IP Library Granted Patent US 10,993,966
Granted Patent B2
US 10,993,966 · App. 15/413,086 · Granted May 4, 2021

Pluripotent stem cell for treatment of cerebral infarction

Inventors: Masanori Yoshida (Akita, JP); Mari Dezawa (Sendai, JP); Teiji Tominaga (Sendai, JP)
Assignees: LIFE SCIENCE INSTITUTE, INC.; TOHOKU UNIVERSITY
A61K35/28A61K35/545
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Quick Facts
Patent No.
US 10,993,966
App. No.
15/413,086
Granted
May 4, 2021
Kind
B2
Abstract

An object of the present invention is to provide a novel medical application to regenerative medicine that uses pluripotent stem cells (Muse cells). The present invention provides a cell preparation for treating cerebral infarction and sequelae associated therewith that contains SSEA-3-positive pluripotent stem cells isolated from mesenchymal tissue in the body or cultured mesenchymal cells. The cell preparation of the present invention is based on a brain tissue regeneration mechanism by which Muse cells differentiate into nerve cells and the like in damaged brain tissue by administering Muse cells into cerebral parenchyma.

Claims (39)

1. A method for treating sequelae from a cerebral infarction in a mammalian subject in need thereof, the method comprising:

administering a cell preparation containing human pluripotent stem cells positive for SSEA-3 isolated from human mesenchymal tissue or cultured human mesenchymal cells to the mammalian subject wherein said pluripotent stem cells have a plurality of properties, said plurality of properties comprising:

(i) CD 105-positively;

(ii) low or absent telomerase activity;

(iii) ability to differentiate into any of the three germ layers;

(iv) absence of demonstration of neoplastic proliferation;

(v) self-renewal ability;

wherein the sequelae is selected from the group consisting of speech and language disorder, disturbance of perception such as numbness, disorder of movement in a limb, headache, vomiting, visual loss, deglutition disorder, articulation disorder, and dementia; and

wherein said pluripotent stem cells are administered 1 to 10 times at 1×10 3 cells to 2×10 7 cells per individual, or the total individual doses of said pluripotent stem cells are 1×10 3 cells to 2×10 8 cells or 1×10 4 cells to 1×10 8 cells, to treat sequelae from the cerebral infarction.

2. The method according to claim 1 , wherein the pluripotent stem cells positive for SSEA-3 contain a concentrated cell fraction as a result of stimulation by external stress.

3. The method according to claim 1 , wherein the pluripotent stem cells are CD117-negative and CD146-negative.

4. The method according to claim 1 , wherein the pluripotent stem cells are CD117-negative, CD146-negative, NG2-negative, CD34-negative, vWF-negative and CD271-negative.

5. The method according to claim 1 , wherein the pluripotent stem cells are CD34-negative, CD117-negative, CD146-negative, CD271-negative, NG2-negative, vWF-negative, Sox10-negative, Snail-negative, Slug-negative, Tyrpl-negative and Dct-negative.

6. The method according to claim 1 , wherein the pluripotent stem cells have the ability to differentiate into one or more cells selected from the group consisting of nerve cells, glial cells, vascular endothelial cells, and/or microglial cells.

7. The method according to claim 1 , wherein the cell preparation containing the pluripotent stem cells reduces infarct size by a mechanism involving regeneration of brain tissue.

8. The method according to claim 1 , wherein the cell preparation containing the pluripotent stem cells improves or restores a brain dysfunction due to cerebral infarction by a mechanism involving regeneration of brain tissue.

9. A method for treating sequelae from a cerebral infarction in a mammalian subject in need thereof, the method comprising:

administering to cerebral parenchyma of the mammalian subject a cell preparation containing human pluripotent stem cells positive for SSEA-3 isolated from human mesenchymal tissue or cultured human mesenchymal cells to the mammalian subject, wherein said pluripotent stem cells have a plurality of properties, said plurality of properties comprising:

(i) CD105-positively;

(ii) low or absent telomerase activity;

(iii) ability to differentiate into any of the three germ layers;

(iv) absence of demonstration of neoplastic proliferation;

(v) self-renewal ability, wherein the sequelae is selected from the group consisting of speech and language disorder, disturbance of perception such as numbness, disorder of movement in a limb, headache, vomiting, visual loss, deglutition disorder, articulation disorder, and dementia; and

wherein said pluripotent stem cells are administered 1 to 10 times at 1×10 3 cells to 2×10 7 cells per individual, or the total individual doses of said pluripotent stem cells are 1×10 3 cells to 2×10 8 cells or 1×10 4 cells to 1×10 8 cells, to treat sequelae from the cerebral infarction.

10. The method according to claim 9 , wherein the pluripotent stem cells positive for SSEA-3 contain a concentrated cell fraction as a result of stimulation by external stress.

11. The method according to claim 9 , wherein the pluripotent stem cells are CD117-negative and CD146-negative.

12. The method according to claim 9 , wherein the pluripotent stem cells are CD117-negative, CD146-negative, NG2-negative, CD34-negative, vWF-negative and CD271-negative.

13. The method according to claim 9 , wherein the pluripotent stem cells are CD34-negative, CD117-negative, CD146-negative, CD271-negative, NG2-negative, vWF-negative, Sox10-negative, Snail-negative, Slug-negative, Tyrpl-negative and Dct-negative.

14. The method according to claim 9 , wherein the pluripotent stem cells have the ability to differentiate into one or more cells selected from the group consisting of nerve cells, glial cells, vascular endothelial cells, and/or microglial cells.

15. The method according to claim 9 , wherein the cell preparation containing the pluripotent stem cells reduces infarct size by a mechanism involving regeneration of brain tissue.

16. The method according to claim 9 , wherein the cell preparation containing the pluripotent stem cells improves or restores a brain dysfunction due to cerebral infarction by a mechanism involving regeneration of brain tissue.

17. The method according to claim 1 , wherein the cell preparation containing the pluripotent stem cells is administered to cerebral parenchyma and accumulate in a region bordering the infarction and differentiate into nerve cells.

18. The method according to claim 9 , wherein the cell preparation containing the pluripotent stem cells accumulate in a region bordering the infarction and differentiate into nerve cells.

19. The method according to claim 1 , wherein the mammalian subject is a human.

20. The method according to claim 9 , wherein the mammalian subject is a human.

21. The method according to claim 1 , wherein the administration of the cell preparation improves motor function of the mammalian subject.

22. The method according to claim 9 , wherein the administration of the cell preparation improves motor function of the mammalian subject.

23. The method according to claim 1 , wherein the administration of the cell preparation is administered within 3 to 48 hours of infarction onset.

24. The method according to claim 9 , wherein the administration of the cell preparation is administered within 3 to 48 hours of infarction onset.

Assignments (3)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jul 17, 2024
From: LIFE SCIENCE INSTITUTE, INC.
To: TOHOKU UNIVERSITY
Reel/Frame 068009/0757 →
MERGER AND CHANGE OF NAME Recorded Feb 23, 2021
From: CLIO, INC.; LIFE SCIENCE INSTITUTE, INC.
To: LIFE SCIENCE INSTITUTE, INC.
Reel/Frame 055380/0521 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Mar 30, 2017
From: YOSHIDA, MASANORI; DEZAWA, MARI; TOMINAGA, TEIJI
To: CLIO, INC.; TOHOKU UNIVERSITY
Reel/Frame 041800/0121 →
Priority Claims (1)
JP JP2014-035725 · Feb 26, 2014 · national
Continuity (2)
Continuation 14695843 · Apr 24, 2015
Related Publication 20170128494A1 · May 11, 2017