IP Library Granted Patent US 12,629,382
Granted Patent B2
US 12,629,382 · App. 17/763,367 · Granted May 19, 2026

Therapy

Inventors: Hans-René Bjørsvik (Bergen, NO); Per Øyvind Enger (Bergen, NO); Davide Cirillo (Merate, IT); Shahin Sarowar (Nesttun, NO)
Assignee: Vestlandets Innovasjonsselskap AS
A61K31/603A61K31/196A61K31/41A61P35/00
View Patent ↗
Loading inventors, assignments & file history…
Monitor This Case
Get email alerts when status or documents change.
Order Certified Copies
Most orders are placed with the USPTO same day — all within 24 business hours.
Order via The Patent Place →
Pre-filled with this patent's details
Quick Facts
Patent No.
US 12,629,382
App. No.
17/763,367
Granted
May 19, 2026
Kind
B2
Abstract

The invention addresses radioresistance in cancer treatment involving radiotherapy and, in particular, limitations associated with the use of the drug sulfasalazine. Specifically, it provides a series of compounds for use as radiosensitizers in the treatment of cancers such as glioblastomas which are lethal and inherently resistant to radiotherapy. In one embodiment, the invention provides compounds of general formula (I), their stereoisomers and pharmaceutically acceptable salts for use as radiosensitizers in the treatment of cancer: wherein ring A is selected from optionally substituted phenyl, biphenyl and fluorenyl; each X is independently selected from: —C 1-6 alkyl (preferably C 1-3 alkyl, e.g. —CH 3 ), —O—C 1-6 alkyl (preferably —O—C 1-3 alkyl, e.g. —OCH 3 ), —S—C 1-6 alkyl (preferably —S—C 1-3 alkyl, e.g. —SCH 3 ), —OH, —SH, —CO 2 R 1 (where R 1 is H or C 1-6 alkyl, preferably C 1-3 alkyl, e.g. —CH 3 ), —SO 2 —C 1-6 alkyl (preferably —SO 2 —C 1-3 alkyl, e.g. —SO 2 —CH 3 ), —SO 2 —NR 2 R 3 (where R 2 is H and R 3 is optionally substituted phenyl), —NR 4 R 5 (wherein R 4 and R 5 are independently selected from H, C 1-6 alkyl (preferably C 1-3 alkyl, e.g. —CH 3 ), and —CO—C 1-6 alkyl (preferably —CO—C 1-3 alkyl, e.g. —CO—CH 3 ), halogen (e.g. F, Cl or Br), and optionally substituted tetrazolyl; n is an integer from 0 to 5, preferably 0 to 2, e.g. 1 or 2; and denotes an E or Z double bond.

Claims (82)

1 . A method of treating cancer, said method comprising the step of administering to a patient in need thereof a pharmaceutically effective amount of a compound of general formula (I), a stereoisomer, or a pharmaceutically acceptable salt thereof, in combination with radiotherapy:

wherein:

ring A is optionally substituted biphenyl;

each X is independently selected from:

—C 1-6 alkyl,

—O—C 1-6 alkyl,

—S—C 1-6 alkyl,

—OH,

—SH,

—CO 2 R 1 where R 1 is H or C 1-6 alkyl,

—SO 2 —C 1-6 alkyl,

—SO 2 —NR 2 R 3 where R 2 is H and R 3 is optionally substituted phenyl,

—NR 4 R 5 wherein R 4 and R 5 are independently selected from H, C 1-6 alkyl, and —CO—C 1-6 alkyl,

halogen, and

optionally substituted tetrazolyl;

n is an integer from 0 to 5; and

denotes an E or Z double bond.

2 . The method of claim 1 , wherein ring A is unsubstituted biphenyl.

3 . A method of treating cancer, said method comprising the step of administering to a patient in need thereof a pharmaceutically effective amount of a compound, a stereoisomer, or a pharmaceutically acceptable salt thereof, in combination with radiotherapy, wherein said compound is selected from the following and their stereoisomers, and pharmaceutically acceptable salts:

Compound

Compound name and

No.

