IP Library Granted Patent US 12,734,196
Granted Patent B2
US 12,734,196 · App. 17/792,632 · Granted Sep 15, 2026

Methods of differentiating neural cells and related compositions and methods of use

Inventors: Andres Bratt-Leal (San Diego, CA); Jeanne Loring (Del Mar, CA); Ha Tran (San Diego, CA); Roy Williams (Rancho Santa Fe, CA); Jim Mossman (San Diego, CA)
Assignee: Aspen Neuroscience, Inc.
A61K35/30A61P25/16C12N5/0619C12N2501/13C12N2501/15C12N2501/41C12N2501/999C12N2506/45
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Quick Facts
Patent No.
US 12,734,196
App. No.
17/792,632
Granted
Sep 15, 2026
Kind
B2
Abstract

The present disclosure provides methods of lineage specific differentiation of pluripotent stem cells, including induced pluripotent stem cells, into floor plate midbrain progenitor cells, determined dopamine (DA) neuron progenitor cells, and/or DA neurons. Also provided are compositions uses thereof, such as for treating neurodegenerative diseases and conditions, including Parkinson's disease.

Claims (47)

1 . A method of differentiating neural cells, the method comprising:

(a) performing a first incubation comprising culturing pluripotent stem cells in a non-adherent culture vessel under conditions to produce a cellular spheroid, wherein beginning at the initiation of the first incubation the cells are exposed to (i) an inhibitor of TGF-β/activin-Nodal signaling; (ii) at least one activator of Sonic Hedgehog (SHH) signaling; (iii) an inhibitor of bone morphogenetic protein (BMP) signaling; and (iv) an inhibitor of glycogen synthase kinase 3β (GSK3β) signaling, wherein day 0 is the first day on which the pluripotent stem cells are exposed to the inhibitor of TGF-β/activin-Nodal signaling and the inhibitor of bone morphogenetic protein (BMP) signaling; and

(b) performing a second incubation comprising culturing cells of the spheroid in a substrate-coated culture vessel under conditions to neurally differentiate the cells.

2 . The method of claim 1 , wherein the second incubation begins on about day 7.

3 . The method of claim 1 , wherein the cells are exposed:

(i) to the inhibitor of TGF-β/activin-Nodal signaling up to a day at or before day 7;

(ii) to the inhibitor of TGF-β/activin-Nodal beginning at day 0 and through day 6, inclusive of each day;

(iii) to the at least one activator of SHH signaling up to a day at or before day 7;

(iv) to the at least one activator of SHH signaling beginning at day 0 and through day 6, inclusive of each day;

(v) to the inhibitor of BMP signaling up to a day at or before day 11;

(vi) to the inhibitor of BMP signaling beginning at day 0 and through day 10, inclusive of each day;

(vii) to the inhibitor of GSK3B signaling up to a day at or before day 13;

(viii) to the inhibitor of GSK3B signaling beginning at day 0 and through day 12, inclusive of each day;

(ix) to transforming growth factor beta-3 (TGFβ3), ascorbic acid, GDNF, and dbcAMP (collectively, “BAGCT”) and an inhibitor of Notch signaling beginning on day 11; and/or

(x) to BAGCT and an inhibitor of Notch signaling beginning at day 11 and until harvest of the neurally differentiated cells, optionally until day 18, optionally until day 25.

4 . The method of claim 1 , wherein culturing the cells under conditions to neurally differentiate the cells comprises exposing the cells to (i) brain-derived neurotrophic factor (BDNF); (ii) ascorbic acid; (iii) glial cell-derived neurotrophic factor (GDNF); (iv) dibutyryl cyclic AMP (dbcAMP); (v) transforming growth factor beta-3 (TGFβ3) (collectively, “BAGCT”); and (vi) an inhibitor of Notch signaling.

