IP Library Granted Patent US 10,663,462
Granted Patent B2
US 10,663,462 · App. 14/592,611 · Granted May 26, 2020

Method of treating vascular insulin resistance in a normoglycemic subject based on biomarkers

Inventors: Andreas Pfuetzner (Mainz, DE); Thomas Forst (Mainz, DE)
G01N33/54306A61K31/426A61K38/28G01N33/53G01N33/74G01N2333/4737G01N2333/62G01N2800/042G01N2800/52
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Quick Facts
Patent No.
US 10,663,462
App. No.
14/592,611
Granted
May 26, 2020
Kind
B2
Abstract

The invention provides compositions and methods for determining insulin resistance and/or pancreatic β-cell dysfunction in a subject. The invention also provides compositions and methods for treating a subject according to the insulin resistance and/or pancreatic β-cell dysfunction in the subject.

Claims (17)

1. A method of treating vascular insulin resistance in a normoglycemic subject at high risk for myocardial infarction comprising

(a) measuring the concentration of a biomarker panel in a sample from the subject, the biomarker panel consisting of adiponectin, C-peptide, insulin and intact proinsulin; and

(b) effecting a therapy with respect to the subject by administering a therapeutically effective amount of gliatozone and an insulin-modulating drug, thereby treating vascular insulin resistance in the normoglycemic subject.

2. The method of claim 1 wherein the subject is not administered a drug or combination of drugs selected from a sulfonylurea and a glinide.

3. The method of claim 1 wherein the subject is further administered one or more additional drugs comprising one or more glucose-lowering drugs.

4. The method of claim 1 wherein the sample comprises blood.

5. The method of claim 1 further comprising taking a measurement of at least one additional biomarker.

6. The method of claim 5 wherein the additional biomarker is selected from the group consisting of leptin, NFκB, IL-6, MMP-9, TNFα, eNOS, PPARγ, MCP-1, PAI-1, ICAM/VCAM, E-selectin, P-selectin, von Willebrand factor, sCD40L, insulin, glucose, HbA1c, free fatty acids, triglycerides, VLDL, small dense LDL, oxidized LDL, resistin, HDL, NO, IκB-α, p105, RelA, MIF, inflammatory cytokines and molecules involved in signaling pathways.

7. The method of claim 1 wherein the insulin-modulating drug is selected from the group consisting of metformin, a glucagon-like peptide 1 (GLP-1) analog, a dipeptidyl peptidase IV (DPPIV) inhibitor, insulin, and an insulin analog.

8. A method of treating vascular insulin resistance in a normoglycemic subject at high risk for myocardial infarction and stroke comprising

(a) measuring the concentration of a biomarker panel in a sample from the subject, the biomarker panel consisting of adiponectin, C-peptide, insulin, intact proinsulin, triglycerides, and HDL; and

(b) effecting a therapy with respect to the subject by administering a therapeutically effective amount of gliatozone and an insulin-modulating drug, thereby treating vascular insulin resistance in the normoglycemic subject.

9. The method of claim 8 wherein the insulin-modulating drug is selected from the group consisting of metformin, a glucagon-like peptide 1 (GLP-1) analog, a dipeptidyl peptidase IV (DPPIV) inhibitor, insulin, and an insulin analog.

10. The method of claim 8 wherein the subject is further administered one or more additional drugs comprising one or more glucose-lowering drugs.

11. The method of claim 8 wherein the sample comprises blood.

12. The method of claim 8 further comprising taking a measurement of at least one additional biomarker.

13. The method of claim 12 wherein the additional biomarker is selected from the group consisting of leptin, NFκB, IL-6, MMP-9, TNFα, eNOS, PPARγ, MCP-1, PAI-1, ICAM/VCAM, E-selectin, P-selectin, von Willebrand factor, sCD40L, insulin, glucose, HbA1c, free fatty acids, VLDL, small dense LDL, oxidized LDL, resistin, NO, IκB-α, IκB-β, p105, RelA, MIF, inflammatory cytokines and molecules involved in signaling pathways.

Continuity (4)
Continuation 13670273 · Nov 6, 2012
Continuation 12505367 · Jul 17, 2009
Provisional Application 61081647 · Jul 17, 2008
Related Publication 20150219639A1 · Aug 6, 2015