IP Library Granted Patent US 12,564,608
Granted Patent B2
US 12,564,608 · App. 17/642,140 · Granted Mar 3, 2026

Use of ghost nanovesicles as therapeutics

Inventors: Jan Lötvall (Lysekil, SE); Kyong-su Park (Gothenburg, SE)
Assignee: Exocure Sweden AB
A61K35/28A61K9/0019A61K38/191A61K38/20A61P29/00A61K39/00
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Quick Facts
Patent No.
US 12,564,608
App. No.
17/642,140
Granted
Mar 3, 2026
Kind
B2
Abstract

The present disclosure provides ghost nanovesicles (gNVs) that are deficient in cytosolic components. Methods of making such vesicles and therapeutic uses of such vesicles are also provided. The gNVs may be used in preventing or treating conditions that may benefit from administration of the gNVs. Such conditions include conditions that involve inflammation.

Claims (33)

1 . A method of making ghost nanovesicles (gNVs) deficient in cytoplasmic proteins and nucleic acids, the method comprising:

disrupting mesenchymal stem cells (MSCs) to generate vesicles;

separating the vesicles based on density and isolating nanovesicles;

exposing the isolated nanovesicles to an alkaline pH to open the nanovesicles thereby generating plasma membrane sheets;

purifying the plasma membrane sheets; and

applying energy to the purified plasma membrane sheets sufficient to convert the plasma membrane sheets into gNVs.

2 . The method of claim 1 , wherein the gNVs are large gNVs having a diameter of 100 nm-200 nm, wherein the gNVs are small gNVs having a diameter of 40 nm-100 nm, or wherein the gNVs comprise large gNVs having a diameter of 100 nm-200 nm and small gNVs having a diameter of 40 nm-100 nm.

3 . The method of claim 1 , further comprising adding a therapeutic agent to a composition comprising the purified membrane sheets and applying energy to the composition sufficient to convert the plasma membrane sheets into gNVs comprising the therapeutic agent.

4 . The method of claim 3 , wherein the therapeutic agent comprises a small molecule, a peptide, a nucleic acid, or a polypeptide.

5 . The method of claim 3 , wherein the therapeutic agent is an anti-inflammatory agent.

6 . The method of claim 4 , wherein the therapeutic agent is not an anti-inflammatory agent.

7 . The method of claim 1 , wherein the MSCs are human MSCs.

8 . Ghost nanovesicles (gNVs) derived from mesenchymal stem cells (MSCs), wherein the gNVs are deficient in cytoplasmic proteins and nucleic acids and are made by a method comprising:

disrupting the MSCs to generate vesicles;

separating the vesicles based on density and isolating nanovesicles;

exposing the isolated nanovesicles to an alkaline pH to open the nanovesicles thereby generating plasma membrane sheets;

purifying the plasma membrane sheets; and

applying energy to the purified plasma membrane sheets sufficient to convert the plasma membrane sheets into gNVs.

9 . The gNVs of claim 8 , wherein the MSCs are human MSCs.

10 . The gNVs of claim 8 , comprising a therapeutic agent.

11 . The gNVs of claim 10 , wherein the therapeutic agent is an anti-inflammatory agent.

12 . The gNVs of claim 10 , wherein the therapeutic agent comprises a small molecule, a peptide, a nucleic acid, or a polypeptide and is not an anti-inflammatory agent.

13 . A method for reducing level of a pro-inflammatory cytokine in a subject with an inflammatory related condition, the method comprising:

disrupting mesenchymal stem cells (MSCs) to generate vesicles;

separating the vesicles based on density and isolating nanovesicles;

exposing the isolated nanovesicles to an alkaline pH to open the nanovesicles thereby generating plasma membrane sheets;

purifying the plasma membrane sheets;

applying energy to the purified plasma membrane sheets sufficient to convert the plasma membrane sheets into ghost nanovesicles (gNVs);

administering to the subject an amount of the gNVs effective to reduce the level of the proinflammatory cytokine in the subject,

wherein the pro-inflammatory cytokine comprises tumor necrosis factor alpha (TNF-α) or interleukin-6 (IL-6).

14 . The method of claim 13 , wherein the subject is human and the MSCs are human MSCs.

15 . The method of claim 13 , wherein the pro-inflammatory cytokine is TNF-α.

16 . The method of claim 13 , wherein the pro-inflammatory cytokine is IL-6.