CAS No. (where appropriate)

Structure

DC01

(E)-2-methoxy-5-styrylbenzoic acid

DC02

(E)-2-hydroxy-5-styrylbenzoic acid CAS 1072937-49-5

DC03

(Z)-2-methoxy-5-styrylbenzoic acid

DC07

(E)-5-(4-fluorostyryl)-2-hydroxybenzoic acid

DC08

(E)-2-hydroxy-5-(4-(methylthio)styryl)benzoic acid

DC10

(E)-5-(2-([1,1′-biphenyl]-4-yl)vinyl)-2-hydroxybenzoic acid

DC12

(E)-2-ammino-5-styrylbenzoic acid

DC13

(E)-2-hydroxy-5-(4-(methylsulfonyl)styryl)benzoic acid

DC15

(E)-4-styryl-2-(1H-tetrazol-5-yl)phenol

DC16

(E)-2-acetamido-5-styrylbenzoic acid CAS No. 380365-20-8

DC18

(E)-5-(2-([1,1′-biphenyl]-3-yl)vinyl)-2-hydroxybenzoic acid

DC19

(E)-5-(2-([1,1′-biphenyl]-2-yl)vinyl)-2-hydroxybenzoic acid

DC21

(E)-5-(2-(9,9-dimethyl-9H-fluoren-2-yl)vinyl)-2- hydroxybenzoic acid

4 . The method of claim 1 , wherein said cancer is a radioresistant cancer.

5 . A method of treating cancer, said method comprising the step of administering to a patient in need thereof a pharmaceutically effective amount of a compound of general formula (I), a stereoisomer, or a pharmaceutically acceptable salt thereof, in combination with radiotherapy:

wherein:

ring A is a fluorenyl group substituted by 1 or 2—CH 3 groups;

each X is independently selected from:

—C 1-6 alkyl,

—O—C 1-6 alkyl,

—S—C 1-6 alkyl,

—OH,

—SH,

—CO 2 R 1 where R 1 is H or C 1-6 alkyl,

—SO 2 —C 1-6 alkyl,

—SO 2 —NR 2 R 3 where R 2 is H and R 3 is optionally substituted phenyl,

—NR 4 R 5 wherein R 4 and R 5 are independently selected from H, C 1-6 alkyl, and —CO—C 1-6 alkyl,

halogen, and

optionally substituted tetrazolyl;

n is an integer from 0 to 5; and

denotes an E or Z double bond.

6 . The method of claim 5 , wherein ring A is 9,9-dimethyl-9H-fluorenyl.

7 . The method of claim 4 , wherein said cancer is selected from the following: malignant gliomas, medulloblastoma, neuroblastoma, Kaposi's sarcoma, head and neck squamous cell carcinoma, tongue cancer, esophageal squamous cell carcinoma, thyroid cancer, melanoma, breast cancer, prostate cancer, laryngeal squamous cell carcinoma, lung cancer, mesothelioma, gastric cancer, hepatocellular carcinoma, pancreatic cancer, cholangiocarcinoma, colon cancer, renal cancer, urothelial cancer, testis cancer, endometrial cancer, ovarian cancer, cervical cancer, and metastases from any of these cancers, as well as myeloma, lymphoma and leukemia.

8 . The method of claim 7 , wherein said cancer is glioblastoma multiforme, lung cancer or breast cancer.

9 . The method of claim 7 , wherein said cancer is glioblastoma multiforme.

10 . The method of claim 3 , wherein said cancer is a radioresistant cancer.

11 . The method of claim 10 , wherein said cancer is selected from the following:

malignant gliomas, medulloblastoma, neuroblastoma, Kaposi's sarcoma, head and neck squamous cell carcinoma, tongue cancer, esophageal squamous cell carcinoma, thyroid cancer, melanoma, breast cancer, prostate cancer, laryngeal squamous cell carcinoma, lung cancer, mesothelioma, gastric cancer, hepatocellular carcinoma, pancreatic cancer, cholangiocarcinoma, colon cancer, renal cancer, urothelial cancer, testis cancer, endometrial cancer, ovarian cancer, cervical cancer, and metastases from any of these cancers, as well as myeloma, lymphoma and leukemia.

12 . The method of claim 11 , wherein said cancer is glioblastoma multiforme, lung cancer or breast cancer.

13 . The method of claim 11 , wherein said cancer is glioblastoma multiforme.

14 . The method of claim 5 , wherein said cancer is a radioresistant cancer.