5 . The method of claim 1 , wherein the method comprises:

(a) beginning at the initiation of the first incubation day, the cells are exposed to (i) an inhibitor of TGF-β/activin-Nodal signaling through day 6, each day inclusive; (ii) at least one activator of Sonic Hedgehog (SHH) signaling through day 6, each day inclusive; (iii) an inhibitor of bone morphogenetic protein (BMP) signaling through day 6, each day inclusive; and (iv) an inhibitor of glycogen synthase kinase 3β (GSK3β) signaling through day 6, each day inclusive; and

(b) the performing a second incubation begins on day 7, and comprises culturing the cells in a culture vessel coated with a substrate selected from the group consisting of: laminin, collagen, entactin, heparin sulfate proteoglycans, and combinations thereof, wherein beginning on day 7, the cells are exposed to (i) an inhibitor of BMP signaling and (ii) an inhibitor of GSK3β signaling; and beginning on day 11, the cells are exposed to (i) brain-derived neurotrophic factor (BDNF); (ii) ascorbic acid; (iii) glial cell-derived neurotrophic factor (GDNF); (iv) dibutyryl cyclic AMP (dbcAMP); (v) transforming growth factor beta-3 (TGFβ3) (collectively, “BAGCT”); and (vi) an inhibitor of Notch signaling.

6 . The method of claim 1 , further comprising harvesting the neurally differentiated cells, optionally wherein the harvesting is carried out at about day 16 or later, wherein the harvesting is carried out between day 18 and day 25, and/or wherein the harvesting is carried out at or about day 18 or about at day 25.

7 . The method of claim 1 , wherein the neurally differentiated cells are determined dopaminergic neuron progenitor cells, optionally wherein the dopaminergic neuron progenitor cells are capable of innervating host tissue upon transplantation into a subject.

8 . The method of claim 1 , wherein, prior to performing the second incubation, the spheroid is dissociated to produce a cell suspension, and cells of the cell suspension are cultured in the substrate-coated culture vessel, optionally wherein the dissociating is carried out at a time when the spheroid cells express at least one of PAX6 and OTX2, and/or wherein the dissociating is carried out on about day 7, and/or wherein the spheroid is dissociated by enzymatic dissociation.

9 . The method of claim 1 , wherein the inhibitor of TGF-β/activin-Nodal signaling is SB431542; wherein the at least one activator of SHH signaling is SHH protein, purmorphamine, C25II SHH protein, or a combination thereof; and/or wherein the inhibitor of BMP signaling is LDN193189, and/or wherein the inhibitor of GSK3β signaling is CHIR99021; and/or wherein the inhibitor of Notch signaling is DAPT.

10 . The method claim 1 , wherein the culturing in the first incubation and/or the second incubation is carried out in media comprising serum or a serum replacement, or wherein the cells are cultured in the absence of serum for the duration of culture.

11 . The method of claim 1 , wherein the cells are exposed to an inhibitor of Rho-associated protein kinase (ROCK) signaling on day 0, day 8, day 16, and/or day 20.

12 . The method of claim 1 , wherein the first incubation is performed in media that comprises the inhibitor of TGF-β/activin-Nodal signaling, the activator of SHH signaling, the inhibitor of BMP signaling, and the inhibitor of GSK3β signaling, and at least about 50% of the media is replaced daily or every other day, or every third day.

13 . The method of claim 6 , further comprising formulating the harvested cells with a cryoprotectant, optionally further comprising cryopreserving the harvested cells, and optionally thawing the cells prior to use.

14 . The method of claim 1 , wherein the pluripotent stem cells are embryonic stem (ES) cells, induced pluripotent stem cells (iPSCs), or a combination thereof.

15 . The method of claim 1 , wherein the pluripotent stem cells are human induced pluripotent stem cells, and/or wherein the pluripotent stem cells are autologous to a subject to be treated with the neurally differentiated cells or allogeneic to a subject to be treated with the neurally differentiated cells.

16 . The method of claim 15 , wherein the pluripotent stem cells are hypoimmunogenic, optionally wherein the pluripotent stem cells are engineered to (a) remove genes encoding one or more of polymorphic HLA-A/-B/-C and HLA class II molecules; and (b) to provide genes encoding one or more of PD-L1, HLA-G, and CD47, optionally into a AAVS1 safe harbor locus.

