IP Library Granted Patent US 10,227,610
Granted Patent B2
US 10,227,610 · App. 14/188,212 · Granted Mar 12, 2019

Methods and compositions for enhancing nuclease-mediated gene disruption

Inventors: Fuqiang Chen (St. Louis, MO); Qiaohua Kang (Saint Louis, MO); Thomas Wechsler (Richmond, CA)
Assignees: Sangamo Therapeutics, Inc.; Sigma Aldrich Co LLC
C12N15/907A61K31/502A61K31/5377C12N9/22C07K2319/80C07K2319/81
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Quick Facts
Patent No.
US 10,227,610
App. No.
14/188,212
Granted
Mar 12, 2019
Kind
B2
Abstract

Methods and compositions for increasing nuclease-mediated genomic modification using DNA repair inhibitors are provided.

Claims (10)

1. A method for targeted genomic disruption via microhomology-mediated end joining (MMEJ) in a cell, the method comprising:

administering at least one nuclease to the cell, wherein the nuclease cleaves endogenous genomic sequences in the cell, wherein the nuclease is a zinc finger nuclease, a TALE effector domain nuclease (TALEN) and/or a CRISPR/Cas nuclease system; and

growing the cell in a medium comprising at least one small molecule inhibitor of a DNA-dependent-protein kinase catalytic subunit (DNA-PKcs) protein and a small molecule inhibitor of a Poly-(ADP-ribose) polymerase 1/2 (PARP1/2) protein at 0.5 to 25 wherein the small molecule inhibitor is a nicotinamide; a isoquinolinone and a dihydroisoquinolinones; a benzimidazole; an indole; phthalazin-1(2H)-one; a quinazolinone; an isoindolinone and analogues and derivatives thereof; a phenanthridine; a phenanthridinone; a benzopyrone and analogues and derivatives thereof; an unsaturated hydroximic acid derivative and analogues and derivatives thereof; a pyridazine; caffeine, theophylline; thymidine and/or NU7026 and/or NU7441, wherein the endogenous genomic sequences in the cell are disrupted via MMEJ after cleavage by the at least one nuclease.

2. The method of claim 1 , wherein the DNA-PKcs protein and/or (PARP1/2) protein is selected from the group consisting of PARP1, Ku70/80, DNA-PKcs, XRCC4/XLF, Ligase IV, Ligase III, XRCC1, Artemis Polynucleotide Kinase (PNK) and combinations thereof.

3. The method of claim 1 , wherein the targeted genomic disruption comprises a deletion.

4. The method of claim 1 , wherein the targeted genomic disruption comprises an insertion.

5. The method of claim 4 , further comprising administering an exogenous sequence to the cell, wherein the exogenous sequence is integrated into the genome via homology directed repair (HDR) mechanisms following cleavage by the nuclease.

6. The method of claim 5 , wherein the exogenous sequence is selected from the group consisting of a sequence encoding a protein, a regulatory sequence, a sequence that encodes a structural RNA and combinations thereof.

7. The method of claim 1 , wherein the nuclease is administered using an expression vector or as mRNA.

8. The method of claim 1 , wherein the cell further comprises a Rad52 mRNA.

Assignments (3)
CHANGE OF NAME Recorded Dec 11, 2018
From: SANGAMO BIOSCIENCES, INC.
To: SANGAMO THERAPEUTICS, INC.
Reel/Frame 047788/0504 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jun 12, 2014
From: CHEN, FUQIANG; KANG, QIAOHUA
To: SIGMA ALDRICH CO. LLC
Reel/Frame 033089/0874 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded May 27, 2014
From: WECHSLER, THOMAS
To: SANGAMO BIOSCIENCE, INC.
Reel/Frame 032969/0736 →
Continuity (2)
Provisional Application 61769038 · Feb 25, 2013
Related Publication 20140242702A1 · Aug 28, 2014
Cited By (2)
US 12,385,025 US 12,735,689