IP Library Granted Patent US 11,980,671
Granted Patent B2
US 11,980,671 · App. 16/046,939 · Granted May 14, 2024

Use of bacterial voltage gated ion channels for human therapies

Inventors: Nenad Bursac (Durham, NC); Hung Nguyen (Durham, NC); Robert Kirkton (Durham, NC)
Assignee: Duke University
A61K48/0058A61K48/0008A61K48/0083A61P9/06A61P9/10C07K14/195A61K48/0075C12N2750/14143
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Quick Facts
Patent No.
US 11,980,671
App. No.
16/046,939
Granted
May 14, 2024
Kind
B2
Abstract

Disclosed herein are compositions including prokaryotic ion channel polypeptides and methods of treating voltage gated ion channel-related condition such as heart arrhythmia, chronic atrial fibrillation, damage from myocardial infarction, damage from stroke, and chronic ischemia.

Claims (30)

1. A method of treating a voltage gated ion channel-related condition in a subject, the method comprising administering directly to the heart of the subject by injection a recombinant AAV9 vector encoding a BacNav prokaryotic ion channel polypeptide,

wherein the subject is a mammal,

wherein the vector comprises a promoter that directs expression of the BacNav prokaryotic ion channel polypeptide,

wherein the BacNav prokaryotic ion channel polypeptide is expressed in the cardiomyocytes of the subject,

wherein the voltage gated ion channel-related condition is selected from heart arrhythmia, chronic atrial fibrillation, damage from myocardial infarction, heart damage from stroke, and chronic ischemia,

wherein the BacNav prokaryotic ion channel polypeptide comprises the amino acid sequence of SEQ ID NO: 24, and

wherein the voltage gated ion channel-related condition in the subject is treated.

2. The method of claim 1 , wherein the vector comprises the polynucleotide sequence of SEQ ID NO: 25.

3. The method of claim 1 , wherein the vector is administered to the subject in a pharmaceutical composition comprising the vector and a pharmaceutically acceptable carrier.

4. The method of claim 1 , wherein the subject is a rat.

5. The method of claim 1 , wherein the subject is a primate.

6. The method of claim 1 , wherein the subject is a human.

7. A method of increasing the conductivity of a cardiac tissue of a subject, the method comprising administering directly to the heart of the subject by injection a recombinant AAV9 vector encoding a BacNav prokaryotic ion channel polypeptide,

wherein the subject is a mammal and has a condition selected from heart arrhythmia, chronic atrial fibrillation, damage from myocardial infarction, heart damage from stroke, and chronic ischemia,

wherein the vector comprises a promoter that directs expression of the BacNav prokaryotic ion channel polypeptide,

wherein the BacNav prokaryotic ion channel polypeptide is expressed in the cardiomyocytes of the subject,

wherein the BacNav prokaryotic ion channel polypeptide comprises the amino acid sequence of SEQ ID NO: 24, and

wherein the conductivity of the cardiac tissue in the subject is increased.

8. A method of increasing the resistance to conduction block of a cardiac tissue of a subject, the method comprising administering directly to the heart of the subject by injection a recombinant AAV9 vector encoding a BacNav prokaryotic ion channel polypeptide,

wherein the subject is a mammal and has a condition selected from heart arrhythmia, chronic atrial fibrillation, damage from myocardial infarction, heart damage from stroke, and chronic ischemia,

wherein the vector comprises a promoter that directs expression of the BacNav prokaryotic ion channel polypeptide,

wherein the BacNav prokaryotic ion channel polypeptide is expressed in the cardiomyocytes of the subject,

wherein the BacNav prokaryotic ion channel polypeptide comprises the amino acid sequence of SEQ ID NO: 24, and

wherein the resistance to conduction block of the cardiac tissue in the subject is increased.

9. A method of increasing the upstroke velocity of a cardiac tissue of a subject, the method comprising administering directly to the heart of the subject by injection a recombinant AAV9 vector encoding a BacNav prokaryotic ion channel polypeptide,

wherein the subject is a mammal and has a condition selected from heart arrhythmia, chronic atrial fibrillation, damage from myocardial infarction, heart damage from stroke, and chronic ischemia,

wherein the vector comprises a promoter that directs expression of the BacNav prokaryotic ion channel polypeptide,

wherein the BacNav prokaryotic ion channel polypeptide is expressed in the cardiomyocytes of the subject,

wherein the BacNav prokaryotic ion channel polypeptide comprises the amino acid sequence of SEQ ID NO: 24, and

wherein the upstroke velocity of the cardiac tissue in the subject is increased.

Assignments (2)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jan 22, 2024
From: NGUYEN, HUNG; KIRKTON, ROBERT
To: DUKE UNIVERSITY
Reel/Frame 066199/0990 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Sep 20, 2018
From: BURSAC, NENAD
To: DUKE UNIVERSITY
Reel/Frame 046925/0645 →
Continuity (3)
Provisional Application 62557957 · Sep 13, 2017
Provisional Application 62537289 · Jul 26, 2017
Related Publication 20190030186A1 · Jan 31, 2019