17 . A method of differentiating neural cells, the method comprising:

(a) performing a first incubation comprising culturing pluripotent stem cells in a culture vessel that is coated with a substrate selected from one or more of laminin, collagen, entactin, heparin sulfate proteoglycans, and combinations thereof, wherein beginning at the initiation of the first incubation the cells are exposed to (i) an inhibitor of TGF-β/activin-Nodal signaling; (ii) at least one activator of Sonic Hedgehog (SHH) signaling; (iii) an inhibitor of bone morphogenetic protein (BMP) signaling; and (iv) an inhibitor of glycogen synthase kinase 3β (GSK3β) signaling, wherein day 0 is the first day on which the pluripotent stem cells are exposed to the inhibitor of TGF-β/activin-Nodal signaling and the inhibitor of bone morphogenetic protein (BMP) signaling, wherein the method does not comprise exposing the cells to fibroblast growth factor 8 (FGF8); and

(b) performing a second incubation comprising culturing cells obtained in step (a) in a substrate-coated culture vessel under conditions to neurally differentiate the cells.

18 . The method of claim 17 , wherein the substrate comprises laminin.

19 . The method of claim 17 , wherein the inhibitor of TGF-β/activin-Nodal signaling is SB431542.

20 . The method of claim 17 , wherein the at least one activator of SHH signaling is SHH protein, purmorphamine, C25II SHH protein, or a combination thereof.

21 . The method of claim 17 , wherein the inhibitor of BMP signaling is LDN193189.

22 . The method of claim 17 , wherein the inhibitor of GSK3B signaling is CHIR99021.

23 . The method of claim 1 , wherein the method does not comprise exposing the cells to fibroblast growth factor 8 (FGF8).

24 . A method of differentiating neural cells, the method comprising:

(a) performing a first incubation comprising culturing pluripotent stem cells in a culture vessel that is coated with a substrate selected from one or more of laminin, collagen, entactin, heparin sulfate proteoglycans, and combinations thereof, wherein beginning at the initiation of the first incubation the cells are exposed to (i) an inhibitor of TGF-β/activin-Nodal signaling; (ii) at least one activator of Sonic Hedgehog (SHH) signaling; (iii) an inhibitor of bone morphogenetic protein (BMP) signaling comprising LDN193189; and (iv) an inhibitor of glycogen synthase kinase 3β (GSK3β) signaling, wherein day 0 is the first day on which the pluripotent stem cells are exposed to the inhibitor of TGF-β/activin-Nodal signaling and the inhibitor of bone morphogenetic protein (BMP) signaling; and

(b) performing a second incubation comprising culturing cells obtained in step (a) in a substrate-coated culture vessel under conditions to neurally differentiate the cells.

25 . The method of claim 24 , wherein the substrate comprises laminin.

26 . The method of claim 24 , wherein the inhibitor of TGF-β/activin-Nodal signaling is SB431542.

27 . The method of claim 24 , wherein the at least one activator of SHH signaling is SHH protein, purmorphamine, C25II SHH protein, or a combination thereof.

28 . The method of claim 24 , wherein the inhibitor of GSK3β signaling is CHIR 99021.

29 . The method of claim 24 , wherein the method does not comprise exposing the cells to fibroblast growth factor 8 (FGF8).

