IP Library Granted Patent US 9,765,115
Granted Patent B2
US 9,765,115 · App. 14/007,683 · Granted Sep 19, 2017

Inhibitors of metastasis, methods of generating such inhibitors and their therapeutic applications

Inventor: Girish V. Shah (West Monroe, LA)
Assignee: Board of Supervisors of Louisiana State University acting on behalf of the University of Louisiana at Monroe
C07K7/08A61K31/4166A61K38/1796C07D233/76C07K14/723C07K16/28C07K16/2869C12N15/1055G01N33/5011G01N33/5032
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Quick Facts
Patent No.
US 9,765,115
App. No.
14/007,683
Granted
Sep 19, 2017
Kind
B2
Abstract

The chain of events that explains calcitonin receptor (CTR)-stimulated invasion and metastasis of prostate cancer cells was identified. The CTR-stimulated events depend on the interaction of CTR with the PDZ3 domain of ZO-1. Small peptides and small molecules were identified that inhibit this interaction. The small inhibitory peptides were synthesized and can be used to attenuate or inhibit metastasis in solid cancer tumors.

Claims (14)

1. A method to inhibit metastasis in a cancerous solid tumor with tumor cells that have a receptor with a C-terminal PDZ binding motif, and for which metastasis depends on disassembly of cellular tight junctions; said method comprising administering a therapeutic amount of a peptide selected from the group consisting of a peptide whose amino acid sequence has 90% or greater sequence identity with SEQ ID NO:2, and a peptide whose amino acid sequence has 90% or greater sequence identity with SEQ ID NO:3; wherein each amino acid, if any, in said peptide that is not identical to the corresponding amino acid of SEQ ID NO:2 or SEQ ID NO:3 is a conservative substitution; wherein said peptide inhibits interaction between the tumor cell receptor's PDZ binding motif and the PDZ motif of zonula occludens-1; whereby the disassembly of cellular tight junctions of the tumor cells is inhibited; whereby metastasis of the tumor is inhibited.

2. The method of claim 1 , wherein the receptor is a calcitonin receptor.

3. The method of claim 1 , wherein the compound is a peptide whose amino acid sequence has 90% or greater sequence identity with SEQ ID NO:2.

4. The method of claim 1 , wherein the compound is a peptide whose amino acid sequence has 90% or greater sequence identity with SEQ ID NO:3.

5. The method of claim 1 , wherein the tumor cells express zonula occludens-1 having a PDZ domain with an amino acid sequence selected from the group consisting of SEQ ID NO:9, SEQ ID NO:10, and SEQ ID NO:11.

6. The method of claim 5 , wherein the PDZ domain of zonula occludens-1 has the amino acid sequence SEQ ID NO:11.

7. The method of claim 1 , wherein the cancerous tumor is selected from the group consisting of prostate carcinoma, bladder carcinoma, esophagus cancer, squamous cell carcinoma, adenocarcinomas of lung, ovarian adenocarcinoma, pancreatic carcinoma, and rectal carcinoma.

8. The method of claim 1 , wherein the cancerous tumor is prostate carcinoma.

9. The method of claim 1 , wherein the compound is a peptide whose amino acid sequence is SEQ ID NO:2.

10. The method of claim 1 , wherein the compound is a peptide whose amino acid sequence is SEQ ID NO:3.

11. The method of claim 1 , wherein the cancerous tumor is prostate carcinoma, and wherein the compound is a peptide whose amino acid sequence is SEQ ID NO:2.

12. The method of claim 1 , wherein the cancerous tumor is prostate carcinoma, and wherein the compound is a peptide whose amino acid sequence is SEQ ID NO:3.

13. The method of claim 5 , wherein the PDZ domain of zonula occludens-1 has the amino acid sequence SEQ ID NO:9.

14. The method of claim 5 , wherein the PDZ domain of zonula occludens-1 has the amino acid sequence SEQ ID NO:10.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jan 22, 2014
From: SHAH, GIRISH V.
To: BOARD OF SUPERVISORS FOR THE UNIVERSITY OF LOUISIANA SYSTEM, ACTING ON BEHALF OF THE UNIVERSITY OF LOUISIANA AT MONROE
Reel/Frame 032014/0384 →
Continuity (2)
Provisional Application 61470678 · Apr 1, 2011
Related Publication 20140286952A1 · Sep 25, 2014