IP Library › Granted Patent US 10,780,117
Granted Patent B2
US 10,780,117 · App. 15/543,095 · Granted Sep 22, 2020

Macrophages eat cancer cells using their own calreticulin as a guide

Inventors: Irving L. Weissman (Stanford, CA); Mingye Feng (Mountain View, CA); Jens-Peter Volkmer (Menlo Park, CA)
Assignee: The Board of Trustees of the Leland Stanford Junior University
A61K35/15A61K31/4745A61K31/519A61K31/713A61K39/39A61K39/3955A61K45/06C12N5/0645A61K2039/55511A61K2039/55583C12N2501/50C12N2501/599
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Quick Facts
Patent No.
US 10,780,117
App. No.
15/543,095
Granted
Sep 22, 2020
Kind
B2
Abstract

Therapeutic and diagnostic methods are provided, which methods relate to the induction of expression of calreticulin on phagocytic cells. Specifically, the methods relate to macrophage-mediated programmed cell removal (PrCR), the methods comprising increasing PrCR by contacting a phagocytic cell with a toll-like receptor (TLR) agonist; or down-regulating PrCR by contacting a phagocytic cell with an inhibitor of Bruton's tyrosine kinase (BTK). In some embodiments, an activator of TLR signaling or a BTK agonist is provided in combination with CD47 blockade.

Claims (17)

1. A method of increasing phagocytosis of cancer cells, the method comprising:

contacting a population of phagocytic cells in vitro with a TLR-3, TLR-4 or TLR-9 agonist in a dose effective to increase expression of calreticulin on the phagocytic cell surface; and with a CD47 blocking agent in a dose effective to increase phagocytosis of cancer cells;

introducing the population of phagocytic cells into a subject following the contacting with a TLR agonist and a CD47 blocking agent, wherein expression of calreticulin on the phagocytic cell surface is measured prior to the introducing step;

wherein programmed cell removal of cancer cells by the phagocytic cells is increased.

2. The method of claim 1 , wherein the CD47 blocking agent specifically binds CD47 to reduce the binding of CD47 to SIRPα.

3. The method of claim 2 , wherein the CD47 blocking agent is a high affinity SIRPα polypeptide.

4. The method of claim 2 , wherein the CD47 blocking agent is an anti-CD47 antibody.

5. The method of claim 1 , wherein the CD47 blocking agent specifically binds SIRPα to reduce the binding of CD47 to SIRPα.

6. The method of claim 5 , wherein the CD47 blocking agent is an anti-SIRPα antibody.

7. The method of claim 1 , wherein the TLR-3, TLR-4 or TLR-9 agonist is selected from LPS, BCG ( Bacillus of Calmette-Guerin), CpG ODN, Poly I:C, and Poly I:CLC.

8. A method of increasing phagocytosis of cancer cells in vivo, the method comprising:

contacting a population of phagocytic cells with a TLR-3, TLR-4 or TLR-9 agonist in vivo in a dose effective to increase expression of calreticulin on the phagocytic cell surface; and with a CD47 blocking agent in a dose effective to increase phagocytosis of cancer cells; and

monitoring expression of calreticulin on the phagocytic cell surface following the contacting;

wherein programmed cell removal of cancer cells by the phagocytic cells is increased.

9. The method of claim 8 , wherein the CD47 blocking agent is an anti-CD47 antibody.

10. The method of claim 8 , wherein the CD47 blocking agent is an anti-SIRPα antibody.

11. The method of claim 8 , wherein the TLR-3, TLR-4 or TLR-9 agonist is selected from LPS, BCG ( Bacillus of Calmette-Guerin), CpG ODN, Poly I:C, and Poly I:CLC.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jul 14, 2017
From: WEISSMAN, IRVING L.; FENG, MINGYE; VOLKMER, JENS-PETER
To: THE BOARD OF TRUSTEES OF THE LELAND STANFORD JUNIOR UNIVERSITY
Reel/Frame 043009/0046 →
Continuity (2)
Provisional Application 62106050 · Jan 21, 2015
Related Publication 20180000865A1 · Jan 4, 2018
Cited By (1)
US 12,404,340