IP Library Granted Patent US 11,890,340
Granted Patent B2
US 11,890,340 · App. 17/051,678 · Granted Feb 6, 2024

Use of epithelial membrane protein 2 [EMP2] targeting agents in treating lung disorders

Inventors: Michael Brian Fessler (Cary, NC); Carmen J. Williams (Chapel Hill, NC); Wan-Chi Lin (Morrisville, NC)
Assignee: The United States of America as Represented By The Secretary of the Department of Health and Human Services
A61K39/3955A61K31/7088A61K31/713A61P11/00A61K45/06A61K2039/505C07K14/705C07K16/28C07K2317/24C07K2317/50C07K2317/54C07K2317/55C07K2317/622C07K2317/626C12N15/113G01N33/6893G01N2333/47
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Quick Facts
Patent No.
US 11,890,340
App. No.
17/051,678
Granted
Feb 6, 2024
Kind
B2
Abstract

Disclosed herein are methods of treating or preventing a lung disorder comprising administering to a subject a composition comprising an agent that modulates activity and/or expression of Epithelial Membrane Protein 2 (EMP2) in an amount effective to treat or prevent the lung disorder and compositions useful in such for methods.

Claims (36)

1. A method of treating or preventing a lung disorder in a subject in need thereof, comprising administering to the subject a composition comprising an agent that modulates the activity and/or expression of Epithelial Membrane Protein 2 (EMP2),

wherein the agent is

(a) an anti-EMP2 antibody selected from the group consisting of PG101, KS83, KS49, KS41, KS89, and a combination thereof, or an antigen-binding fragment thereof; or

(b) a small interfering RNA (siRNA) capable of binding to an EMP2-encoding nucleotide sequence; or

(c) an antisense oligonucleotide capable of binding to the EMP2-encoding nucleotide sequence, and

wherein the lung disorder is

(i) an acute disease selected from the group consisting of acute lung injury, acute respiratory distress syndrome, pneumonia, viral infection, and airway hyperresponsiveness;

(ii) a neutrophil-dependent lung disorder;

(iii) an epithelial lipid raft-dependent and/or caveolae-dependent lung disease;

(iv) a TGF-β (Transforming growth factor beta)-mediated lung disorder selected from the group consisting of pulmonary fibrosis and acute lung injury;

(v) an eosinophil-dependent lung disorder selected from the group consisting of asthma, acute eosinophilic pneumonia, and chronic eosinophilic pneumonia; or a monocyte-dependent lung disorder selected from the group consisting of lung fibrosis and acute lung injury; or a lymphocyte-dependent lung disorder selected from the group consisting of lymphocytic interstitial pneumonia and lymphocytic bronchiolitis;

(vi) idiopathic pulmonary fibrosis comprising epithelium-matrix interaction;

(vii) a chronic disease selected from the group consisting of chronic obstructive pulmonary disease (COPD), bronchiectasis, radiation- or chemotherapeutic-induced pneumonitis, idiopathic or induced interstitial lung disease, bronchopulmonary dysplasia, and lung fibrosis;

(viii) a lung disorder due to exposure to a toxic agent selected from the group consisting of a bioterroristic agent, an occupational hazardous agent and an environmental pollutant or

(ix) chemical pneumonitis due to chemical or acid or hydrocarbon aspiration; or chemical pneumonitis due to smoke inhalation.

2. The method of claim 1 , wherein the anti-EMP2 antibody is KS83.

3. The method of claim 1 , wherein the composition is administered to the subject systemically.

4. The method of claim 1 , wherein the composition is administered to the subject parenterally.

5. The method claim 1 , wherein the composition is administered to the subject topically, intranasally, intravenously, subcutaneously, intramuscularly, intradermally, or intraperitoneally.

6. The method of claim 1 , wherein the composition is administered to the subject by inhalation.

7. The method of claim 6 , wherein the composition is administered by a nebulizer or an inhaler.

8. The method of claim 1 , wherein the composition is formulated as a nasal spray, gel, ointment, liquid, suspension, aerosol, tablet, pill or powder.

9. The method of claim 1 , wherein the agent is in an amount sufficient to reduce intra-airway leukocyte accumulation in the subject.

10. The method of claim 1 , wherein the subject is an infant at risk for acute lung injury due to meconium aspiration or prematurity.

11. A method to target a molecule to alveolar epithelial type 1 cells in a subject with a lung disorder comprising co-administering the molecule with an agent that binds EMP2,

wherein the agent is an anti-EMP2 antibody selected from the group consisting of PG101, KS83, KS49, KS41, KS89, and a combination thereof, or an antigen-binding fragment thereof,

wherein the lung disorder is

(i) an acute disease selected from the group consisting of acute lung injury, acute respiratory distress syndrome, pneumonia, viral infection, and airway hyperresponsiveness;

(ii) a neutrophil-dependent lung disorder;

(iii) an epithelial lipid raft-dependent and/or caveolae-dependent lung disease;

(iv) a TGF-β (Transforming growth factor beta)-mediated lung disorder selected from the group consisting of pulmonary fibrosis and acute lung injury;

(v) an eosinophil-dependent lung disorder selected from the group consisting of asthma, acute eosinophilic pneumonia, and chronic eosinophilic pneumonia; or a monocyte-dependent lung disorder selected from the group consisting of lung fibrosis and acute lung injury; or a lymphocyte-dependent lung disorder selected from the group consisting of lymphocytic interstitial pneumonia and lymphocytic bronchiolitis;

(vi) idiopathic pulmonary fibrosis comprising epithelium-matrix interaction;

(vii) a chronic disease selected from the group consisting of chronic obstructive pulmonary disease (COPD), bronchiectasis, radiation- or chemotherapeutic-induced pneumonitis, idiopathic or induced interstitial lung disease, bronchopulmonary dysplasia, and lung fibrosis;

(viii) a lung disorder due to exposure to a toxic agent selected from the group consisting of a bioterroristic agent, an occupational hazardous agent and an environmental pollutant or

(ix) chemical pneumonitis due to chemical or acid or hydrocarbon aspiration; or chemical pneumonitis due to smoke inhalation.

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Oct 23, 2023
From: FESSLER, MICHAEL BRIAN; WILLIAMS, CARMEN J.; LIN, WAN-CHI
To: THE UNITED STATES OF AMERICA, AS REPRESENTED BY THE SECRETARY, DEPARTMENT OF HEALTH & HUMAN SERVICES
Reel/Frame 065312/0386 →
Continuity (3)
Provisional Application 62771326 · Nov 26, 2018
Provisional Application 62664805 · Apr 30, 2018
Related Publication 20210309737A1 · Oct 7, 2021