IP Library › Granted Patent US 10,195,292
Granted Patent B2
US 10,195,292 · App. 15/608,350 · Granted Feb 5, 2019

Method of gene transfer for the treatment of recessive catecholaminergic polymorphic ventricular tachycardia (CPVT)

Inventors: Silvia G. Priori (Milan, IT); Carlo Napolitano (Milan, IT); Marco Denegri (Stradella, IT)
Assignee: Istituti Clinici Scientifici Maugeri SpA SB
A61K48/005C07K14/4728
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Quick Facts
Patent No.
US 10,195,292
App. No.
15/608,350
Granted
Feb 5, 2019
Kind
B2
Abstract

The present invention concerns a method for the treatment of recessive Catecholaminergic Polymorphic Ventricular Tachycardia comprising delivering a gene into a cardiac cell.

Claims (9)

1. A method of treating recessive catecholaminergic polymorphic ventricular tachycardia (CPVT) in a human patient that exhibits arrhythmia and has a deleterious mutation in the gene encoding calsequestrin 2 (CASQ2) protein, wherein said method comprises introducing an adeno-associated viral (AAV) vector comprising a nucleic acid encoding a wild-type CASQ2 protein into a cardiomyocyte in said patient, wherein the amino acid sequence of said CASQ2 protein is at least 95% identical to the amino acid sequence of SEQ ID NO: 1 or SEQ ID NO: 2, wherein administration of said AAV vector to said patient results in an antiarrhythmic effect on said patient, thereby treating CPVT in the patient.

2. The method of claim 1 , wherein said AAV vector is an AAV2/9 vector.

3. The method of claim 1 , wherein the amino acid sequence of said CASQ2 protein is at least 95% identical to the amino acid sequence of SEQ ID NO: 2.

4. The method of claim 1 , wherein said mutation is a homozygous mutation.

5. The method of claim 4 , wherein said homozygous mutation is R33Q.

6. The method of claim 1 , wherein physiological levels of expression of CASQ2 are restored following administration of said AAV vector to said patient.

7. The method of claim 1 , wherein physiological abundance and spatial organization of triadin and junctin are restored following administration of said AAV vector to said patient.

8. The method of claim 1 , wherein intracellular Ca 2+ storage and Ca 2+ fluxes are restored following administration of said AAV vector to said patient.

9. The method of claim 1 , wherein said anti-arrhythmic effect is assessed by electrocardiography.

Assignments (2)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jun 6, 2018
From: PRIORI, SILVIA G.; NAPOLITANO, CARLO; DENEGRI, MARCO
To: FONDAZIONE SALVATORE MAUGERI CLINICA DEL LAVORO E DELLA RIABILITAZIONE (ITALIAN RESEARCH FOUNDATION)
Reel/Frame 045999/0398 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jun 6, 2018
From: FONDAZIONE SALVATORE MAUGERI CLINICA DEL LAVORO E DELLA RIABILITAZIONE (ITALIAN RESEARCH FOUNDATION)
To: ISTITUTI CLINICI SCIENTIFICI MAUGERI SPA SB
Reel/Frame 046000/0875 →
Continuity (4)
Continuation 14294674 · Jun 3, 2014
Continuation 13569490 · Aug 8, 2012
Provisional Application 61521076 · Aug 8, 2011
Related Publication 20170360957A1 · Dec 21, 2017