IP Library Granted Patent US 8,609,093
Granted Patent B2
US 8,609,093 · App. 13/409,585 · Granted Dec 17, 2013

Methods of treatment using anti-IL-17F antibodies

Inventors: Krzysztof Masternak (Mollens, CH); Francois Rousseau (Collonges sous Saleve, FR)
Assignee: Novimmune S. A.
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Quick Facts
Patent No.
US 8,609,093
App. No.
13/409,585
Granted
Dec 17, 2013
Kind
B2
Abstract

This invention provides fully human monoclonal antibodies that recognize IL-17F and/or the heterodimeric IL-17A/IL-17F complex, but do not recognize IL-17A. The invention further provides methods of using such monoclonal antibodies as a therapeutic, diagnostic, and prophylactic.

Claims (30)

1. A method of treating an autoimmune disease or inflammatory disorder, the method comprising administering an effective amount of an anti-IL-17F antibody, or an antigen-binding fragment thereof, to a subject in need thereof, wherein said antibody, or antigen-binding fragment thereof, comprises:

(a) a V H CDR1 region comprising the amino acid sequence of SEQ ID NO: 45;

(b) a V H CDR2 region comprising the amino acid sequence of SEQ ID NO: 46;

(c) a V H CDR3 region comprising the amino acid sequence of SEQ ID NO: 47;

(d) a V L CDR1 region comprising the amino acid sequence of SEQ ID NO: 74;

(e) a V L CDR2 region comprising the amino acid sequence of SEQ ID NO: 75; and

(f) a V L CDR3 region comprising the amino acid sequence of SEQ ID NO: 76.

2. The method of claim 1 , wherein said subject is a human.

3. A method of treating rheumatoid arthritis, Crohn's disease, psoriasis, multiple sclerosis, chronic obstructive pulmonary disease, ulcerative colitis or asthma in a subject, the method comprising administering an effective amount of an antagonist of IL-17F to a subject in need thereof, wherein said antagonist comprises a monoclonal anti-IL-17F antibody, or an antigen-binding fragment thereof, comprising:

(a) a V H CDR1 region comprising the amino acid sequence of SEQ ID NO: 45;

(b) a V H CDR2 region comprising the amino acid sequence of SEQ ID NO: 46;

(c) a V H CDR3 region comprising the amino acid sequence of SEQ ID NO: 47;

(d) a V L CDR1 region comprising the amino acid sequence of SEQ ID NO: 74;

(e) a V L CDR2 region comprising the amino acid sequence of SEQ ID NO: 75; and

(f) a V L CDR3 region comprising the amino acid sequence of SEQ ID NO: 76.

4. The method of claim 3 , wherein said antibody comprises a heavy chain variable sequence comprising the amino acid sequence of SEQ ID NO: 10, and a light chain variable sequence comprising the amino acid sequence of SEQ ID NO: 12 or 103.

5. The method of claim 3 , wherein said subject is a human.

6. A method of treating, ameliorating, or delaying the progression of an inflammatory arthritic condition, the method comprising administering an effective amount of an anti-IL-17F antibody, or an antigen-binding fragment thereof, to a subject in need thereof, wherein said antibody, or antigen-binding fragment thereof, comprises:

(a) a V H CDR1 region comprising the amino acid sequence of SEQ ID NO: 45;

(b) a V H CDR2 region comprising the amino acid sequence of SEQ ID NO: 46;

(c) a V H CDR3 region comprising the amino acid sequence of SEQ ID NO: 47;

(d) a V L CDR1 region comprising the amino acid sequence of SEQ ID NO: 74;

(e) a V L CDR2 region comprising the amino acid sequence of SEQ ID NO: 75; and

(f) a V L CDR3 region comprising the amino acid sequence of SEQ ID NO: 76.

7. The method of claim 6 , wherein said subject is a human.

8. The method of claim 6 , wherein the arthritic condition is an autoimmune arthritic condition.

9. The method of claim 6 , wherein the inflammatory arthritic condition is rheumatoid arthritis.

10. The method of claim 3 , 1 , or 6 , wherein said antibody is an IgG isotype.

11. The method of claim 10 , wherein said antibody is an IgG1 isotype.

12. The method of claim 3 , 1 , or 6 , wherein said antibody does not bind the IL-17A homodimer.

Continuity (3)
Division 12774188 · May 5, 2010
Provisional Application 61175512 · May 5, 2009
Related Publication 20120164152A1 · Jun 28, 2012