Assignments (1)
ASSIGNMENT OF ASSIGNOR'S INTEREST Recorded Jan 14, 2025
From: BRATT-LEAL, ANDRES, DR.; LORING, JEANNE, DR.; TRAN, HA, DR.
To: ASPEN NEUROSCIENCE, INC.
Reel/Frame 069860/0168 →
Continuity (2)
Provisional Application 62960669 · Jan 13, 2020
Related Publication 20230059010A1 · Feb 23, 2023
References Cited (165)
US 5033252A · Carter · 1991 [cited by applicant]
US 5052558A · Carter · 1991 [cited by applicant]
US 5323907A · Kalvelage · 1994 [cited by applicant]
US 5538848A · Livak · 1996 [cited by applicant]
US 5925517A · Tyagi · 1999 [cited by applicant]
US 6174670B1 · Wittwer · 2001 [cited by applicant]
US 6326145B1 · Whitcombe · 2001 [cited by applicant]
US 6329144B1 · Kubista · 2001 [cited by applicant]
US 6635427B2 · Wittwer · 2003 [cited by applicant]
US 7510687B2 · Mazzeo · 2009 [cited by applicant]
US 8442772B2 · Loring · 2013 [cited by applicant]
US 8642334B2 · Chambers · 2014 [cited by applicant]
US 9453198B2 · Studer · 2016 [cited by applicant]
US 9926529B2 · Gonzalez · 2018 [cited by applicant]
US 10260041B2 · Chambers · 2019 [cited by applicant]
US 10273452B2 · Chambers · 2019 [cited by applicant]
US 10280398B2 · Studer · 2019 [cited by applicant]
US 10287546B2 · Chambers · 2019 [cited by applicant]
US 10711243B2 · Studer · 2020 [cited by applicant]
US 10828335B2 · George · 2020 [cited by applicant]
US 10858625B2 · Studer · 2020 [cited by applicant]
US 11236302B2 · Kirkeby · 2022 [cited by applicant]
US 11261425B2 · Takahashi · 2022 [cited by applicant]
US 11473058B2 · Takahashi · 2022 [cited by applicant]
US 11560546B2 · Chambers · 2023 [cited by applicant]
US 12013388B2 · Jungblut · 2024 [cited by applicant]
US 20110118130A1 · Loring · 2011 [cited by applicant]
US 20140017212A1 · Rebar · 2014 [cited by applicant]
US 20150064139A1 · Shoemaker · 2015 [cited by applicant]
US 20150265652A1 · George · 2015 [cited by applicant]
US 20160201032A1 · Studer · 2016 [cited by applicant]
US 20160215260A1 · Takahashi · 2016 [cited by examiner]
US 20170292112A1 · Chang · 2017 [cited by applicant]
US 20180298326A1 · Studer · 2018 [cited by applicant]
US 20200338148A1 · Abeliovich · 2020 [cited by applicant]
US 20200407680A1 · Studer · 2020 [cited by applicant]
US 20210000929A1 · Mason · 2021 [cited by applicant]
US 20210123017A1 · Ozaki · 2021 [cited by applicant]
US 20210123018A1 · Studer · 2021 [cited by applicant]
US 20210292715A1 · Schrepfer · 2021 [cited by applicant]
US 20210393691A1 · Cooper · 2021 [cited by examiner]
US 20220177835A1 · Studer · 2022 [cited by applicant]
US 20220186180A1 · Studer · 2022 [cited by applicant]
US 20220254448A1 · Loring · 2022 [cited by applicant]
US 20230081881A1 · Bratt-Leal · 2023 [cited by applicant]
US 20230165909A1 · Zhang · 2023 [cited by applicant]
US 20230233617A1 · Ericson · 2023 [cited by applicant]
US 20230340407A1 · Yuejun · 2023 [cited by applicant]
CA 3167713 · 2021 [cited by applicant]
EP 3042951 · 2016 [cited by applicant]
EP 3042951A1 · 2016 [cited by examiner]
EP 3061809 · 2016 [cited by applicant]
EP 3447130 · 2019 [cited by applicant]
WO 2008051604 · 2008 [cited by applicant]
WO 2008132176 · 2008 [cited by applicant]
WO 2010126614 · 2010 [cited by applicant]
WO 2011019092 · 2011 [cited by applicant]
WO 2011149762 · 2011 [cited by applicant]
WO 2013015457 · 2013 [cited by applicant]
WO 2013067362 · 2013 [cited by applicant]
WO 2014200905 · 2014 [cited by applicant]
WO 2015143342 · 2015 [cited by applicant]
WO 2016162747 · 2016 [cited by applicant]
WO 2017070633 · 2017 [cited by applicant]
WO 2017132596 · 2017 [cited by applicant]
WO 2017160234 · 2017 [cited by applicant]
WO 2017222248 · 2017 [cited by applicant]
WO 2019068854 · 2019 [cited by applicant]
