IP Library › Granted Patent US 10,259,879
Granted Patent B2
US 10,259,879 · App. 15/074,505 · Granted Apr 16, 2019

Anti-CD154 antibodies having impaired FcR binding and/or complement binding properties

Inventor: Randolph J. Noelle (Plainfield, NH)
Assignee: THE TRUSTEES OF DARTMOUTH COLLEGE
C07K16/2875A61K2039/505C07K2317/24C07K2317/41C07K2317/52C07K2317/565C07K2317/71C07K2317/732
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Quick Facts
Patent No.
US 10,259,879
App. No.
15/074,505
Granted
Apr 16, 2019
Kind
B2
Abstract

Improved anti-CD154 antibodies are provided herein which have ablated FcR binding and/or complement binding/activation. The use of these antibodies for inducing tolerance and treating immune diseases including autoimmunity, inflammation and allergic disorders is disclosed herein.

Claims (16)

1. A human or humanized antibody of the human IgG1 isotype that specifically binds human CD154 which contains light chain CDR1, CDR2 and CDR3 polypeptides respectively comprising the polypeptide sequences of SEQ ID NO's: 17, 18 and 19, and heavy chain CDR1, CDR2 and CDR3 polypeptides respectively comprising the polypeptide sequences of SEQ ID NO's: 24, 25 and 26, wherein the Fc region of the IgG1 antibody contains an E269R mutation and further contains another mutation which eliminates C 1q binding activity, which mutation is selected from the group consisting of K322A, P331G, and P331G/K322A.

2. The human or humanized antibody of claim 1 , wherein said antibody does not elicit thrombosis in a transgenic mouse that expresses human FcγRIIa (FCGR2A mouse).

3. The human or humanized antibody of claim 1 , wherein the mutation in the Fc region which eliminates C 1q binding activity is a K322A mutation.

4. The antibody of claim 1 , which contains a humanized V L polypeptide comprising a sequence selected from SEQ ID NO:13, 14, 15 and 16, and a humanized V H polypeptide comprising a sequence selected from SEQ ID NO:20, 21, 22 and 23.

5. The human or humanized antibody of claim 3 , which contains a humanized V L polypeptide comprising a sequence selected from SEQ ID NO:13, 14, 15 and 16, and a humanized V H polypeptide comprising a sequence selected from SEQ ID NO:20, 21, 22 and 23.

6. A composition suitable for human immunotherapy comprising a therapeutically effective amount of the human or humanized antibody according to claim 1 .

7. A composition suitable for human immunotherapy comprising a therapeutically effective amount of the human or humanized antibody according to claim 3 .

8. A composition suitable for human immunotherapy comprising a therapeutically effective amount of the human or humanized antibody according to claim 5 .

9. The antibody of claim 1 wherein the mutation which eliminates C 1q binding activity is a P331G mutation.

10. The antibody of claim 1 wherein the mutation which eliminates C 1q binding activity is a P331G/K322A mutation.

11. A composition suitable for human immunotherapy comprising a therapeutically effective amount of a human or humanized antibody according to claim 9 .

12. A composition suitable for human immunotherapy comprising a therapeutically effective amount of a human or humanized antibody according to claim 10 .

13. The antibody of claim 9 which contains a humanized V L polypeptide comprising a sequence selected from SEQ ID NO:13, 14, 15 and 16, and a humanized V H polypeptide comprising a sequence selected from SEQ ID NO:20, 21, 22 and 23.

14. The antibody of claim 10 which contains a humanized V L polypeptide comprising a sequence selected from SEQ ID NO:13, 14, 15 and 16, and a humanized V H polypeptide comprising a sequence selected from SEQ ID NO:20, 21, 22 and 23.

15. A composition suitable for human immunotherapy comprising a therapeutically effective amount of a humanized antibody according to claim 13 .

16. A composition suitable for human immunotherapy comprising a therapeutically effective amount of a humanized antibody according to claim 14 .

Continuity (4)
Division 13648808 · Oct 10, 2012
Continuation In Part 13439186 · Apr 4, 2012
Provisional Application 61471287 · Apr 4, 2011
Related Publication 20160333102A1 · Nov 17, 2016