IP Library › Granted Patent US 10,731,178
Granted Patent B2
US 10,731,178 · App. 15/613,646 · Granted Aug 4, 2020

CNS targeting AAV vectors and methods of use thereof

Inventors: Guangping Gao (Westborough, MA); Hongwei Zhang (Worcester, MA); Hongyan Wang (Worcester, MA); Zuoshang Xu (Wellesley, MA)
Assignee: University of Massachusetts
C12N15/86A61K31/713A61K38/50A61K48/0075A61P25/00C12N7/00C12N9/80C12N15/1137C12N15/8645C12Y305/01015A61K48/00A61K48/0058C12N15/635C12N2310/141C12N2750/14133C12N2750/14141C12N2750/14143C12N2750/14145C12N2750/14162C12N2810/10C12N2840/007
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Quick Facts
Patent No.
US 10,731,178
App. No.
15/613,646
Granted
Aug 4, 2020
Kind
B2
Abstract

The invention in some aspects relates to recombinant adeno-associated viruses useful for targeting transgenes to CNS tissue, and compositions comprising the same, and methods of use thereof. In some aspects, the invention provides methods and compositions for treating CNS-related disorders.

Claims (16)

1. A method for treating amyotrophic lateral sclerosis (ALS) in a subject in need thereof, the method comprising:

administering an effective amount of a rAAV to CNS tissue of the subject, wherein the rAAV comprises: (i) a capsid protein comprising a sequence as set forth in SEQ ID NO: 9; and (ii) a nucleic acid comprising a promoter operably linked with a region encoding an inhibitory RNA that binds specifically to SOD1 mRNA and inhibits expression of SOD1 in the subject, wherein the inhibitory RNA comprises the sequence as set forth in SEQ ID NO: 26.

2. The method of claim 1 , wherein the inhibitory RNA is an antisense RNA, a shRNA or a miRNA.

3. A method for treating amyotrophic lateral sclerosis (ALS) in a subject in need thereof, the method comprising:

administering an effective amount of a rAAV to the subject, wherein the rAAV comprises a nucleic acid comprising a promoter operably linked with a region encoding the sequence as set forth in SEQ ID NO: 26 and wherein the rAAV infects cells of CNS tissue in the subject.

4. The method of claim 1 , wherein the rAAV is administered at a dose in a range of 10 10 genome copies to 10 11 genome copies.

5. The method of claim 1 , wherein the CNS tissue is selected from cortex, hippocampus, thalamus, hypothalamus, cerebellum, brain stem, cervical spinal cord, thoracic spinal cord, and lumbar spinal cord.

6. The method of claim 1 , wherein the promoter comprises a chicken beta-actin (CBA) promoter.

7. The method of claim 6 , wherein the promoter is a CAG promoter.

8. The method of claim 1 , wherein the promoter is a tissue-specific promoter.

9. The method of claim 8 , wherein the tissue-specific promoter is a neuron-specific promoter.

10. The method of claim 3 , wherein the CNS tissue is selected from cortex, hippocampus, thalamus, hypothalamus, cerebellum, brain stem, cervical spinal cord, thoracic spinal cord, and lumbar spinal cord.

11. The method of claim 3 , wherein the promoter comprises a chicken beta-actin (CBA) promoter.

12. The method of claim 11 , wherein the promoter is a CAG promoter.

13. The method of claim 3 , wherein the promoter is a tissue-specific promoter.

14. The method of claim 13 , wherein the tissue-specific promoter is a neuron-specific promoter.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Aug 31, 2017
From: GAO, GUANGPING; ZHANG, HONGWEI; WANG, HONGYAN; XU, ZUOSHANG
To: UNIVERSITY OF MASSACHUSETTS
Reel/Frame 043455/0833 →
Continuity (5)
Continuation 14445670 · Jul 29, 2014
Continuation 13642719
Provisional Application 61327627 · Apr 23, 2010
Related Publication 20170349911A1 · Dec 7, 2017
Related Publication 20180148738A9 · May 31, 2018