WO 2019111258 · 2019 [cited by applicant]
WO 2019217943 · 2019 [cited by applicant]
WO 2021016607 · 2021 [cited by applicant]
WO 2021081229 · 2021 [cited by applicant]
WO 2021087145 · 2021 [cited by applicant]
WO 2021203009 · 2021 [cited by applicant]
WO 2021216622 · 2021 [cited by applicant]
WO 2021216623 · 2021 [cited by applicant]
WO 2021216846 · 2021 [cited by applicant]
WO 2021224496 · 2021 [cited by applicant]
WO 2022062960 · 2022 [cited by applicant]
WO 2022216911 · 2022 [cited by applicant]
WO 2023004366 · 2023 [cited by applicant]
WO 2023004370 · 2023 [cited by applicant]
WO 2023004371 · 2023 [cited by applicant]
Deuse et al., “Hypoimmunogenic derivatives of induced pluripotent stem cells evade immune rejection in fully immunocompetent allogeneic recipients”. Nat Biotechnol 37, 252-258 (2019). (Year: 2019). [cited by examiner]
Borghese et al., “Inhibition of notch signaling in human embryonic stem cell-derived neural stem cells delays G1/S phase transition and accelerates neuronal differentiation in vitro and in vivo”. Stem Cells. 2010, Abstr… [cited by examiner]
Vallier et al., “Signaling pathways controlling pluripotency and early cell fate decisions of human induced pluripotent stem cells,” Stem Cells (2009) 27: 2655-2666. [cited by applicant]
Lavaute et al., “Regulation of Neural specification from human embryonic stem cells by BMP and FGF,” Stem Cells (2009) 27: 1741-1749. [cited by applicant]
Patani et al., “Activin/Nodal inhibition alone accelerates highly efficient neural conversion from human embryonic stem cells and imposes a caudal positional identity,” PlosOne (2009) 4:e7327. [cited by applicant]
Cuny et al., “Structure-activity relationship study of bone morphogenetic protein (BMP) signaling inhibitors,” Bioorg & Medicinal Chemistry Letters (2008) 18: 4388-4392. [cited by applicant]
Hu et al., “Neural differentiation of human induced pluripotent stem cells follows developmental principles but with variable potency,” Proc. Nat'l. Acad. Sci USA (2010) 9: 4335-4340. [cited by applicant]
Lamb et al., “Fibroblast growth factor is a direct neuronal inducer, which combined with noggin generates anterior-posterior neural pattern,” Development (1995) 121: 3627-3636. [cited by applicant]
European Patent Office Decision to Grant dated Jun. 5, 2024. [cited by applicant]
European Patent Office Communication pursuant to Article 94(3) EPC dated Nov. 27, 2023. [cited by applicant]
International Preliminary Report on Patentability for PCT/US2021/013324, Aspen Neuroscience, Inc., filed Jan. 13, 2021, dated Jul. 19, 2022. [cited by applicant]
Arenas et al., “How to make a midbrain dopaminergic neuron,” Development (2015) 142(11):1918-1936. [cited by applicant]
Arjona et al., “Autotransplantation of human carotid body cell aggregates for treatment of Parkinson's disease,” Neurosurgery. (2003) 53(2):321-8. [cited by applicant]
Baden et al., “Insights into GBA Parkinson's disease pathology and therapy with induced pluripotent stem cell model systems,” Neurobiol Dis. (2019), 127:1-12. [cited by applicant]
Badger et al., “Parkinson's disease in a dish—Using stem cells as a molecular tool,” Neuropharmacology (2014) 76:88-96. [cited by applicant]
Bain et al., “Embryonic stem cells express neuronal properties in vitro,” Dev Biol. (1995) 168(2):342-57. [cited by applicant]
Bakay et al., “Implantation of Spheramine in advanced Parkinson's disease (PD),” Front Biosci. (2004) 9:592-602. [cited by applicant]
Berge-Seidl et al., “The GBA variant E326K is associated with Parkinson's disease and explains a genome-wide association signal,” Neuroscience Letters (2017) 658:48-52. [cited by applicant]
