IP Library Granted Patent US 9,670,259
Granted Patent B2
US 9,670,259 · App. 14/775,936 · Granted Jun 6, 2017

Method of inhibiting angiogenesis using inhibitors of G-protein-coupled receptor kinase interacting protein-1 (GIT1)

Inventors: Bradford C. Berk (Pittsford, NY); Jinjiang Pang (Rochester, NY); Syamantak Majumder (Rochester, NY)
Assignee: University of Rochester
C07K14/4722A61K38/10A61K38/16A61K38/1709A61K39/3955A61K47/48346C07K16/28
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Quick Facts
Patent No.
US 9,670,259
App. No.
14/775,936
Granted
Jun 6, 2017
Kind
B2
Abstract

The present invention is directed to methods and compositions comprising G-protein-coupled receptor kinase interacting protein-1 (GIT1) inhibitors that are suitable for inhibiting angiogenesis in a subject and treating related conditions.

Claims (15)

1. A method of inhibiting angiogenesis in a subject comprising:

selecting a subject having a condition associated with abnormal angiogenesis and

administering a G-protein-coupled receptor kinase interacting protein-1 (GIT1) inhibitor to the selected subject, wherein the GIT1 inhibitor is (i) an antibody or antigen-binding portion thereof that binds to at least a portion of an amino acid sequence of SEQ ID NO: 8, or (ii) a peptide comprising the amino acid sequence of SEQ ID NO: 17 or SEQ ID NO: 18, under conditions effective to inhibit angiogenesis in the selected subject.

2. The method of claim 1 , wherein the condition associated with abnormal angiogenesis is selected from the group consisting of cancer, macular degeneration, and pulmonary arterial hypertension.

3. The method of claim 1 , wherein the at least a portion of the amino acid sequence of SEQ ID NO: 8 bound by the antibody or antigen-binding portion thereof comprises an ankyrin repeat domain sequence (TPLH) of SEQ ID NO: 8.

4. The method of claim 1 , wherein the antibody or antigen-binding portion thereof binds to at least a portion of an amino acid sequence selected from the group consisting of SEQ ID NO: 2, SEQ ID NO:3, SEQ ID NO:4, SEQ ID NO:5, and SEQ ID NO: 6.

5. The method of claim 1 , wherein the GIT1 inhibitor is administered via a delivery vehicle.

6. The method of claim 5 , wherein the delivery vehicle is selected from the group consisting of viral vectors, biodegradable microspheres, microparticles, nanoparticles, liposomes, collagen minipellets, and cochleates.

7. The method of claim 6 , wherein the delivery vehicle is a nanoparticle.

8. The method of claim 1 , wherein the GIT1 inhibitor is coupled to an endothelial cell-targeting moiety.

9. The method of claim 8 , wherein the endothelial cell-targeting moiety is an endothelial cell surface receptor ligand selected from the group consisting of vascular endothelial growth factor (VEGF), fibroblast growth factor (FGF), integrin, fibronectin, intracellular adhesion molecule (ICAM), platelet derived growth factor (PDGF), P-selectin ligand, vascular cell adhesion molecule (VCAM-1) ligand, and ICAM-1 ligand.

10. The method of claim 8 , wherein the endothelial cell-targeting moiety comprises a tripeptide sequence of Arg-Gly-Asp (RGD) or Asn-Gly-Arg (NGR).

11. The method of claim 1 , wherein the GIT1 inhibitor is administered via a nanoparticle delivery vehicle coupled to an endothelial cell-targeting moiety.

12. The method of claim 1 , wherein the GIT1 inhibitor is coupled to a cell-penetrating moiety.

13. The method of claim 12 , wherein the cell-penetrating moiety comprises a trans-activator of transcription (TAT) peptide.

Assignments (2)
CONFIRMATORY LICENSE Recorded Oct 16, 2017
From: UNIVERSITY OF ROCHESTER
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 044209/0355 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Apr 26, 2016
From: BERK, BRADFORD C.; PANG, JINJIANG; MAJUMDER, SYAMANTAK
To: UNIVERSITY OF ROCHESTER
Reel/Frame 038569/0377 →
Continuity (2)
Provisional Application 61781832 · Mar 14, 2013
Related Publication 20160024163A1 · Jan 28, 2016