IP Library Granted Patent US 7,608,586
Granted Patent B2
US 7,608,586 · App. 10/558,984 · Granted Oct 27, 2009

Soluble low-density lipoprotein receptor related protein binds directly to Alzheimer's amyloid-beta peptide

Assignees: The University of Rochester; Socratech L.L.C.
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Quick Facts
Patent No.
US 7,608,586
App. No.
10/558,984
Granted
Oct 27, 2009
Kind
B2
Abstract

A soluble derivative of low-density lipoprotein receptor related protein-1 (sLRP-1) binds directly to Alzheimer's amyloid-β peptide (Aβ). This binding may be used to detect Aβ or to separate Aβ from the rest of a subject's body. In Alzheimer's disease, it may be used to provide diagnostic results by detecting Aβ, treatment by removing Aβ, or both.

Claims (52)

1. A method of binding amyloid-β peptide (Aβ) in a body fluid and/or tissue of a subject, said method comprising:

(a) providing a soluble derivative of low-density lipoprotein receptor related protein-1 (LRP-1), wherein said soluble LRP-1 derivative is comprised of cluster II, cluster IV, or both;

(b) contacting said soluble LRP-1 derivative with at least said body fluid and/or tissue of said subject such that said Aβ is specifically bound; and

(c) detecting said soluble LRP-1 derivative bound to Aβ.

2. The method of claim 1 , wherein said soluble LRP-1 derivative binds said Aβ inside said subject's body.

3. The method of claim 1 , wherein said soluble LRP-1 derivative binds said Aβ outside said subject's body.

4. The method of claim 1 , wherein said soluble LRP-1 derivative bound to Aβ is removed from said subject's body.

5. The method of claim 1 , wherein said soluble LRP-1 derivative bound to Aβ is inactivated such that amyloid deposits are reduced in said subject's body.

6. The method of claim 1 , wherein said soluble LRP-1 derivative is comprised of cluster II.

7. The method of claim 1 , wherein said soluble LRP-1 derivative is comprised of cluster IV.

8. The method of claim 1 , wherein said soluble LRP-1 derivative consists essentially of cluster II, cluster IV, or both.

9. The method of claim 1 , wherein said soluble LRP-1 derivative is comprised of at least one domain which mediates secretion.

10. The method of claim 1 , wherein said soluble LRP-1 derivative is not comprised of a domain which mediates attachment to a lipid bilayer.

11. The method of claim 1 , wherein said soluble LRP-1 derivative is reversibly attached to a solid substrate.

12. The method of claim 1 , wherein said soluble LRP-1 derivative is irreversibly attached to a solid substrate.

13. The method of claim 1 , wherein said soluble LRP-1 derivative is derived from human.

14. The method of claim 1 , wherein said soluble LRP-1 derivative does not elicit an immune response in a human.

15. The method of claim 1 , wherein said soluble LRP-1 derivative further comprises at least one heterologous domain.

16. The method of claim 15 , wherein at least one detectable label is covalently attached to heterologous domain.

17. The method of claim 1 , wherein at least one detectable label is covalently attached to said soluble LRP-1 derivative.

18. The method of claim 1 , wherein said soluble LRP-1 derivative is comprised of both cluster II and cluster IV.

19. A method of binding amyioid-β peptide (Aβ) in a body fluid of a human subject, said method comprising:

(a) providing a soluble fragment of low-density lipoprotein receptor related protein-1 (LRP-1) comprising cluster II, cluster IV, or both of human LRP-1;

(b) contacting said soluble fragment of LRP-1 with at least said body fluid of said human subject such that said Aβ is specifically bound; and

(c) detecting said soluble fragment of LRP-1 bound to Aβ.

20. The method of claim 19 , wherein said soluble fragment of LRP-1 bound to Aβ is removed from said human subject's body.

21. The method of claim 19 , wherein said soluble fragment of LRP-1 bound to Aβ is inactivated such that amyloid deposits are reduced in said human subject's body.

22. A method of binding amyloid-β peptide (Aβ) in a body fluid and/or tissue of a subject, said method comprising:

(a) providing a soluble derivative of low-density lipoprotein receptor related protein-1 (LRP-1), wherein said soluble LRP-1 derivative is comprised of cluster II, cluster IV, or both;

(b) contacting said soluble LRP-1 derivative with at least said body fluid and/or tissue of said subject such that said Aβ is specifically bound; and

(c) removing said soluble LRP-1 derivative bound to Aβ from said subject's body.

23. The method of claim 22 , wherein said soluble LRP-1 derivative binds said Aβ inside said subject's body.

24. The method of claim 22 , wherein said soluble LRP-1 derivative binds said Aβ outside said subject's body.

25. The method of claim 22 , wherein said soluble LRP-1 derivative bound to Aβ is inactivated such that amyloid deposits are reduced in said subject's body.

26. The method of claim 22 , wherein said soluble LRP-1 derivative is comprised of cluster lI.

27. The method of claim 22 , wherein said soluble LRP-1 derivative is comprised of cluster IV.

28. The method of claim 22 , wherein said soluble LRP-1 derivative consists essentially of cluster II, cluster IV, or both.

29. The method of claim 22 , wherein said soluble LRP-1 derivative is comprised of at least one domain which mediates secretion.

30. The method of claim 22 , wherein said soluble LRP-1 derivative is not comprised of a domain which mediates attachment to a lipid bilayer.

31. The method of claim 22 , wherein said soluble LRP-1 derivative is reversibly attached to a solid substrate.

32. The method of claim 22 , wherein said soluble LRP-1 derivative is irreversibly attached to a solid substrate.

33. The method of claim 22 , wherein said soluble LRP-1 derivative is derived from human.

34. The method of claim 22 , wherein said soluble LRP-1 derivative does not elicit an immune response in a human.

35. The method of claim 22 , wherein said soluble LRP-1 derivative further comprises at least one heterologous domain.

36. The method of claim 35 , wherein at least one detectable label is covalently attached to said heterologous domain.

37. The method of claim 22 , wherein at least one detectable label is covalently attached to said soluble LRP-1 derivative.

38. The method of claim 22 , wherein said soluble LRP-1 derivative is comprised of both cluster II and cluster IV.

39. A method of binding amyloid-β peptide (Aβ) in a body fluid of a human subject, said method comprising:

(a) providing a soluble fragment of low-density lipoprotein receptor related protein-1 (LRP-1) comprising cluster II, cluster IV, or both of human LRP-1;

(b) contacting said soluble fragment of LRP-1 with at least said body fluid of said human subject such that said Aβ is specifically bound; and

(c) removing said soluble fragment of LRP-1 derivative bound to Aβ from said subject's body.

40. The method of claim 39 , wherein said soluble fragment of LRP-1 derivative bound to Aβ is inactivated such that amyloid deposits are reduced in said human subject's body.

Assignments (4)
CONFIRMATORY LICENSE Recorded Feb 24, 2014
From: UNIVERSITY OF ROCHESTER
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 032329/0885 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded May 31, 2011
From: SOCRATECH L.L.C.
To: ZZ ALZTECH L.L.C.
Reel/Frame 026449/0737 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Aug 17, 2006
From: ZLOKOVIC, BERISLAV V.
To: THE UNIVERSITY OF ROCHESTER; SOCRATECH L.L.C.
Reel/Frame 018188/0930 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Aug 17, 2006
From: DEANE, RASHID
To: ROCHESTER, THE UNIVERSITY OF
Reel/Frame 018189/0277 →
Continuity (2)
Provisional Application 6047740400 · Jun 11, 2003
Related Publication 20070054318A1 · Mar 8, 2007