

Claims (28)

1. A composition for treating Alzheimer's disease, the composition comprising a USP-9x inhibitor having the structure:

2. The composition of claim 1 , further comprising a pharmaceutically acceptable carrier.

3. The composition of claim 1 , wherein the USP-9x inhibitor is present in a therapeutically effective amount.

4. The composition of claim 1 , wherein the USP-9x inhibitor is present in a prophylactically effective amount.

5. The composition of claim 1 , wherein the SCF(β-TrCP) enhancer is present in a therapeutically effective amount.

6. The composition of claim 1 , wherein the SCF(β-TrCP) enhancer is present in a prophylactically effective amount.

7. The composition of claim 1 , wherein the SCF(β-TrCP) enhancer is a gene therapy.

8. The composition of claim 1 , wherein the SCF(β-TrCP) enhancer increases the neddylation of Cullin.

9. The composition of claim 8 , wherein the SCF(β-TrCP) enhancer increases the expression or activity of NEDD8 or NAE.

10. The composition of claim 8 , wherein the SCFM(β-TrCP) enhancer is a gene therapy that increases expression of NEDD8 or NAE.

11. The composition of claim 8 , wherein the SCFM(β-TrCP) enhancer is a small molecule activator of NAE.

12. The composition of claim 8 , wherein the SCFM(β-TrCP) enhancer increases activity and/or expression of CAND1.

13. The composition of claim 8 , wherein the SCFM(β-TrCP) enhancer increases expression of CAND1 by a gene therapy.

14. The composition of claim 8 , wherein the SCFM(β-TrCP) enhancer increases activity of CAND1 through administration of a small molecule activator of CAND1.

15. The composition of claim 8 , wherein the SCFM(β-TrCP) enhancer decreases Cullin deneddylation.

16. The composition of claim 15 , wherein the SCFM(β-TrCP) enhancer inhibits CSN.

17. The composition of claim 15 , wherein the SCFM(β-TrCP) enhancer is RNAi targeting CSN.

18. The composition of claim 17 , wherein the RNAi is miRNA targeting CSN.

19. The composition of claim 17 , wherein the RNAi is sRNA targeting CSN.

20. The composition of claim 17 , wherein, the RNAi is shRNA targeting CSN.

21. The composition of claim 17 , wherein the SCFM(β-TrCP) enhancer is a small molecule inhibitor of CSN.

22. A kit comprising a USP-9x inhibitor having the structure:

a SCFM(β-TrCP) enhancer; and

a pharmaceutically acceptable carrier thereof.

23. The kit of claim 22 , wherein the inhibitor and the enhancer are coformulated.

24. The kit of claim 22 , wherein the inhibitor and the enhancer are copackaged.

25. The kit of claim 22 , further comprising instructions for treatment of Alzheimer's disease.

26. The composition of claim 1 , wherein the composition is effective in treating tauopathies selected from the group consisting of: Creutzfeldt-Jakob disease, dementia pugilistica, Down's syndrome, Gerstmann-Straussler-Sheinker disease, inclusion-body myositis, frontotemporal dementia (FTD), Pick's disease, tangle-predominant Alzheimer's disease, corticobasal degeneration, amyotrophic lateral sclerosis/parkinsonism-dementia complex, argyrophilic grain dementia, diffuse neurofibrillary tangles with calcification, Hallevorden-Spatz disease, multiple system atrophy, Niemann-Pick disease, Type C1 progressive subcortial gliosis, progressive supranuclear palsy and subacute sclerosing panencephalitis.

Assignments (2)
CONFIRMATORY LICENSE Recorded Sep 8, 2014
From: THE BOARD OF TRUSTEES OF THE LELAND STANFORD JUNIOR UNIVERSITY
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 033695/0324 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jun 18, 2014
From: LU, BINGWEI; LEE, SEONGSOO
To: THE BOARD OF TRUSTEES OF THE LELAND STANFORD JUNIOR UNIVERSITY
Reel/Frame 033124/0790 →
Continuity (2)
Provisional Application 61792009 · Mar 15, 2013
Related Publication 20150174150A1 · Jun 25, 2015