IP Library › Granted Patent US 10,617,757
Granted Patent B2
US 10,617,757 · App. 15/502,182 · Granted Apr 14, 2020

Methods for treating multiple myeloma

Inventor: Jeffrey M. Venstrom (San Francisco, CA)
Assignee: The Regents of the University of California
A61K39/39558A61K31/454A61K31/573C07K16/2896C07K16/3061C12Q1/6881C12Q1/6886C07K2317/24C07K2317/51C07K2317/56C07K2317/565C07K2317/567C07K2317/732C07K2317/92C12Q2600/106C12Q2600/158C12Q2600/172
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Quick Facts
Patent No.
US 10,617,757
App. No.
15/502,182
Granted
Apr 14, 2020
Kind
B2
Abstract

The present disclosure provides methods of treating multiple myeloma in an individual, the methods comprising genotyping an HLA allele and a KIR allele in the individual; and, depending on the outcome of the genotyping, administering a multiple myeloma therapy to the individual. In some embodiments, the multiple myeloma therapy comprises an anti-CD38 antibody, lenalidomide, and dexamethasone. The present disclosure provides methods for selecting a multiple myeloma patient for a multiple myeloma therapy. The present disclosure provides methods for identifying a multiple myeloma patient as likely to experience a beneficial clinical outcome from a multiple myeloma therapy.

Claims (13)

1. A method of treating an individual having multiple myeloma, the method comprising:

a) determining a genotype of a KIR allele of the individual;

b) determining a genotype of an HLA allele of the individual, and

c) when said determining of steps (a) and (b) indicates that the individual has a 3DL1 KIR allele and an HLA-B Bw4-I80 allele, administering a treatment to the individual, wherein the treatment comprises an anti-CD38 antibody, lenalidomide, and dexamethasone, and

wherein the anti-CD38 antibody comprises VHCDR1 of SEQ ID NO:1 (DYWMQ), VH CDR2 of SEQ ID NO:2 (TIYPGDGDTGYAQKFQG), VH CDR3 of SEQ ID NO:3 (GDYYGSNSLDY), VL CDR1 of SEQ ID NO:4 (KASQDVSTWA), VL CDR2 of SEQ ID NO:5 (SASYRYI), and VL CDR3 of SEQ ID NO:6 (QQHYSPPYT).

2. The method of claim 1 , wherein the anti-CD38 antibody kills a CD38 + cell by apoptosis, by antibody-dependent cell-mediated cytotoxicity (ADCC), or by complement-dependent cytotoxicity (CDC).

3. The method of claim 1 , wherein the anti-CD38 antibody binds CD38 with a kD of 3×10 −9 or greater.

4. The method of claim 1 , wherein the anti-CD38 antibody comprises a humanized heavy chain framework region and/or a humanized light chain framework region.

5. The method of claim 1 , wherein the anti-CD38 antibody comprises a VH region comprising the amino acid sequence set forth in SEQ ID NO:7, and comprises a VL region comprising the amino acid sequence set forth in SEQ ID NO:8.

6. The method of claim 1 , wherein the anti-CD38 antibody comprises a heavy chain region of the isotype IgG1, IgG2, IgG3, or IgG4.

7. The method of claim 1 , further comprising determining the copy number of the HLA-B Bw 4-I80 allele.

8. The method of claim 1 , wherein said administering increases progression-free survival (PFS) and/or increases time to progression (TTP).

9. The method of claim 1 , wherein the multiple myeloma is relapsed refractory multiple myeloma.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Sep 1, 2017
From: VENSTROM, JEFFREY M.
To: THE REGENTS OF THE UNIVERSITY OF CALIFORNIA
Reel/Frame 043749/0686 →
Continuity (2)
Provisional Application 62035304 · Aug 8, 2014
Related Publication 20170224817A1 · Aug 10, 2017