IP Library Granted Patent US 10,000,511
Granted Patent B2
US 10,000,511 · App. 14/705,764 · Granted Jun 19, 2018

Small molecules for the modulation of MCL-1 and methods of modulating cell death, cell division, cell differentiation and methods of treating disorders

Inventors: Loren D. Walensky (Newton Centre, MA); Michelle L. Stewart (Brookline, MA); Nicole Cohen (Newton, MA)
Assignee: Dana-Farber Cancer Institute, Inc.
C07D513/04C07C251/20C07C251/84C07C311/20C07C335/16C07C337/08C07D239/60C07D249/12C07D277/34C07D279/06C07D307/54C07D327/04C07D401/12C07D403/04C07D403/06C07D403/08C07D405/12C07D405/14C07D409/06C07D409/12C07D409/14C07D413/06C07D413/14C07D417/04C07D417/14C07C2602/10
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Quick Facts
Patent No.
US 10,000,511
App. No.
14/705,764
Granted
Jun 19, 2018
Kind
B2
Abstract

This invention relates to compounds which selectively bind to the survival protein MCL-1 with high affinity and selectivity, pharmaceutical compositions containing such compounds and the use of those compounds or compositions for modulating MCL-1 activity and for treating hyperproliferative disorders, angiogenesis disorders, cell cycle regulation disorders, autophagy regulation disorders, inflammatory disorders, and/or infectious disorders and/or for enhancing cellular engraftment and/or wound repair, as a sole agent or in combination with other active ingredients.

Claims (27)

1. A pharmaceutical composition comprising:

a pharmaceutically acceptable diluent or carrier; and

a compound of formula (J) or a pharmaceutically acceptable salt, solvate, hydrate or stereoisomer thereof:

wherein

R 22 is substituted or unsubstituted aryl, wherein R 22 , when substituted, is substituted by hydroxyl or halogen;

R 23 is C 1 -C 8 alkyl, C 2 -C 8 alkenyl, C 2 -C 8 alkynyl, C 1 -C 6 alkoxy, substituted or unsubstituted aryl, substituted or unsubstituted heteroaryl, substituted or unsubstituted cycloalkyl, or substituted or unsubstituted heterocycloalkyl, wherein R 23 , when substituted, is substituted by Z;

R 24 is C 1 -C 8 alkyl, C 2 -C 8 alkynyl, C 1 -C 6 alkoxy, substituted or unsubstituted aryl, substituted or unsubstituted heteroaryl, substituted or unsubstituted cycloalkyl, or substituted or unsubstituted heterocycloalkyl, wherein R 24 , when substituted, is substituted by Z; and

each Z, independently, is halogen, hydroxyl, C 1 -C 8 alkyl, C 2 -C 8 alkenyl, C 2 -C 8 alkynyl, C 1 -C 6 alkoxy, substituted or unsubstituted aryl, substituted or unsubstituted heteroaryl, substituted or unsubstituted cycloalkyl, or substituted or unsubstituted heterocycloalkyl;

provided that the compound is not

2. The composition of claim 1 , wherein R 22 is hydroxylphenyl, dihydroxylphenyl, trihydroxylphenyl or iodophenyl.

3. The composition of claim 1 , wherein R 23 is C 1 -C 8 alkyl, aryl, heteroaryl or heterocycloalkyl, in which each of C 1 -C 8 alkyl, aryl, heteroaryl and heterocycloalkyl is unsubstituted or substituted by Z.

4. The composition of claim 3 , wherein R 23 is C 1 -C 8 alkyl; unsubstituted heteroaryl; benzoxazinonyl; or aryl substituted by halogen, C 1 -C 8 alkyl or C 1 -C 6 alkoxy.

5. The composition of claim 4 , wherein R 23 is methyl, thienyl, furyl, fluorophenyl, dichlorophenyl, dimethylphenyl, difluoromethoxyphenyl, or benzoxazinonyl.

6. The composition of claim 1 , wherein R 24 is C 1 -C 8 alkyl, aryl, or cycloalkyl, in which each of C 1 -C 8 alkyl, aryl, and cycloalkyl is unsubstituted or substituted by Z.

7. The composition of claim 6 , wherein R 24 is C 1 -C 8 alkyl, C 1 -C 8 alkyl substituted by phenyl, aryl substituted by halogen, or cycloalkyl.

8. The composition of claim 7 , wherein R 24 is methyl, ethyl, isopropyl, benzyl, cyclohexyl or fluorophenyl.

9. A pharmaceutical composition, comprising:

or a physiologically acceptable salt, solvate, hydrate or stereoisomer thereof; and

a pharmaceutically acceptable diluent or carrier.

10. A method for modulating apoptotic cell death in a cell, comprising contacting the cell with the composition of claim 9 , thereby regulating apoptotic cell death in said cell.

11. A method for modulating metabolism in a cell, comprising contacting the cell with the composition of claim 9 , thereby regulating energy production and consumption to effect cell viability in said cell.

12. A method of treating the hyperproliferative disorder in a mammal, comprising administering to a mammal in need thereof, a therapeutically effective amount of the composition of claim 9 .

13. The method of claim 12 , wherein the hyperproliferative disorder is cancer.

14. A method for modulating apoptotic cell death in a cell, comprising contacting the cell with the composition of claim 1 , thereby regulating apoptotic cell death in said cell.

15. A method for modulating metabolism in a cell, comprising contacting the cell with the composition of claim 1 , thereby regulating energy production and consumption to effect cell viability in said cell.

16. A method of treating a hyperproliferative disorder in a mammal, comprising administering to the mammal in need thereof, a therapeutically effective amount of the composition of claim 1 .

17. The method of claim 16 , wherein the hyperproliferative disorder is cancer.

Assignments (2)
CONFIRMATORY LICENSE Recorded Dec 5, 2016
From: DANA-FARBER CANCER INST
To: NATIONAL INSTITUTES OF HEALTH (NIH), U.S. DEPT. OF HEALTH AND HUMAN SERVICES (DHHS), U.S. GOVERNMENT
Reel/Frame 040807/0168 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded May 6, 2015
From: WALENSKY, LOREN D.; STEWART, MICHELLE L.; COHEN, NICOLE
To: DANA-FARBER CANCER INSTITUTE, INC.
Reel/Frame 035580/0822 →
Continuity (3)
Division 13576116
Provisional Application 61299803 · Jan 29, 2010
Related Publication 20150315211A1 · Nov 5, 2015
Cited By (1)
US 12,398,178