IP Library Patent Application 17238773
Patent Application
App. No. 17/238,773

METHODS AND COMPOSITIONS FOR INCREASING THE SUPPRESSIVE FUNCTION OF REGULATORY T-CELLS (TREGS)

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Patent No.
US None
App. No.
17/238,773
Abstract

Methods and compositions for increasing the suppressive function of regulatory T-cells (Tregs) are provided.

Claims (47)

1 . A method of reducing or eliminating the vimentin protein and/or the RLTPR protein and/or the PKC-θ protein in Treg cells, comprising:

contacting the Treg cells with a vimentin-specific and/or a RLTPR-specific and/or a PKC-θ-specific inhibitory nucleic acid molecule.

2 . The method of claim 1 , wherein the vimentin-specific inhibitory nucleic acid molecule is complementary to at least a portion of the sequence shown in SEQ ID NO: 1.

3 . The method of claim 1 , wherein the RLTPR-specific inhibitor nucleic acid is complementary to at least a portion of the sequence shown in SEQ ID NO: 5.

4 . The method of claim 1 , wherein the PKC-θ-specific inhibitory nucleic acid molecule is complementary to at least a portion of the sequence shown in SEQ ID NO: 9.

5 . The method of claim 1 , wherein the vimentin-specific and/or the RLTPR-specific and/or the PKC-θ-specific inhibitory nucleic acid molecule is a RNAi nucleic acid molecule.

6 . The method of claim 1 , wherein the vimentin-specific and/or the RLTPR-specific and/or the PKC-θ-specific inhibitory nucleic acid molecule is an antisense nucleic acid molecule.

7 . The method of claim 1 , wherein the vimentin-specific and/or the RLTPR-specific and/or the PKC-θ-specific inhibitor nucleic acid molecule is a siRNA nucleic acid molecule.

8 . The method of claim 1 , wherein the vimentin-specific inhibitory nucleic acid molecule has the sequence shown in SEQ ID NO: 13, 14, 15 or 16.

9 . The method of claim 1 , wherein the Treg cells are contacted in vitro.

10 . The method of claim 1 , wherein the Treg cells are contacted in situ.

11 . The method of claim 1 , wherein the Treg cells are contacted in vivo in an individual who has received or is receiving a bone marrow transplant.

12 . The method of claim 1 , wherein the Treg cells exhibit a phenotype of at least one of the following:

reduced PKC-θ auto-phosphorylation at Ser676;

improved ability to suppress CD4+ and CD8+ Tcon proliferation;

increased surface expression of Nrp1;

increased surface expression of Lag3;

increased basal and maximal oxygen consumption rate (OCR);

increased BoDipy C1-C12 uptake;

increased expression of CD71;

increased expression of CD98;

increased expression of CPT1a; or

reduced activity of mTORC2,

relative to Tregs that lack the vimentin-specific and/or the RLTPR-specific and/or the PKC-θ-specific inhibitory nucleic acid molecule.

13 . A method of increasing or augmenting the suppressor cell potency of Treg cells, comprising:

reducing or eliminating vimentin and/or RLTPR and/or PKC-θ in the Treg cells.

14 . The method of claim 13 , wherein reducing or eliminating the vimentin and/or the RLTPR and/or the PKC-θ in the Treg cells comprising contacting the Treg cells with a moiety selected from the group consisting of a nucleic acid, a nuclease, an antibody, a ligand, a peptide, a drug, a chemical, or a small molecule.

15 . The method of claim 14 , wherein the nucleic acid is a vimentin-specific and/or a RLTPR-specific and/or a PKC-θ-specific inhibitory nucleic acid molecule.

16 . The method of claim 15 , wherein the vimentin-specific and/or the RLTPR-specific and/or the PCK-θ-specific inhibitory nucleic acid molecule is selected from the group consisting of a RNAi nucleic acid molecule, an antisense nucleic acid molecule, and a siRNA nucleic acid molecule.

17 . The method of claim 15 , wherein the vimentin-specific inhibitory nucleic acid molecule is complementary to at least a portion of the sequence shown in SEQ ID NO: 1.

18 . The method of claim 15 , wherein the RLTPR-specific inhibitory nucleic acid molecule is complementary to at least a portion of the sequence shown in SEQ ID NO: 5.

19 . The method of claim 15 , wherein the PKC-θ-specific inhibitory nucleic acid molecule is complementary to at least a portion of the sequence shown in SEQ ID NO: 9.

20 . The method of claim 13 , wherein the method is performed in vitro.

21 . The method of claim 13 , wherein the method is performed in situ.

22 . The method of claim 21 , wherein the method is performed on an individual who has received or is receiving a bone marrow transplant.

23 . The method of claim 13 , wherein the Treg cells in which the vimentin, and/or RLTPR and/or PCK-θ has been reduced or eliminated exhibit a phenotype of at least one of the following:

reduced PKC-θ auto-phosphorylation at Ser676;

improved ability to suppress CD4+ and CD8+ Tcon proliferation;

increased surface expression of Nrp1;

increased surface expression of Lag3;

increased basal and maximal oxygen consumption rate (OCR);

increased BoDipy C1-C12 uptake;

increased expression of CD71;

increased expression of CD98;

increased expression of CPT1a; or

reduced activity of mTORC2,

relative to Tregs in which vimentin, RLTPR and/or PCK-θ is not reduced or eliminated.

Assignments (3)
CONFIRMATORY LICENSE Recorded Nov 14, 2023
From: UNIVERSITY OF MINNESOTA
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 065565/0708 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded May 20, 2021
From: BLAZAR, BRUCE R.; MCDONALD-HYMAN, CAMERON; HIPPEN, KELI
To: REGENTS OF THE UNIVERSITY OF MINNESOTA
Reel/Frame 056305/0570 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded May 20, 2021
From: DUSTIN, MICHAEL; KUMARI, SUDHA; NEUBERT, TOM; MULLER, JAMES
To: NEW YORK UNIVERSITY
Reel/Frame 056305/0728 →