15 . The method of claim 14 , wherein said cancer is selected from the following:

malignant gliomas, medulloblastoma, neuroblastoma, Kaposi's sarcoma, head and neck squamous cell carcinoma, tongue cancer, esophageal squamous cell carcinoma, thyroid cancer, melanoma, breast cancer, prostate cancer, laryngeal squamous cell carcinoma, lung cancer, mesothelioma, gastric cancer, hepatocellular carcinoma, pancreatic cancer, cholangiocarcinoma, colon cancer, renal cancer, urothelial cancer, testis cancer, endometrial cancer, ovarian cancer, cervical cancer, and metastases from any of these cancers, as well as myeloma, lymphoma and leukemia.

16 . The method of claim 15 , wherein said cancer is glioblastoma multiforme, lung cancer or breast cancer.

17 . The method of claim 15 , wherein said cancer is glioblastoma multiforme.

Priority Claims (1)
GB 1913785 · Sep 25, 2019 · national
Continuity (1)
Related Publication 20220362265A1 · Nov 17, 2022
References Cited (22)
US 8343994B2 · Motomura · 2013 [cited by examiner]
US 20070249647A1 · Vander Jagt · 2007 [cited by examiner]
US 20120196874A1 · Watt · 2012 [cited by examiner]
De Filippis, et al.; ChemMedChem, v12, pp. 558-570 (2017). (Year: 2017). [cited by examiner]
Patani, G. A. and LaVoie, E. J.; Chemical Reviews, v96, pp. 3147-3176; 1996 (Year: 1196). [cited by examiner]
Michelakis, et al.; British Journal of Cancer, v99, pp. 989-994; 2008 (Year: 2008). [cited by examiner]
Traversi, et al.; Mutagenesis, v31, pp. 433-441; 2016 (Year: 2016). [cited by examiner]
Wrobel, et al.; Journal of Medicinal Chemistry, v32, pp. 2493-2500; 1989 (Year: 1989). [cited by examiner]
Patani, G. A. and LaVoie, E. J.; Chemical Reviews, v96, pp. 3147-3176; 1996 (Year: 1996). [cited by examiner]
Shukla, et al.; Bioorganic & Medicinal Chemistry Letters, v21, pp. 6184-6187; 2011 (Year: 2011). [cited by examiner]
Sleire, et al.; Oncogene, v34, pp. 5951-5959; 2015 (Year: 2015). [cited by examiner]
PLoS ONE, vol. 12, No. 1, 2017, Chen et al., “Sensitization of Radioresistant Prostate Cancer Cells by Resveratrol Isolated from Arachis hypogaea Stems”. [cited by applicant]
International Journal of Biological Sciences, vol. 15, No. 2, 2019, pp. 430-440; Chen et ah, “Polydatin Increases Radiosensitivity by Inducing Apoptosis of Stem Cells in Colorectal Cancer”. [cited by applicant]
Phytomedicine, vol. 23, No. 5, 2016, pp. 566-577, Baek et al., “Resveratrol inhibits STAT3 signaling pathway through the induction of SOCS-1 : Role in apoptosis induction and radiosensitization in head and neck tumor ce… [cited by applicant]
Cancer Science, vol. 103, No. 6, 2012, pp. 1090-1098; Fang et ah, “Resveratrol enhances radiation sensitivity in prostate cancer by inhibiting cell proliferation and promoting cell senescence and apoptosis”. [cited by applicant]
International Journal of Oncology, vol. 43, No. 6, 2013, pp. 1999-2006; Luo et al., “Resveratrol enhances ionizing radiation-induced premature senescence in lung cancer cells”. [cited by applicant]
Radiation Oncology, vol. 6, 2011, Article No. 144; Rashid et al., “Resveratrol enhances prostate cancer cell response to ionizing radiation. Modulation of the AMPK, Akt and mTOR pathways”. [cited by applicant]
UK Search Report; Intellectual Property Office; United Kingdom Patent Application No. GB1913785.0; Mar. 19, 2020; 5 pages. [cited by applicant]
Intemational Search Report; International Searching Authority; International Patent Application No. PCT/GB2020/052335; Jan. 12, 2021; 5 pages. [cited by applicant]
Written Opinion; International Searching Authority; International Patent Application No. PCT/GB2020/052335; Jan. 12, 2021; 9 pages. [cited by applicant]
International Preliminary Report on Patentability; International Searching Authority; International Patent Application No. PCT/GB2020/052335; Apr. 7, 2022; 7 pages. [cited by applicant]
Shukla et al., “Inhibition of Xc- transporter-mediated cystine update by sulfasalazine analogs”, Bioorganic & Medicinal Chemistry Letters, vol. 21, 2011, pp. 6184-6187. [cited by applicant]