IP Library Granted Patent US 10,434,078
Granted Patent B2
US 10,434,078 · App. 15/786,440 · Granted Oct 8, 2019

GABA agonists in the treatment of disorders associated with metabolic syndrome and GABA combinations in treatment or prophylaxis of type I diabetes

Inventors: Daniel L. Kaufman (Los Angeles, CA); Jide Tian (Los Angeles, CA)
Assignee: The Regents of The University of California
A61K31/197A61K31/4465A61K38/2013A61K39/0008A61K39/39A61K45/06
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Quick Facts
Patent No.
US 10,434,078
App. No.
15/786,440
Granted
Oct 8, 2019
Kind
B2
Abstract

In certain embodiments methods are provided for the therapeutic or prophylactic amelioration of one or more symptoms or disorders associated with metabolic syndrome. In various embodiments the methods involve administering to a subject in need thereof, a GABA receptor agonist, in an amount sufficient to ameliorate said one or more symptoms. In certain embodiments methods are provided for the prophylaxis or treatment of type I diabetes and related pathologies that involve the use of GABA or GABA agonists in combination with certain other compounds (e.g., one more antigens (e.g., GAD) that have a therapeutic effect in type I diabetes and/or an anti-CD3 antibody, an anti-CD20 antibody, exendin-4, and/or or a pro-insulin therapeutic).

Claims (20)

1. A method of delaying the onset of Type I diabetes, and/or slowing the progression of, and/or reducing the severity of diabetes in a mammal, said method comprising coadministering to said mammal:

a first compound comprising gamma-aminobutyric acid (GABA); and

a second compound comprising sitagliptin and, optionally, an immunosuppressant;

wherein said first compound and said second compound are administered in an amount sufficient to delay the onset of diabetes, and/or slow the progression of diabetes, and/or reduce the severity of diabetes in said mammal.

2. A method of delaying the onset of hyperglycemia, and/or slowing the progression of hyperglycemia, and/or reducing the severity of hyperglycemia, said method comprising coadministering to said mammal:

a first compound comprising gamma-aminobutyric acid (GABA); and

a second compound comprising sitagliptin and, optionally, an immunosuppressant;

wherein said first compound and said second compound are administered in an amount sufficient to delay the onset of hyperglycemia, and/or slow the progression of hyperglycemia, and/or reduce the severity of hyperglycemia in said mammal.

3. A method of promoting transplanted islet cell survival in a mammal having Type I diabetes or at risk for type I diabetes who is a recipient of transplanted islet cells, said method comprising coadministering to said mammal:

a first compound comprising gamma-aminobutyric acid (GABA); and

a second compound comprising sitagliptin and, optionally, an immunosuppressant;

wherein said first compound and said second compound are administered in an amount sufficient to promote transplanted islet cell survival in said mammal.

4. A method of delaying the onset of an immune response, and/or slowing the progression of an immune response, and/or reducing the severity of an immune response, and/or suppressing an immune response in a mammal, said method comprising coadministering to said mammal:

a first compound comprising gamma-aminobutyric acid (GABA); and

a second compound comprising sitagliptin and, optionally, an immunosuppressant;

wherein said first compound and said second compound are administered in an amount sufficient to delay the onset of an immune response, and/or slow the progression of an immune response, and/or reduce the severity of an immune response, and/or suppress an immune response in said mammal.

5. A method of protecting beta-cells in a mammal from oxidative stress induced apoptosis said method comprising coadministering to said mammal:

a first compound comprising gamma-aminobutyric acid (GABA); and

a second compound comprising sitagliptin and, optionally, an immunosuppressant;

wherein said first compound and said second compound are administered in an amount sufficient to partially or fully protect said beta cells from oxidative stress induced apoptosis.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Aug 14, 2019
From: KAUFMAN, DANIEL; TIAN, JIDE
To: THE REGENTS OF THE UNIVERSITY OF CALIFORNIA
Reel/Frame 050051/0579 →
Continuity (5)
Division 14752487 · Jun 26, 2015
Division 13876147
Provisional Application 61387398 · Sep 28, 2010
Provisional Application 61433089 · Jan 14, 2011
Related Publication 20180193295A1 · Jul 12, 2018
Cited By (1)
US 12,565,529