IP Library Patent Application 14766105
Patent Application
App. No. 14/766,105

CHIMERIC ANTIGEN RECEPTOR AND METHODS OF USE THEREOF

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Patent No.
US None
App. No.
14/766,105
Abstract

The present disclosure provides a heterodimeric, conditionally active chimeric antigen receptor (CAR), and a nucleic acid comprising a nucleotide sequence encoding the CAR. The present disclosure provides cells genetically modified to produce the CAR. A CAR of the present disclosure can be used in various methods, which are also provided.

Claims (71)

1 . A heterodimeric, conditionally active chimeric antigen receptor (CAR) comprising:

a) a first polypeptide comprising:

i) a first member of a specific binding pair;

ii) a first modulatory domain;

iii) a first member of a dimerization pair; and

iv) a transmembrane domain interposed between the first member of a specific binding pair and the first modulatory domain; and

b) a second polypeptide comprising:

i) a transmembrane domain;

ii) a second modulatory domain;

iii) a second member of the dimerization pair; and

iv) an intracellular signaling domain;

or comprising:

a) a first polypeptide comprising:

i) a first member of a specific binding pair;

ii) a modulatory domain;

iii) a first member of a dimerization pair;

iv) a transmembrane domain interposed between the first member of a specific binding pair and the modulatory domain; and

b) a second polypeptide comprising:

i) a second member of the dimerization pair; and

ii) an intracellular signaling domain.

2 . The heterodimeric, conditionally active CAR of claim 1 , wherein the first polypeptide comprises a hinge region interposed between the first member of the specific binding pair and the transmembrane domain.

3 . The heterodimeric, conditionally active CAR of claim 1 , wherein the first member of the specific binding pair is an antibody or antibody fragment, a ligand, or a receptor.

4 . The heterodimeric, conditionally active CAR of claim 2 , wherein the hinge region is an immunoglobulin IgG hinge region or a hinge derived from CD8.

5 . The heterodimeric, conditionally active CAR of claim 1 , wherein the first and second modulatory domains are selected from 4-1BB (CD137), CD28, ICOS, BTLA, OX-40, CD27, CD30, GITR, HVEM, DAP10, DAP12, and CD28.

6 . The heterodimeric, conditionally active CAR of claim 1 , wherein the intracellular signaling domain is selected from ZAP70 and CD3-zeta.

7 . The heterodimeric, conditionally active CAR of claim 1 , wherein the intracellular signaling domain comprises an immunoreceptor tyrosine-based activation motif (ITAM).

8 . The heterodimeric, conditionally active CAR of claim 1 , wherein the first and second members of the dimerization pair form a homodimer in the presence of a small molecule dimerizer.

9 . The heterodimeric, conditionally active CAR of claim 1 , wherein the first and second members of the dimerization pair form a heterodimer in the presence of a small molecule dimerizer.

10 . The heterodimeric, conditionally active CAR of claim 1 , wherein the first and second members of the dimerization pair are selected from:

a) FK506 binding protein (FKBP) and FKBP;

b) FKBP and calcineurin catalytic subunit A (CnA);

c) FKBP and cyclophilin;

d) FKBP and FKBP-rapamycin associated protein (FRB);

e) gyrase B (GyrB) and GyrB;

f) dihydrofolate reductase (DHFR) and DHFR;

g) DmrB and DmrB;

h) PYL and ABI;

i) Cry2 and CIP;

j) GAI and GID1.

11 . The heterodimeric, conditionally active CAR of claim 1 , wherein:

i) the first and second modulatory domains are derived from 4-1BB;

ii) the first and second members of the dimerization pair are FKBP and FRB; and

ii) the signaling domain comprises an ITAM.

12 . The heterodimeric, conditionally active CAR of claim 1 , wherein the first member of the specific binding pair is a single-chain Fv.

13 . The heterodimeric, conditionally active CAR of claim 1 , wherein the first member of the specific binding pair binds an epitope present on a cell, on a solid surface, or a lipid bilayer.

14 . The heterodimeric, conditionally active CAR of claim 13 , wherein the cell is a cancer cell.

15 . A mammalian cell genetically modified to produce the heterodimeric, conditionally active CAR of claim 1 .

