IP Library Granted Patent US 11,186,840
Granted Patent B2
US 11,186,840 · App. 16/892,995 · Granted Nov 30, 2021

CTLA-4 aptamer conjugates

Inventors: Hua Yu (Glendora, CA); Andreas Herrmann (Pasadena, CA); Marcin Tomasz Kortylewski (Monrovia, CA)
Assignee: CITY OF HOPE
C12N15/113A61K31/713A61K45/06A61K47/549C12N15/111C12N15/115C12N15/1138C12N2310/11C12N2310/12C12N2310/13C12N2310/14C12N2310/16C12N2310/3519C12N2320/30C12N2320/32
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Quick Facts
Patent No.
US 11,186,840
App. No.
16/892,995
Granted
Nov 30, 2021
Kind
B2
Abstract

Provided herein are, inter alia, nucleic acid compounds useful for targeting CTLA-4-expressing cells and modulating cell activity of the CTLA-4-expressing cells. The compositions provided herein may be part of pharmaceutical compositions and may be used for treatment of cancer, inflammatory diseases, infectious diseases or metabolic diseases.

Claims (14)

1. A method of stimulating the immune system of a subject in need thereof, said method comprising administering to a subject a therapeutically effective amount of a CTLA-4 aptamer nucleic acid conjugated to a cell activity modulating nucleic acid, wherein said cell activity modulating nucleic acid is a FoxP3 antisense nucleic acid, an anti-tyrosine kinase antisense nucleic acid, or a Signal Transducer and Activator of Transcription (STAT) antisense nucleic acid, thereby stimulating the immune system of said subject.

2. The method of claim 1 , wherein stimulating the immune system comprises maturation, differentiation, or proliferation of natural killer cells, T cells, monocytes, or macrophages.

3. The method of claim 1 , wherein stimulating the immune system comprises an increase in a T H 1-type immune response.

4. The method of claim 1 , wherein stimulating the immune system comprises stimulating a plasmacytoid dendritic cell, myeloid dendritic cell, myeloid-derived suppressor cell, macrophage, B cell, activated NK cell, or activated neutrophil.

5. The method of claim 1 , wherein stimulating the immune system comprises increasing the activity of a cytotoxic T cell.

6. The method of claim 5 , wherein the cytotoxic T cell is a tumor-specific cytotoxic T cell.

7. The method of claim 1 , wherein said CTLA-4 aptamer nucleic acid is conjugated to said cell activity modulating nucleic acid through a linker.

8. The method of claim 7 , wherein said linker comprises a non-nucleic acid spacer.

9. The method of claim 8 , wherein said non-nucleic acid spacer connects said cell activity modulating nucleic acid to the 3′ terminal end of said linker nucleic acid sequence.

10. The method of claim 7 , wherein said linker connects said cell activity modulating nucleic acid to the 3′ terminal end of said CTLA-4 aptamer nucleic acid.

11. The method of claim 1 , wherein said antisense nucleic acid is a siRNA.

12. The method of claim 1 , wherein said cell activity modulating nucleic acid is a FoxP3 antisense nucleic acid.

13. The method of claim 1 , wherein said cell activity modulating nucleic acid is an anti-tyrosine kinase antisense nucleic acid.

14. The method of claim 1 , wherein said cell activity modulating nucleic acid is a Signal Transducer and Activator of Transcription (STAT) antisense nucleic acid.

Assignments (2)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Sep 30, 2025
From: SWIDERSKI, PIOTR MAREK
To: CITY OF HOPE
Reel/Frame 072427/0947 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Dec 2, 2020
From: YU, HUA; HERRMANN, ANDREAS; KORTYLEWSKI, MARCIN TOMASZ
To: CITY OF HOPE
Reel/Frame 054517/0096 →
Continuity (3)
Continuation 14602182 · Jan 21, 2015
Provisional Application 61929832 · Jan 21, 2014
Related Publication 20200377890A1 · Dec 3, 2020
Cited By (2)
US 12,404,509 US 12,655,429