IP Library Granted Patent US 10,111,901
Granted Patent B2
US 10,111,901 · App. 15/386,887 · Granted Oct 30, 2018

Beta-glucan in combination with anti-cancer agents affecting the tumor microenvironment

Inventors: Nandita Bose (Plymouth, MN); Keith Gorden (Woodbury, MN); Anissa S H. Chan (Arden Hills, MN); Steven Leonardo (Rosemount, MN); Jeremy Graff (Indianapolis, IN); Xiaohong Qiu (Edina, MN); Takashi Kangas (Shoreview, MN); Kathryn A. Fraser (St. Paul, MN); Adria Bykowski Jonas (Eden Prairie, MN); Nadine Ottoson (Lakeville, MN); Ross Fulton (St. Paul, MN)
Assignee: BIOTHERA, INC.
A61K31/716A61K9/0019A61K39/3955C07K16/2827C07K16/303C07K16/3015C07K16/3023C07K16/3038C07K16/3046C07K16/3053C07K16/3061C07K16/44A61K2039/505
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Quick Facts
Patent No.
US 10,111,901
App. No.
15/386,887
Granted
Oct 30, 2018
Kind
B2
Abstract

The present invention relates to the combination of soluble β-glucan and anti-cancer agents that affect the tumor microenvironment. Soluble β-glucan promotes an immunostimulatory environment, which allows enhanced effectiveness of anti-cancer agents.

Claims (19)

1. A method of treating a subject having cancer, the method comprising administering soluble β(1,6)-[poly-(1,3)-D-glucopyranosyl]-poly-β(1,3)-D-glucopyranose and an anti-PD-L1 antibody, wherein the anti-PD-L1 antibody is a non-complement-activating antibody.

2. The method according to claim 1 , wherein the cancer is melanoma, renal cell carcinoma, or lung cancer.

3. The method according to claim 1 , wherein the cancer is breast cancer, pancreatic cancer, colon cancer, or B cell lymphoma.

4. The method according to claim 1 , wherein the β(1,6)-[poly-(1,3)-D-glucopyranosyl]-poly-β(1,3)-D-glucopyranose and the anti-PD-L1 antibody are in a single formulation.

5. The method according to claim 1 , wherein the β(1,6)-[poly-(1,3)-D-glucopyranosyl]-poly-β(1,3)-D-glucopyranose and the anti-PD-L1 antibody are in separate formulations.

6. The method according to claim 1 , wherein the β(1,6)-[poly-(1,3)-D-glucopyranosyl]-poly-β(1,3)-D-glucopyranose is derived from yeast.

7. The method according to claim 6 , wherein the yeast is Saccharomyces cerevisiae.

8. The method according to claim 1 , wherein the β(1,6)-[poly-(1,3)-D-glucopyranosyl]-poly-β(1,3)-D-glucopyranose stimulates the subject's immune system.

9. The method according to claim 1 , wherein the anti-PD-L1 antibody is an Fc-engineered IgG 1 antibody.

10. The method according to claim 1 , wherein the anti-PD-L1 antibody is an IgG 4 antibody.

11. The method according to claim 1 , wherein the β(1,6)-[poly-(1,3)-D-glucopyranosyl]-poly-β(1,3)-D-glucopyranose and the anti-PD-L1 antibody are administered intravenously.

12. The method according to claim 1 , wherein the method further comprises administration of a tumor targeting antibody.

13. The method according to claim 1 , wherein the method further comprises beta-glucan antibodies.

14. The method according to claim 1 , wherein the subject has high response toward soluble β(1,6)-[poly-(1,3)-D-glucopyranosyl]-poly-β(1,3)-D-glucopyranose.

15. A method of stimulating a subject's immune system against cancer cells, the method comprising administering soluble β(1,6)-[poly-(1,3)-D-glucopyranosyl]-poly-β(1,3)-D-glucopyranose and an anti-PD-L1 antibody, wherein the anti-PD-L1 antibody is a non-complement-activating antibody.

16. The method according to claim 15 , wherein the immune stimulation comprises activation of M1 macrophages, N1 neutrophils, NK cells, T cells, B cells or dendritic cells.

17. The method according to claim 15 , wherein the immune stimulation comprises activation of interleukin-12, interferon-γ, tumor-necrosis factor α, or a combination thereof.

18. A method of removing immune suppression in a tumor microenvironment, the method comprising administering soluble β(1,6)-[poly-(1,3)-D-glucopyranosyl]-poly-β(1,3)-D-glucopyranose and an anti-PD-L1 antibody, wherein the anti-PD-L1 antibody is a non-complement-activating antibody.

19. The method according to claim 18 , wherein the method comprises suppression of M2 macrophages, N2 neutrophils, myeloid-derived suppressor cells, or a combination thereof.

Assignments (2)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jul 10, 2020
From: BIOTHERA, INC.
To: HIBERCELL, INC.
Reel/Frame 053177/0368 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Apr 19, 2017
From: BOSE, NANDIT; GORDEN, KEITH; CHAN, ANISSA SH.; LEONARDO, STEVEN; GRAFF, JEREMY; QIU, XIAOHONG; KANGAS, TAKASHI; FRASER, KATHRYN A.; BYKOWSKI JONAS, ADRIA; OTTOSON, NADINE; FULTON, ROSS
To: BIOTHERA, INC.
Reel/Frame 042062/0456 →
Continuity (6)
Continuation PCTUS2015039977 · Jul 10, 2015
Provisional Application 62149892 · Apr 20, 2015
Provisional Application 62115895 · Feb 13, 2015
Provisional Application 62076094 · Nov 6, 2014
Provisional Application 62022754 · Jul 10, 2014
Related Publication 20170100425A1 · Apr 13, 2017
Cited By (2)
US 12,377,116 US 12,533,373