16 . The cell of claim 15 , wherein the cell is a stem cell, a progenitor cell, or a cell derived from a stem cell or a progenitor cell.

17 . The cell of claim 15 , wherein the cell is a T lymphocyte or an NK cell.

18 . A nucleic acid comprising nucleotide sequences encoding the heterodimeric, conditionally active CAR of claim 1 .

19 . The nucleic acid of claim 18 , wherein the nucleotide sequences are operably linked to a T lymphocyte-specific promoter or an NK cell-specific promoter.

20 . The nucleic acid of claim 18 , wherein the nucleic acid is in vitro transcribed RNA.

21 . A recombinant expression vector comprising the nucleic acid of claim 18 .

22 . A method of activating a T lymphocyte, the method comprising contacting the T lymphocyte with a dimerizing agent and a second member of a specific binding pair, wherein the T lymphocyte is genetically modified to produce a heterodimeric, conditionally active CAR of claim 1 , and wherein, in the presence of the dimerizing agent and the second member of a specific binding pair, the heterodimeric, conditionally active CAR dimerizes and activates the T lymphocyte, thereby producing an activated T lymphocyte.

23 . The method of claim 22 , wherein the second member of a specific binding pair is an antigen.

24 . The method of claim 22 , wherein said contacting occurs in vivo.

25 . The method of claim 22 , wherein the activated T lymphocyte mediates killing of a target cell.

26 . The method of claim 22 , wherein the activated T lymphocyte produces IL-2 and/or IFN-γ.

27 . The method of claim 25 , wherein the target cell is a cancer cell.

28 . The method of claim 22 , wherein the first member of the specific binding pair of the heterodimeric, conditionally active CAR is an antibody specific for an epitope on a cancer cell.

29 . A method of making the cell of claim 15 , the method comprising genetically modifying a mammalian cell with an expression vector comprising nucleotide sequences encoding the heterodimeric, conditionally active CAR of claim 1 , or genetically modifying a mammalian cell with an RNA comprising nucleotide sequences encoding the heterodimeric, conditionally active CAR of claim 1 .

30 . The method of claim 29 , wherein said genetic modification is carried out ex vivo.

31 . The method of claim 29 , wherein the cell is a T lymphocyte, a stem cell, an NK cell, a progenitor cell, a cell derived from a stem cell, or a cell derived from a progenitor cell.

32 . A method of treating a cancer in an individual, the method comprising:

i) genetically modifying T lymphocytes obtained from the individual with an expression vector comprising nucleotide sequences encoding the heterodimeric, conditionally active CAR of claim 1 , wherein the antigen-binding domain of the heterodimeric, conditionally active CAR is specific for an epitope on a cancer cell in the individual, and wherein said genetic modification is carried out ex vivo;

ii) introducing the genetically modified T lymphocytes into the individual; and

iii) administering to the individual an effective amount of a dimerizing agent, wherein the dimerizing agent induces dimerization of the heterodimeric, conditionally active CAR, wherein said dimerization provides for activation of the genetically modified T lymphocytes and killing of the cancer cell, thereby treating the cancer.

33 . The method of claim 32 , wherein the dimerizing agent is a rapalog.

34 . A method of modulating the activity of a host cell, the method comprising contacting the host cell with a dimerizing agent and a second member of a specific binding pair, wherein the T lymphocyte is genetically modified to produce a heterodimeric, conditionally active CAR of claim 1 , and wherein, in the presence of the dimerizing agent and the second member of a specific binding pair, the heterodimeric, conditionally active CAR dimerizes and modulates at least one activity of the host cell.

35 . The method of claim 34 , wherein the activity is proliferation, cell survival, apoptosis, gene expression, or immune activation.

36 . The method of claim 34 , wherein the second member of a specific binding pair is an antigen.

Assignments (2)
CONFIRMATORY LICENSE Recorded Sep 30, 2015
From: UNIVERSITY OF CALIFORNIA, SAN FRANCISCO
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 036732/0570 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Aug 7, 2015
From: WU, CHIA-YUNG; ONUFFER, JAMES; LIM, WENDELL A.
To: THE REGENTS OF THE UNIVERSITY OF CALIFORNIA
Reel/Frame 036281/0429 →