IP Library Granted Patent US 8,354,110
Granted Patent B2
US 8,354,110 · App. 12/044,435 · Granted Jan 15, 2013

Compositions and methods for the prevention and treatment of autoimmune conditions

Inventors: Pedro Santamaria (Calgary, CA); Anna Moore (Dracut, MA)
Assignees: UTI Limited Partnership; The General Hospital Corporation
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Quick Facts
Patent No.
US 8,354,110
App. No.
12/044,435
Granted
Jan 15, 2013
Kind
B2
Abstract

The methods include selectively reducing or expanding T cells according to the antigenic specificity of the T cells. Therefore, the present invention can be used to reduce or eliminate pathogenic T cells that recognize autoantigens, such as beta cell specific T cells. As such, the present invention can be used to prevent, treat or ameliorate autoimmune diseases such as IDDM. Furthermore, the present invention can be used to expand desirable T cells, such as anti-pathogenic T cells to prevent, treat and/or ameliorate autoimmune diseases.

Claims (34)

1. A method of treating type I diabetes or prediabetes, in a patient which method comprises:

identifying a patient having type I diabetes or prediabetes; and

administering to said patient an antigen-MHC complex operatively coupled to a non-liposomal nanoparticle in an amount sufficient to expand low avidity anti-pathogenic autoreactive T cells;

wherein the antigen is a type I diabetes relevant; and

further wherein said nanoparticle has a diameter of less than 1 μm.

2. The method of claim 1 , wherein the MHC component is a MHC class I component.

3. The method of claim 2 , wherein the MHC class I component comprises all or part of a HLA-A, HLA-B, HLA-C, HLA-E, HLA-F, HLA-G or CD-1 molecule.

4. The method of claim 2 , wherein, the MHC class I component comprises all or part of a HLA-A molecule.

5. The method of claim 2 , wherein the MHC class I component comprises all or part of a HLA-A*0201 MHC class I molecule.

6. The method of claim 1 , wherein the nanoparticle comprises a metal ion.

7. The method of claim 6 , wherein the metal ion is iron (III).

8. The method of claim 1 , wherein the antigen-MHC complex is covalently bound to the nanoparticle.

9. The method of claim 8 , wherein the complex is bound to the nanoparticle via a linker.

10. The method of claim 9 , wherein the linker is one or more of the group a peptide linker, ethylene glycol, biotin, and strepdavidin.

11. The method of claim 1 , wherein the T cells expanded by the treatment have been preactivated by type I diabetes and have a memory phenotype.

12. The method of claim 1 , wherein the T cell is a CD4 + or CD8 + T cell.

13. A method of treating type I diabetes or prediabetes in a patient in need thereof which method comprises:

identifying a patient having type I diabetes or prediabetes; and

administering to said patient a peptide-MHC complex operatively coupled to a non-liposomal nanoparticle in an amount sufficient to expand low avidity anti-pathogenic autoreactive T cells;

wherein the peptide is a type I diabetes relevant and the MHC is a classical MHC molecule;

wherein said nanoparticle has a diameter of less than 1 μm; and

further wherein the peptide is an epitope from an autoantigen expressed by pancreatic beta cells.

14. The method of claim 13 , wherein the autoantigen is islet-specific glucose-6-phosphatase catalytic subunit-related protein, insulin, glutamic acid decarboxylase or IA-2 protein.

15. The method of claim 13 , wherein the autoantigen is an epitope derived from an endocrine or neurocrine component.

16. The method of claim 15 wherein the endocrine or neuroendocrine component is peri-islet Schwann cells.

17. The method of claim 1 or 13 , wherein the method treats type I diabetes.

18. The method of claim 1 or 13 , wherein the method treats prediabetes.

19. A method of treating type I diabetes or prediabetes in a patient which method comprises:

identifying a patient having type I diabetes or prediabetes; and

administering to said patient an antigen-MHC class II complex operatively coupled to a non-liposomal nanoparticle in an amount sufficient to expand low avidity anti-pathogenic autoreactive T cells;

wherein the antigen is a type I diabetes relevant antigen; and

wherein said nanoparticle has a diameter of less than 1 μm.

20. The method of claim 19 , wherein the MHC class II component comprises all or part of a HLA-DR, HLA-DQ, or HLA-DP.

21. The method of any one of claims 1 , 13 and 19 , wherein the non-liposomal nanoparticle comprises a solid core.

Assignments (4)
CONFIRMATORY LICENSE Recorded Dec 29, 2014
From: THE GENERAL HOSPITAL CORPORATION
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 034711/0464 →
NUNC PRO TUNC ASSIGNMENT Recorded Nov 18, 2011
From: UTI LIMITED PARTNERSHIP; MOORE, ANNA
To: MOORE, ANNA
Reel/Frame 027259/0893 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Nov 18, 2011
From: MOORE, ANNA
To: THE GENERAL HOSPITAL CORPORATION
Reel/Frame 027259/0903 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jul 14, 2008
From: SANTAMARIA, PEDRO; MOORE, ANNA
To: UTI LIMITED PARTNERSHIP
Reel/Frame 021231/0460 →
Continuity (2)
Provisional Application 60893530 · Mar 7, 2007
Related Publication 20090155292A1 · Jun 18, 2009