Bjorklund et al., “Neural transplantation for the treatment of Parkinson's disease,” Lancet Neurol. (2003) 2 (7):437-45. [cited by applicant]
Brundin et al., “Neural grafting in Parkinson's disease Problems and possibilities,” Prog Brain Res. (2010) 184:265-94. [cited by applicant]
Brunet et al., “Metagenes and molecular pattern discovery using matrix factorization,” Proceedings of the National Academy of Sciences (2004) 101(12):4164-4169. [cited by applicant]
Cyranoski, “‘Reprogrammed’ stem cells implanted into patient with Parkinson's disease,” Nature. (2018) doi: https://doi.org/10.1038/d41586-018-07407-9. [cited by applicant]
Deng et al., “The genetics of Parkinson disease,” Ageing Research Reviews (2018) 42:72-85. [cited by applicant]
Doi et al., “Isolation of human induced pluripotent stem cell-derived dopaminergic progenitors by cell sorting for successful transplantation,” Stem Cell Reports (2014) 2(3):337-50. [cited by applicant]
Fitzpatrick et al., “Cell-based therapies for Parkinson's disease: past, present, and future,” Antioxid Redox Signal. (2009) 11(9):2189-2208. [cited by applicant]
Ghatak et al., “Parkinson's disease: what the model systems have taught US so far,” Journal of Genetics (2018) 97 (3):729-751. [cited by applicant]
Han et al., “Generation of hypoimmunogenic human pluripotent stem cells,” Proc Natl Acad Sci USA. (2019) 116 (21):10441-10446. [cited by applicant]
Hanss et al., “Quality Control Strategy for CRISPR-Cas9-Based Gene Editing Complicated by a Pseudogene,” Frontiers in Genetics (2020) 10:1297. [cited by applicant]
Heijer et al., “A Large-Scale Full GBA1 Gene Screening in Parkinson's Disease in the Netherlands,” Mov Disord. (2020) 35(9):1667-1674. [cited by applicant]
Kan et al., “Dopaminergic differentiation of human mesenchymal stem cells—utilization of bioassay for tyrosine hydroxylase expression,” Neurosci Lett (2007) 419(1):28-33. [cited by applicant]
Kawaguchi et al., “Single-cell gene profiling defines differential progenitor subclasses in mammalian neurogenesis,” Development (2008) 135(18):3113-24. [cited by applicant]
Kim et al., “miR-371-3 expression predicts neural differentiation propensity in human pluripotent stem cells,” Cell Stem Cell (2011) 8(6):695-706. [cited by applicant]
Kirkeby et al., “Generation of Regionally Specified Neural Progenitors and Functional Neurons from Human Embryonic Stem Cells under Defined Conditions,” Cell Reports (2012) 1(6):703-714. [cited by applicant]
Kordower et al., “Neuropathological evidence of graft survival and striatal reinnervation after the transplantation of fetal mesencephalic tissue in a patient with Parkinson's disease,” N Engl J Med. (1995) 332(17):1118… [cited by applicant]
Kriks et al., “Floor plate-derived dopamine neurons from hESCs efficiently engraft in animal models of PD,” Nature (2011) 480(7378):547-51. [cited by applicant]
Lee et al., “Efficient generation of midbrain and hindbrain neurons from mouse embryonic stem cells,” Nat Biotechnol. (2000) 18(6):675-9. [cited by applicant]
Lehnen et al., “IAP-Based Cell Sorting Results in Homogeneous Transplantable Dopaminergic Precursor Cells Derived from Human Pluripotent Stem Cells,” Stem Cell Reports (2017) 9(4):1207-1220. [cited by applicant]
Lindvall et al., “Stem cells in human neurodegenerative disorders—time for clinical translation?” J Clin Invest. (2010) 120(1):29-40. [cited by applicant]
Liu et al., “Genome wide profiling of human embryonic stem cells (hESCs), their derivatives and embryonal carcinoma cells to develop base profiles of U.S. Federal government approved hESC lines,” BMC Dev Biol (2006) 6:2… [cited by applicant]