Assignments (3)
CORRECTIVE ASSIGNMENT TO CORRECT THE CORRECT THE NATURE OF CONVEYANCE ON THE COVERSHEET FOR THE ORIGINAL RECORDATION, NATURE OF CONVEYANCE SHOULD BE "ASSIGNMENT" AND NOT "CHANGE OF NAME". PREVIOUSLY RECORDED AT REEL: 64189 FRAME: 174. ASSIGNOR(S) HEREBY CONFIRMS THE ASSIGNMENT. Recorded Nov 12, 2024
From: EXOCURE BIOSCIENCES, INC.
To: EXOCURE SWEDEN AB
Reel/Frame 069530/0601 →
CHANGE OF NAME Recorded Jul 7, 2023
From: EXOCURE BIOSCIENCES, INC.
To: EXOCURE SWEDEN AB
Reel/Frame 064189/0174 →
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Aug 24, 2022
From: LÖTVALL, JAN; PARK, KYONG-SU
To: EXOCURE BIOSCIENCES, INC.
Reel/Frame 060884/0751 →
Continuity (2)
Provisional Application 62899931 · Sep 13, 2019
Related Publication 20220387505A1 · Dec 8, 2022
References Cited (45)
US 9149542B2 · Gho et al. · 2015 [cited by applicant]
US 9220763B2 · Gho et al. · 2015 [cited by applicant]
US 11333665B2 · Lotvall et al. · 2022 [cited by applicant]
US 20080207723A1 · Kopreski · 2008 [cited by applicant]
US 20150086639A1 · Huang · 2015 [cited by applicant]
US 20150218254A1 · Sabbadini et al. · 2015 [cited by applicant]
US 20160061842A1 · Di Vizio · 2016 [cited by applicant]
US 20160120818A1 · Grandi et al. · 2016 [cited by applicant]
US 20180036240A1 · Gho et al. · 2018 [cited by applicant]
US 20180296483A1 · Gho et al. · 2018 [cited by applicant]
US 20180318409A1 · Valiante et al. · 2018 [cited by applicant]
US 20200249234A1 · Lotvall et al. · 2020 [cited by applicant]
US 20220080035A1 · Park et al. · 2022 [cited by applicant]
EP 2617413A2 · 2013 [cited by applicant]
EP 3251659A1 · 2017 [cited by applicant]
EP 2450032B1 · 2018 [cited by applicant]
WO WO2009051427 · 2009 [cited by applicant]
WO WO2009130649 · 2009 [cited by applicant]
WO WO2010010983 · 2010 [cited by applicant]
WO WO2010056337 · 2010 [cited by applicant]
WO WO2010070124 · 2010 [cited by applicant]
WO WO2013063439 · 2013 [cited by applicant]
WO WO2015085096 · 2015 [cited by applicant]
WO WO2016136372 · 2016 [cited by applicant]
WO WO2017161010 · 2017 [cited by applicant]
WO WO2017205810 · 2017 [cited by applicant]
WO WO2018171947 · 2018 [cited by applicant]
WO WO2019022671A1 · 2019 [cited by examiner]
Makrygiannakis et al. Local administration of glucocorticoids decreases synovial citrullination in rheumatoid arthritis. Arthritis Research & Therapy 2012, 14: R20, p. 1-9 (Year: 2012). [cited by examiner]
Lewis et al. Current Murine Models of Sepsis. Surgical Infections vol. 17, No. 4, p. 385-393 (Year: 2016). [cited by examiner]
U.S. Appl. No. 17/731,833, filed Apr. 28, 2022. [cited by applicant]
Coumans et al., (2017) “Methodological guidelines to study extracellular Vesicles”, Circ. Res., 120(10):1632-1648. [cited by applicant]
Jeppesen et al., (2014) “Quantitative proteomics of fractionated membrane and lumen exosome proteins from isogenic metastatic and nonmetastatic bladder cancer cells reveal differential expression of EMT factors”, Proteo… [cited by applicant]
Karimi et al., (2018) “Detailed analysis of the plasma extracellular vesicle proteome after separation from lipoproteins”, Cell. Mol. Life Sci., 75(15):2873-2886. [cited by applicant]
Mariantonia et al., (2009) “High levels of exosomes expressing CD63 and caveolin-1 in plasma of melanoma patients. e5219”, PLOS ONE, 4(4):1-10. [cited by applicant]
Matsushita et al., (1989) “Effect of Extracellular pH on the Respiratory Chain and Energetics of Gluconobacter suboxydans”, Agricultural and Biological Chemistry,53(11):2895-2902. [cited by applicant]
Shin et al., (2015) “High-yield isolation of extracellular vesicles using aqueous two-phase system”, Scientific Reports, (5)1:1-11. [cited by applicant]
Tauro et al., (2012) “Two Distinct Populations of Exosomes Are Released from LIM1863 Colon Carcinoma Cell-derived Organoids”, Molecular & Cellular Proteomics, 12(3):587-598. [cited by applicant]
Yoshioka et al., (2014) “Ultra-sensitive liquid biopsy of circulating extracellular vesicles using ExoScreen”. Nature Communications, (5)3591:1-8. [cited by applicant]
D'Atri et al., (2019) “Nano-Ghosts: mesenchymal stem cells derived nanoparticles as a novel approach for cartilage regeneration.” Journal of Extracellular Vesicles, vol. 8, 1 page. [cited by applicant]
Furman et al., (2013) “Reconstructed Stem Cell Nano ghosts: a Natural Tumor Targeting Platform.” Nano Letters, vol. 13, No. 7, pp. 3248-3255. [cited by applicant]
Go et al., (2018) “Extracellular Vesicle-Mimetic Ghost Nanovesicles for Delivering Anti-Inflammatory Drugs to Mitigate Gram-Negative Bacterial Outer Membrane Vesicle-Induced Systemic Inflammatory Response Syndrome.” Adv… [cited by applicant]
Corrales et al., (2015) “Direct Activation of STING in the Tumor Microenvironment Leads to Potent and Systemic Tumor Regression and Immunity”, Cell Reports, 11(7):1018-1030, XP055771217. [cited by applicant]
Van Der Pol et al. (2015) “Outer membrane vesicles as platform vaccine technology”, Biotechnology Journal, 10(11):1689-1706, XP055465407. [cited by applicant]
Morein et al. (1994) “Separation of inner and outer membrane vesicles from [cited by applicant]