IP Library Granted Patent US 11,187,704
Granted Patent B2
US 11,187,704 · App. 16/660,524 · Granted Nov 30, 2021

IL-6 signaling and breast cancer

Inventor: Peter P. Lee (Duarte, CA)
Assignee: CITY OF HOPE
G01N33/57415C12Q1/6886G01N33/6869C12Q2600/118C12Q2600/158G01N2333/4703G01N2333/5412G01N2333/7155G01N2800/54
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Quick Facts
Patent No.
US 11,187,704
App. No.
16/660,524
Granted
Nov 30, 2021
Kind
B2
Abstract

The disclosure provides, inter alia, methods of detecting IL-6 signaling activity in T cells in breast cancer patients, such as breast cancer patients in remission.

Claims (34)

1. A method of predicting an increased probability of breast cancer relapse in a breast cancer patient in need thereof, the method comprising:

(i) obtaining a peripheral blood sample comprising T cells from the patient;

(ii) contacting the peripheral blood sample with either:

(a) an IL-6 pathway detection agent capable of binding an IL-6 pathway protein, wherein the IL-6 pathway protein is phosphorylated STAT1, phosphorylated STAT3, IL-6Rα, or ADAM17, thereby forming a detectable complex; or

(b) an IL-6 pathway probe capable of hybridizing an IL-6 pathway mRNA expression sequence, wherein the IL-6 pathway mRNA expression sequence is gp130, thereby forming a hybridization complex;

(iii) detecting and/or quantifying: (a) the detectable complex, or (b) the hybridization complex, respectively;

(iv) predicting an increased probability of breast cancer relapse in the breast cancer patient when:

(1) the T cells from the breast cancer patient are less responsive to IL-6 relative to a standard control;

(2) the breast cancer patient has a lower level of IL-6Rα relative to a standard control;

(3) the breast cancer patient has a higher level of ADAM17 relative to a standard control; or

(4) the breast cancer patient has a lower level of gp130 relative to a standard control; and

(v) administering to the breast cancer patient having an increased probability of breast cancer relapse an effective amount of an anti-cancer agent.

2. The method of claim 1 , further comprising monitoring the patient for breast cancer relapse.

3. The method of claim 1 , wherein the anti-cancer agent is hormone replacement therapy, a chemotherapeutic agent, radiation therapy, or a combination of two or more thereof.

4. The method of claim 1 , further comprising monitoring the patient for breast cancer relapse and administering to the patient an anti-cancer agent.

5. The method of claim 1 , further comprising classifying the patient as having increased risk for relapse when the patient has a different IL-6 activity relative to a standard control.

6. The method of claim 1 , further comprising classifying the patient as having decreased risk for relapse when the patient has a similar IL-6 activity relative to a standard control.

7. The method of claim 1 , wherein the T cells are CD4 + T cells.

8. The method of claim 7 , wherein the CD4 + T cells are naïve CD4 + T cells.

9. A method of treating breast cancer in a patient having an increased risk for breast cancer relapse, the method comprising:

(i) determining IL-6 activity in the patient, and identifying the patient as having an increased risk for breast cancer relapse when:

(a) the T cells from the patient are less responsive to IL-6 relative to a standard control;

(b) the patient has a lower level of IL-6Rα relative to a standard control;

(c) the patient has a higher level of ADAM17 relative to a standard control; or

(d) the patient has a lower level of gp130 relative to a standard control; and

(ii) administering to the patient an effective amount of an anti-cancer agent.

10. The method of claim 9 , further comprising monitoring the patient for breast cancer relapse.

11. The method of claim 9 , wherein determining the IL-6 activity in the patient comprises:

(i) obtaining a peripheral blood sample comprising T cells from the patient;

(ii) contacting the peripheral blood sample with either:

(a) an IL-6 pathway detection agent capable of binding an IL-6 pathway protein, wherein the IL-6 pathway protein is phosphorylated STAT1, phosphorylated STAT3, IL-6Rα, or ADAM17; thereby forming a detectable complex; or

(b) an IL-6 pathway probe capable of hybridizing an IL-6 pathway mRNA expression sequence, wherein the IL-6 pathway mRNA expression sequence is gp130; thereby forming a hybridization complex;

(iii) detecting and/or quantifying: (a) the detectable complex, or (b) the hybridization complex, respectively.

12. The method of claim 9 , wherein the anti-cancer agent comprises hormone replacement therapy, a chemotherapeutic agent, radiation therapy, or a combination of two or more thereof.

Assignments (2)
CONFIRMATORY LICENSE Recorded Apr 16, 2025
From: BECKMAN RESEARCH INSTITUTE/CITY OF HOPE
To: UNITED STATES GOVERNMENT
Reel/Frame 070863/0918 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Feb 28, 2020
From: LEE, PETER P.
To: CITY OF HOPE
Reel/Frame 051963/0383 →
Continuity (3)
Continuation 15806883 · Nov 8, 2017
Provisional Application 62419813 · Nov 9, 2016
Related Publication 20200116724A1 · Apr 16, 2020
Cited By (1)
US 12,385,920