Madrazo et al., “Open microsurgical autograft of adrenal medulla to the right caudate nucleus in two patients with intractable Parkinson's disease,” N Engl J Med. (1987) 316(14):831-4. [cited by applicant]
Martinez-Cerdeno et al., “Neural Progenitor Cell Terminology,” Front Neuroanat (2018) 12:104. [cited by applicant]
Mendez et al., I., “Dopamine neurons implanted into people with Parkinson's disease survive without pathology for 14 years,” Nat Med (2008) 14(5):507-509. [cited by applicant]
Muller et al., “Regulatory networks define phenotypic classes of human stem cell lines,” Nature (2008) 455:401-405. [cited by applicant]
Ninkovic et al., “The transcription factor Pax6 regulates survival of dopaminergic olfactory bulb neurons via crystallin αA,” Neuron (2010) 68(4):682-94. [cited by applicant]
Noisa et al., “Neural Progenitor Cells Derived from Human Embryonic Stem Cells as an Origin of Dopaminergic Neurons,” Stem Cells Int (2015) 2015:647437. [cited by applicant]
Nowrousian et al., “Next-generation sequencing techniques for eukaryotic microorganisms: sequencing-based solutions to biological problems,” Eukaryot Cell. (2010) 9(9):1300-10. [cited by applicant]
Ochalek et al., “Generation of Cholinergic and Dopaminergic Interneurons from Human Pluripotent Stem Cells as a Relevant Tool for In Vitro Modeling of Neurological Disorders Pathology and Therapy,” Stem Cells Int. (2016… [cited by applicant]
Okabe et al., “Development of neuronal precursor cells and functional postmitotic neurons from embryonic stem cells in vitro,” Mech Dev. (1996) 59(1):89-102. [cited by applicant]
Okamoto et al., “Highly efficient genome editing for single-base substitutions using optimized ssODNs with Cas9-RNPs,” Scientific Reports (2019) 9(1):1-11. [cited by applicant]
Paix et al, “Precision genome editing using CRISPR-Cas9 and linear repair templates in C. elegans,” Methods (2017) 121-122:86-93. [cited by applicant]
Piccini et al., “Dopamine release from nigral transplants visualized in vivo in a Parkinson's patient,” Nat Neurosci. (1999) 2(12):1137-40. [cited by applicant]
Raikwar et al., “Next generation precision medicine: CRISPR-mediated genome editing for the treatment of neurodegenerative disorders,” Journal of Neuroimmune Pharmacology (2019) 14(4):608-641. [cited by applicant]
Reinhardt et al., “Genetic correction of a LRRK2 mutation in human iPSCs links parkinsonian neurodegeneration to ERK-dependent changes in gene expression,” Cell Stem Cell (2013) 12(3):354-367. [cited by applicant]
Romito et al., “Pluripotent Stem Cells: Current Understanding and Future Directions,” Stem Cells Int. (2016) 2016:9451492, 20 pages. [cited by applicant]
Sanders et al., “LRRK2 mutations cause mitochondrial DNA damage in iPSC-derived neural cells from Parkinson's disease patients: reversal by gene correction,” Neurobiology of Disease (2014) 62:381-386. [cited by applicant]
Shendure et al., “Advanced sequencing technologies: methods and goals,” Nat Rev Genet. (2004) 5(5):335-44. [cited by applicant]
Sison et al., “Using patient-derived induced pluripotent stem cells to identify Parkinson's disease-relevant phenotypes,” Current Neurology and Neuroscience Reports (2018) 18(12):84, 1-14. [cited by applicant]
Smits et al., “Modeling Parkinson's disease in midbrain-like organoids,” NPJ Parkinson's Disease (2019) 5(1):1-8. [cited by applicant]
Stepanichev, “Prospects for the Use of Genome-Editing Technology to Correct Neurodegenerative Diseases,” Advances in Gerontology (2019) 9(2):154-163. [cited by applicant]
Tabar et al., “Therapeutic cloning in individual parkinsonian mice,” Nat Med. (2008) 14(4):379-81. [cited by applicant]
