IP Library Patent Application 19060033
Patent Application
App. No. 19/060,033

Gene Therapy for NMNAT1-Associated Retinal Degeneration

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Quick Facts
Patent No.
US None
App. No.
19/060,033
Abstract

Methods and compositions for gene therapy of retinal degeneration related to mutations in nicotinaminde mononucleotide adenylyltransferase 1 (NMNAT1).

Claims (26)

1 - 12 . (canceled)

13 . An adeno-associated virus (AAV) vector genome comprising from 5′ to 3′:

(i) a CASI promoter;

(ii) a human nicotinamide mononucleotide adenylyltransferase 1 (NMNAT1) coding sequence; and

(iii) a bovine growth hormone polyadenylation signal (bGHpA) sequence.

14 . The AAV vector genome of claim 13 , wherein the human NMNAT1 coding sequence has at least 90% sequence identity to the nucleotide sequence set forth in SEQ ID NO: 1.

15 . The AAV vector genome of claim 13 , wherein the human NMNAT1 coding sequence has at least 95% sequence identity to the nucleotide sequence set forth in SEQ ID NO: 1.

16 . The AAV vector genome of claim 13 , wherein the human NMNAT1 coding sequence encodes the amino acid sequence set forth in SEQ ID NO: 3.

17 . The AAV vector genome of claim 13 , wherein the human NMNAT1 coding sequence has at least 90% sequence identity to the nucleotide sequence set forth in SEQ ID NO: 1 and encodes the amino acid sequence set forth in SEQ ID NO: 3.

18 . The AAV vector genome of claim 13 , wherein the human NMNAT1 coding sequence has at least 95% sequence identity to the nucleotide sequence set forth in SEQ ID NO: 1 and encodes the amino acid sequence set forth in SEQ ID NO: 3.

19 . The AAV vector genome of claim 13 , wherein the CASI promoter drives expression in photoreceptor cells.

20 . The AAV vector genome of claim 13 , wherein the AAV vector genome is a single-stranded or self-complementary AAV vector genome.

21 . The AAV vector genome of claim 13 , wherein the AAV vector genome is a self-complementary AAV vector genome.

22 . A recombinant AAV (rAAV) comprising an AAV capsid and the AAV vector genome of claim 1 .

23 . The rAAV of claim 22 , wherein the AAV capsid is an AAV9 or Anc80 capsid.

24 . The rAAV of claim 22 , wherein the AAV capsid is an AAV9 capsid.

25 . A recombinant adeno-associated virus (rAAV) comprising an AAV9 capsid and an AAV vector genome comprising from 5′ to 3′:

(i) a CASI promoter;

(ii) a human nicotinamide mononucleotide adenylyltransferase 1 (NMNAT1) coding sequence, wherein the human NMNAT1 coding sequence has at least 90% sequence identity to the nucleotide sequence set forth in SEQ ID NO: 1 and encodes the amino acid sequence set forth in SEQ ID NO: 3; and

(iii) a bovine growth hormone polyadenylation signal (bGHpA) sequence.

26 . A pharmaceutical composition comprising the rAAV of claim 22 .

27 . A pharmaceutical composition comprising the rAAV of claim 25 .

28 . A method of increasing expression of NMNAT1 in the eye of a human subject, the method comprising delivering to an eye of the subject a therapeutically effective amount of the recombinant AAV of claim 22 .

29 . A method of treating a disease caused by one or more mutations in a nicotinamide mononucleotide adenylyltransferase 1 (NMNAT1) gene in a human subject, the method comprising delivering to an eye of the subject a therapeutically effective amount of the recombinant AAV of claim 22 .

30 . The method of claim 29 , wherein the disease is selected from the group consisting of retinal degeneration, Leber congenital amaurosis, early-onset severe retinal dystrophy, and loss of vision.

31 . A method of treating a disease caused by one or more mutations in a nicotinamide mononucleotide adenylyltransferase 1 (NMNAT1) gene in a human subject, the method comprising delivering to an eye of the subject a therapeutically effective amount of the recombinant AAV of claim 25 .