IP Library Granted Patent US 11,434,501
Granted Patent B2
US 11,434,501 · App. 15/507,569 · Granted Sep 6, 2022

Sprr1A as a genetic target for treating neurodegenerative diseases

Inventors: Jack W. Lipton (Grand Rapids, MI); Nicholas Kanaan (Grand Rapids, MI); Timothy Collier (Grand Rapids, MI)
Assignee: Board of Trustees of Michigan State University
C12N15/86A61K35/30A61K38/04A61K38/39A61K39/395A61K48/005A61K49/14G01N33/5008C12N15/00C12N2750/14143G01N2800/2835
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Quick Facts
Patent No.
US 11,434,501
App. No.
15/507,569
Granted
Sep 6, 2022
Kind
B2
Abstract

This invention relates to the prevention and treatment of neurodegenerative diseases by administering compositions that increase the activity of a small proline-rich repeat 1 A protein (Sprr1A), or derivative thereof to the brain. Specifically, the compositions may comprise a nucleic acid molecule encoding a Sprr1A protein or a biologically-active portion thereof. The neurodegenerative disease may be Parkinson's Disease, Alzheimer's disease, amyotrophic lateral sclerosis, or traumatic brain injury.

Claims (14)

1. A method for treating Parkinson's Disease in a subject in need thereof, comprising administering to the subject a therapeutically effective amount of a composition comprising a nucleic acid molecule that encodes a Sprr1A protein, or a biologically-active portion thereof, wherein said nucleic acid molecule comprises at least 90% sequence homology to the nucleotide sequence set forth in SEQ ID NO:1, and wherein said composition increases the activity of a small proline-rich repeat 1a protein (“Sprr1A”), and wherein the composition is delivered to the brain.

2. The method of claim 1 , wherein said composition comprises a virus.

3. The method of claim 2 , wherein the virus is an adeno-associated virus (“AAV”), adenovirus, herpes simplex virus, or lentivirus.

4. The method of claim 3 , wherein the virus is AAV.

5. The method of claim 4 , wherein the virus is AAV1.

6. The method of claim 2 , wherein the genome of said virus comprises said nucleic acid molecule.

7. The method of claim 1 , wherein said nucleic acid molecule comprises a nucleotide sequence that has at least 95% sequence homology with the nucleotide sequence set forth in SEQ ID NO:1.

8. The method of claim 1 , wherein said nucleic acid molecule encodes an amino acid sequence that has at least 80% sequence homology with the amino acid sequence set forth in SEQ ID NO:2.

9. The method of claim 1 , wherein said nucleic acid molecule encodes an amino acid sequence that has at least 95% sequence homology with the amino acid sequence set forth in SEQ ID NO:2.

10. The method of claim 1 , wherein the composition is delivered to the substantia nigra, nigrostriatal system, hippocampus, medial temporal lobe, cerebral cortex, or spinal cord.

11. The method of claim 1 , wherein delivering the composition comprises injecting the composition.

12. The method of claim 1 , wherein the subject is a mammal.

13. The method of claim 12 , wherein the subject is a primate, porcine, canine, ovine, or rodent.

14. The method of claim 12 , wherein the subject is a human.

Assignments (2)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jun 14, 2017
From: LIPTON, JACK W.; KANAAN, NICHOLAS; COLLIER, TIMOTHY
To: BOARD OF TRUSTEES OF MICHIGAN STATE UNIVERSITY
Reel/Frame 042808/0994 →
CONFIRMATORY LICENSE Recorded Mar 7, 2017
From: MICHIGAN STATE UNIVERSITY
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 041896/0744 →
Continuity (2)
Provisional Application 62043103 · Aug 28, 2014
Related Publication 20170283829A1 · Oct 5, 2017