Takahashi et al., “Induction of pluripotent stem cells from mouse embryonic and adult fibroblast cultures by defined factors,” Cell (2006) 126(4):663-676. [cited by applicant]
Takahashi, “Strategies for bringing stem cell-derived dopamine neurons to the clinic: The Kyoto trial,” Prog Brain Res. (2017) 230:213-226. [cited by applicant]
Tieng et al., “Engineering of midbrain organoids containing long-lived dopaminergic neurons,” Stem Cells and Development (2014) 23(13):1535-1547. [cited by applicant]
Tsuji et al., “Genetic heterogeneity in type 1 Gaucher disease: multiple genotypes in Ashkenazic and non-Ashkenazic individuals,” Proceedings of the National Academy of Sciences (1988) 85(7):2349-2352. [cited by applicant]
Vakulskas et al., “A high-fidelity Cas9 mutant delivered as a ribonucleoprotein complex enables efficient gene editing in human hematopoietic stem and progenitor cells,” Nature Medicine (2018) 24(8):1216-1224. [cited by applicant]
Velez-Pardo et al., “The distribution and risk effect of GBA variants in a large cohort of PD patients from Colombia and Peru,” Parkinsonism & Related Disorders (2019) 63:204-208. [cited by applicant]
Vetchinova et al., “Cytogenetic analysis of the results of genome editing on the cell model of Parkinson's disease,” Bulletin of Experimental Biology and Medicine (2018) 165(3):378-381. [cited by applicant]
Kim et al., “Neural stem cells derived from human midbrain organoids as a stable source for treating Parkinson's disease Midbrain organoid-NSCs (Og-NSC) as a stable source for PD treatment,” Progress in Neurobiology (20… [cited by applicant]
Ye et al., “FGF and Shh signals control dopaminergic and serotonergic cell fate in the anterior neural plate,” Cell. (1998) 93(5):755-66. [cited by applicant]
Yu et al., “Human induced pluripotent stem cells free of vector and transgene sequences,” Science (2009) 324 (5928):797-801. DOI: 10.1126/science.1172482. [cited by applicant]
Kirkeby Agnete et al, “Predictive Markers Guide Differentiation to Improve Graft Outcome in Clinical Translation of hESC-Based Therapy for Parkinson's Disease”, Cell Stem Cell, Amsterdam, NL, (Jan. 1, 2017), vol. 20, No… [cited by applicant]
Chambers et al., “Highly efficient neural conversion of human ES and iPS cells by dual inhibition of SMAD signaling,” Nat Biotechnol (2009) 27(3):275-80. [cited by applicant]
Fasano et al. “Retraction Notice to: Efficient derivation of functional floor plate tissue from human embryonic stem cells,” Cell Stem Cell (2023) 30(6): 905. [cited by applicant]
Fasano et al., “Efficient derivation of functional floor plate tissue from human embryonic stem cells,” Cell Stem Cell (2010) 6(4):336-47. [cited by applicant]
Nolbrant et al.. “Generation of high-purity human ventral midbrain dopaminergic progenitors for in vitro maturation and intracerebral transplantation”. Nature Protocols (2017) 12(9): 1962-1979. [cited by applicant]
Shimojo et al., “Rapid, efficient, and simple motor neuron differentiation from human pluripotent stem cells,” Molecular Brain (2015) 8:79. [cited by applicant]
First Office Action for Chinese Patent Application No. 2021800206479 (English Translation). [cited by applicant]
Patent Search Report for Chinese Patent Application No. 2021800206479 (English Translation). [cited by applicant]
“Neural Stem Cells”, Military Science Publishing House, published on Dec 31, 2006) (English translation). [cited by applicant]
Patent Search Report for Chinese Patent Application No. 2024116141573. [cited by applicant]
Rejection Decision for Chinese Patent Application No. 202411614157.3 (English translation). [cited by applicant]
First Office Action for Chinese Patent Application No. 202411614157.3 (English translation). [cited by applicant]