IP Library Granted Patent US 10,760,065
Granted Patent B2
US 10,760,065 · App. 14/475,785 · Granted Sep 1, 2020

Tuning microbial populations with programmable nucleases

Inventors: Timothy Kuan-Ta Lu (Charlestown, MA); Robert James Citorik (Kingston, NH); Mark Kyle Mimee (Cambridge, MA)
Assignee: Massachusetts Institute of Technology
C12N9/22A61K38/465C12N15/74
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Quick Facts
Patent No.
US 10,760,065
App. No.
14/475,785
Granted
Sep 1, 2020
Kind
B2
Abstract

Various aspects and embodiments of the invention are directed to methods and compositions for reversing antibiotic resistance or virulence in and/or destroying pathogenic microbial cells such as, for example, pathogenic bacterial cells. The methods include exposing microbial cells to a delivery vehicle with at least one nucleic acid encoding an engineered autonomously distributed circuit that contains a programmable nuclease targeted to one or multiple genes of interest.

Claims (23)

1. A method of detecting a nucleic acid of interest in a viable bacterial cell, comprising delivering to a human subject at least one delivery vehicle that comprises:

(a) a nucleic acid comprising a promoter operably linked to nucleotide sequences encoding a programmable nuclease circuit that induces a double-strand break in the nucleic acid of interest in the bacterial cell in the human subject;

(b) a nucleic acid comprising a RecA promoter or a LexO promoter operably linked to a gene encoding a reporter molecule, wherein the RecA promoter or the LexO promoter activates expression of the reporter molecule in the presence of the double-strand break; and

(c) detecting presence or absence of the nucleic acid of interest based on expression of the reporter molecule in the viable bacterial cell.

2. The method of claim 1 , wherein step (c) comprises detecting transcription or translation of the reporter molecule.

3. The method of claim 1 , wherein the programmable nuclease circuit comprises an RNA-guided nuclease.

4. The method of claim 3 , wherein the programmable nuclease circuit comprises a Cas9 nuclease, a guide RNA (gRNA), and/or a transactivating small RNA (tracrRNA) derived from the Type II CRISPR-Cas system of Streptococcus pyogenes.

5. The method of claim 4 , wherein the gRNA and the tracrRNA form a chimeric single-guide RNA molecule.

6. The method of claim 1 , wherein the programmable nuclease circuit comprises a transcriptional activator like (TAL)-effector nuclease (TALEN) or a zinc finger nuclease (ZFN).

7. The method of claim 1 , wherein the delivery vehicle is a bacteriophage.

8. The method of claim 1 , wherein the delivery vehicle is a phagemid.

9. The method of claim 1 , wherein the delivery vehicle is a donor bacterial cell.

10. The method of claim 9 , wherein the donor bacterial cell is a commensal bacterial cell.

11. The method of claim 10 , wherein the commensal bacterial cell is loaded with at least one conjugative plasmid containing the nucleic acid of (a) and/or (b).

12. The method of claim 11 , wherein the at least one conjugative plasmid comprises a broad-host-range origin of replication.

13. The method of claim 1 , wherein the bacterial cells are pathogenic bacterial cells.

14. The method of claim 13 , wherein the pathogenic bacterial cells are selected from extended-spectrum beta-lactamase-producing (ESBL) Escherichia coli , ESBL Klebsiella pneumonia, Pseudomonas aeruginosa , vancomycin-resistant Enterococcus (VRE), methicillin-resistant Staphylococcus aureus (MRSA), multidrug-resistant (MDR) Acinetobacter baumannii , and MDR Enterobacter spp.

15. The method of claim 1 , wherein the nucleic acid of interest includes a mutation.

16. The method of claim 15 , wherein the mutation is a single nucleotide polymorphism (SNP).

17. The method of claim 1 , wherein the nucleic acid of interest comprises a virulence factor gene, a toxin gene, an antibiotic resistance gene, a remodeling gene or a modulatory gene.

18. The method of claim 17 , wherein the nucleic acid of interest comprises an antibiotic resistance gene that confers aminoglycoside resistance, beta-lactam resistance, daptomycin resistance, macrolide-lincosamide-streptogramin B resistance, quinolone resistance, trimethoprim/sulfonamide resistance or vancomycin resistance.

19. The method of claim 1 , wherein the promoter of (a) is an inducible promoter.

20. The method of claim 1 , wherein the reporter molecule is a fluorescent protein.

Assignments (2)
CONFIRMATORY LICENSE Recorded Jun 22, 2016
From: MASSACHUSETTS INSTITUTE OF TECHNOLOGY
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 039113/0891 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded May 26, 2015
From: LU, TIMOTHY KUAN-TA; CITORIK, ROBERT JAMES; MIMEE, MARK KYLE
To: MASSACHUSETTS INSTITUTE OF TECHNOLOGY
Reel/Frame 035713/0102 →
Continuity (3)
Provisional Application 62010976 · Jun 11, 2014
Provisional Application 61873894 · Sep 5, 2013
Related Publication 20150064138A1 · Mar 5, 2015
Cited By (6)
US 12,226,430 US 12,318,445 US 12,448,619 US 12,502,401 US 12,514,867 US 12,514,869